C. Boyer

1.8k total citations
45 papers, 1.6k citations indexed

About

C. Boyer is a scholar working on Oncology, Radiology, Nuclear Medicine and Imaging and Immunology. According to data from OpenAlex, C. Boyer has authored 45 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Oncology, 11 papers in Radiology, Nuclear Medicine and Imaging and 9 papers in Immunology. Recurrent topics in C. Boyer's work include Monoclonal and Polyclonal Antibodies Research (11 papers), HER2/EGFR in Cancer Research (7 papers) and Toxin Mechanisms and Immunotoxins (4 papers). C. Boyer is often cited by papers focused on Monoclonal and Polyclonal Antibodies Research (11 papers), HER2/EGFR in Cancer Research (7 papers) and Toxin Mechanisms and Immunotoxins (4 papers). C. Boyer collaborates with scholars based in United States, France and Switzerland. C. Boyer's co-authors include Robert C. Bast, Regina S. Whitaker, Andrew Berchuck, Joseph A. Zasadzinski, Ben S. Cooper, Fengfeng Xu, Jon R. Wiener, Shu Wu, C. A. Evans and Bret A. Coldren and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Clinical Oncology.

In The Last Decade

C. Boyer

44 papers receiving 1.5k citations

Peers

C. Boyer
Comparison fields: 5 of 118
  • Molecular Biology 533
  • Oncology 375
  • Electronic, Optical and Magnetic Materials 315
  • Immunology 277
  • Radiology, Nuclear Medicine and Imaging 250
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Citations per field, relative to C. Boyer
C. Boyer · 1×
Citations per year, relative to C. Boyer
C. Boyer · 1×

Countries citing papers authored by C. Boyer

Since Specialization
Citations

This map shows the geographic impact of C. Boyer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by C. Boyer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites C. Boyer more than expected).

Fields of papers citing papers by C. Boyer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by C. Boyer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by C. Boyer. The network helps show where C. Boyer may publish in the future.

Co-authorship network of co-authors of C. Boyer

This figure shows the co-authorship network connecting the top 25 collaborators of C. Boyer. A scholar is included among the top collaborators of C. Boyer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with C. Boyer. C. Boyer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 4
2
DURABILITY OF IMMUNE RESPONSES INDUCED BY THREE LEADING CANDIDATE EBOLA VACCINE REGIMES; RVSV ZEBOV, CHAD3 EBO Z-MVA BN-FILO AND ADHU26. ZEBOV-MVA BN FILO
1
3 2
4 3
5 14
6
Effects of mesenchymal stem cell transplantation and DMEM administration in a 3NP rat model of Huntington's disease: morphological and behavioral investigations
1
7
Absence of immune response of the rat striatum after allo/xeno transplantation of mesenchymal stem cells
4
8 80
9
Mesenchymal stem cell transplants reduce behavioral deficits in teh 3-nitropropionic rat model of Huntington's disease
3
10 13
11 135
12
Synergistic interaction between anti-p185HER-2 ricin A chain immunotoxins and radionuclide conjugates for inhibiting growth of ovarian and breast cancer cells that overexpress HER-2.
11
13
The outcome of heregulin-induced activation of ovarian cancer cells depends on the relative levels of HER-2 and HER-3 expression.
58
14 8
15
Cell surface density of p185(c-erbB-2) determines susceptibility to anti-p185(c-erbB-2)-ricin A chain (RTA) immunotoxin therapy alone and in combination with anti-p170(EGFR)-RTA in ovarian cancer cells.
24
16
Heregulin and agonistic anti-p185(c-erbB2) antibodies inhibit proliferation but increase invasiveness of breast cancer cells that overexpress p185(c-erbB2): increased invasiveness may contribute to poor prognosis.
61
17 9
18 74
19 57
20 56

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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