Catarina Dias
- Cell Biology top 10%
- Cellular transport and secretion 6
- Hemoglobin structure and function 2
-
- Lipid Membrane Structure and Behavior 3
- S100 Proteins and Annexins 3
- Mitochondrial Function and Pathology 1
-
- Phagocytosis and Immune Regulation 2
-
- Autophagy in Disease and Therapy 5
-
- Genetic Neurodegenerative Diseases 3
- Co-authors
- Jesper NylandstedStine Lauritzen SønderAdam Cohen SimonsenLisa B. FrankelAnna MularskiFrederik W. LundManolis FantoOlga Baron
- Cited by
- Cell BiologyMolecular BiologyAging
- Partner nations
- DenmarkUnited KingdomCzechia
In The Last Decade
Catarina Dias
16 papers receiving 402 citations
Hit Papers
Peers
Comparison fields: 5 of 92
- Cell Biology 118
- Molecular Biology 224
- Aging 5
- Physiology 12
- Immunology 47
Countries citing papers authored by Catarina Dias
This map shows the geographic impact of Catarina Dias's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Catarina Dias with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Catarina Dias more than expected).
Fields of papers citing papers by Catarina Dias
This network shows the impact of papers produced by Catarina Dias. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Catarina Dias. The network helps show where Catarina Dias may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Catarina Dias, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 10 | |
| 2 | 2023 | 3 | |
| 3 | 2023 | 2 | |
| 4 | 2022 | 6 | |
| 5 | 2022 | 1 | |
| 6 | 2021 | 18 | |
| 7 | 2021 | 6 | |
| 8 | Plasma membrane integrity in health and disease: significance and therapeutic potentialbreakdown → | 2021 | 173 |
| 9 | 2021 | 6 | |
| 10 | 2021 | 17 | |
| 11 | 2021 | 48 | |
| 12 | 2021 | 20 | |
| 13 | 2020 | 32 | |
| 14 | 2019 | 7 | |
| 15 | 2017 | 56 | |
| 16 | The Functions of CHIP in Age Related Disease | 2016 | 1 |
About Catarina Dias
Catarina Dias is a scholar working on Cell Biology, Aging, Developmental Neuroscience, Cellular and Molecular Neuroscience and Rehabilitation, having authored 16 papers that have together received 406 indexed citations. Recurring topics across this work include Cellular transport and secretion (6 papers), Autophagy in Disease and Therapy (5 papers), Lipid Membrane Structure and Behavior (3 papers), Genetic Neurodegenerative Diseases (3 papers), S100 Proteins and Annexins (3 papers), Hemoglobin structure and function (2 papers), Phagocytosis and Immune Regulation (2 papers) and Mitochondrial Function and Pathology (1 paper). The work is most often cited by research in Cell Biology (118 citations), Molecular Biology (224 citations), Aging (5 citations), Physiology (12 citations) and Immunology (47 citations). Catarina Dias has collaborated with scholars based in Denmark, United Kingdom and Czechia. Frequent co-authors include Jesper Nylandsted, Stine Lauritzen Sønder, Adam Cohen Simonsen, Lisa B. Frankel, Anna Mularski, Frederik W. Lund, Manolis Fanto, Olga Baron, Gillian P. Bates and Gema Vizcay‐Barrena. Their work appears in journals such as Scientific Reports, iScience, Current Biology, Frontiers in Cell and Developmental Biology and Cell Discovery.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.