Carlos E. Morado-Urbina
- Physiology
- Cellular and Molecular Neuroscience
- Infectious Diseases
- Psychiatry and Mental health
- Molecular Biology
- Co-authors
- Juan Miguel Jiménez‐AndradeJames C. EisenachAbigail Alvarado-VázquezChristopher M. PetersKen‐ichiro HayashidaCamilla I. SvenssonTakashi SutoMasafumi Kimura
- Topics
- Pain Mechanisms and Treatments (6 papers)Neuropeptides and Animal Physiology (4 papers)Stress Responses and Cortisol (2 papers)
- Journals
- The LancetPainAnesthesiology
- Partner nations
- United StatesSwedenMexico
In The Last Decade
Carlos E. Morado-Urbina
13 papers receiving 300 citations
Peers
Comparison fields: 5 of 73
- Physiology 135
- Cellular and Molecular Neuroscience 70
- Infectious Diseases 49
- Psychiatry and Mental health 48
- Molecular Biology 44
Countries citing papers authored by Carlos E. Morado-Urbina
This map shows the geographic impact of Carlos E. Morado-Urbina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Carlos E. Morado-Urbina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Carlos E. Morado-Urbina more than expected).
Fields of papers citing papers by Carlos E. Morado-Urbina
This network shows the impact of papers produced by Carlos E. Morado-Urbina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Carlos E. Morado-Urbina. The network helps show where Carlos E. Morado-Urbina may publish in the future.
Co-authorship network of co-authors of Carlos E. Morado-Urbina
This figure shows the co-authorship network connecting the top 25 collaborators of Carlos E. Morado-Urbina. A scholar is included among the top collaborators of Carlos E. Morado-Urbina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Carlos E. Morado-Urbina. Carlos E. Morado-Urbina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 0 | |
| 3 | 42 | |
| 4 | 2 | |
| 5 | 10 | |
| 6 | 41 | |
| 7 | 25 | |
| 8 | 10 | |
| 9 | 18 | |
| 10 | 42 | |
| 11 | 19 | |
| 12 | 10 | |
| 13 | 39 | |
| 14 | 49 |
About Carlos E. Morado-Urbina
Carlos E. Morado-Urbina is a scholar working on Behavioral Neuroscience, Biological Psychiatry and Cellular and Molecular Neuroscience, having authored 14 papers that have together received 308 indexed citations. Recurring topics across this work include Pain Mechanisms and Treatments (6 papers), Neuropeptides and Animal Physiology (4 papers) and Stress Responses and Cortisol (2 papers). The work is most often cited by research in Physiology (135 citations), Neurology (39 citations) and Behavioral Neuroscience (15 citations). Carlos E. Morado-Urbina has collaborated with scholars based in United States, Sweden and Mexico. Frequent co-authors include Juan Miguel Jiménez‐Andrade, James C. Eisenach, Abigail Alvarado-Vázquez, Christopher M. Peters, Ken‐ichiro Hayashida, Camilla I. Svensson, Takashi Suto, Masafumi Kimura, Aracely Evangelina Chávez‐Piña and Kim Kultima. Their work appears in journals such as The Lancet, Pain and Anesthesiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.