Bruns A. Watts

1.3k total citations
35 papers, 1.1k citations indexed

About

Bruns A. Watts is a scholar working on Molecular Biology, Physiology and Nephrology. According to data from OpenAlex, Bruns A. Watts has authored 35 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 13 papers in Physiology and 7 papers in Nephrology. Recurrent topics in Bruns A. Watts's work include Ion Transport and Channel Regulation (18 papers), Nitric Oxide and Endothelin Effects (13 papers) and Ion channel regulation and function (9 papers). Bruns A. Watts is often cited by papers focused on Ion Transport and Channel Regulation (18 papers), Nitric Oxide and Endothelin Effects (13 papers) and Ion channel regulation and function (9 papers). Bruns A. Watts collaborates with scholars based in United States and Italy. Bruns A. Watts's co-authors include David W. Good, Thampi George, David Sheikh‐Hamad, Robert Safirstein, D. Rouse, Wadi N. Suki, Edward R. Sherwood, John F. Di Mari, Robert E. Beach and Claude R. Benedict and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Bruns A. Watts

35 papers receiving 1.1k citations

Peers

Countries citing papers authored by Bruns A. Watts

Since Specialization

Citations

This map shows the geographic impact of Bruns A. Watts's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bruns A. Watts with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bruns A. Watts more than expected).

Fields of papers citing papers by Bruns A. Watts

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bruns A. Watts. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bruns A. Watts. The network helps show where Bruns A. Watts may publish in the future.

Co-authorship network of co-authors of Bruns A. Watts

This figure shows the co-authorship network connecting the top 25 collaborators of Bruns A. Watts. A scholar is included among the top collaborators of Bruns A. Watts based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bruns A. Watts. Bruns A. Watts is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Monophosphoryl lipid A prevents impairment of medullary thick ascending limb HCO3− absorption and improves plasma HCO3− concentration in septic mice 2018 ·American Journal of Physiology-Renal Physiology ·Bruns A. Watts, Thampi George, Edward R. Sherwood, David W. Good	3
2	Toll-like Receptor 2 Is Required for LPS-induced Toll-like Receptor 4 Signaling and Inhibition of Ion Transport in Renal Thick Ascending Limb 2012 ·Journal of Biological Chemistry ·David W. Good, Thampi George, Bruns A. Watts	62
3	High sodium intake increases HCO₃⁻absorption in medullary thick ascending limb through adaptations in basolateral and apical Na⁺/H⁺exchangers 2011 ·American Journal of Physiology-Renal Physiology ·David W. Good, Thampi George, Bruns A. Watts	9
4	Basolateral LPS inhibits NHE3 and HCO₃⁻ absorption through TLR4/MyD88-dependent ERK activation in medullary thick ascending limb 2011 ·American Journal of Physiology-Cell Physiology ·Bruns A. Watts, Thampi George, Edward R. Sherwood, David W. Good	26
5	Nerve Growth Factor Inhibits Na+/H+ Exchange and HCO3- Absorption through Parallel Phosphatidylinositol 3-Kinase-mTOR and ERK Pathways in Thick Ascending Limb 2008 ·Journal of Biological Chemistry ·David W. Good, Thampi George, Bruns A. Watts	26
6	Aldosterone inhibits apical NHE3 and HCO₃⁻absorption via a nongenomic ERK-dependent pathway in medullary thick ascending limb 2006 ·American Journal of Physiology-Renal Physiology ·Bruns A. Watts, Thampi George, David W. Good	43
7	The Basolateral NHE1 Na+/H+ Exchanger Regulates Transepithelial HCO3− Absorption through Actin Cytoskeleton Remodeling in Renal Thick Ascending Limb 2005 ·Journal of Biological Chemistry ·Bruns A. Watts, Thampi George, David W. Good	40
8	Nongenomic regulation by aldosterone of the epithelial NHE3 Na⁺/H⁺exchanger 2005 ·American Journal of Physiology-Cell Physiology ·David W. Good, Thampi George, Bruns A. Watts	32
9	Transepithelial HCO₃⁻ absorption is defective in renal thick ascending limbs from Na⁺/H⁺ exchanger NHE1 null mutant mice 2004 ·American Journal of Physiology-Renal Physiology ·David W. Good, Bruns A. Watts, Thampi George, Jamie W. Meyer, Gary E. Shull	50
10	An apical K⁺-dependent HCO₃⁻ transport pathway opposes transepithelial HCO₃⁻ absorption in rat medullary thick ascending limb 2004 ·American Journal of Physiology-Renal Physiology ·Bruns A. Watts, David W. Good	8
11	Aldosterone potentiates 1,25-dihydroxyvitamin D₃action in renal thick ascending limb via a nongenomic, ERK-dependent pathway 2003 ·American Journal of Physiology-Cell Physiology ·David W. Good, Thampi George, Bruns A. Watts	23
12	Aldosterone inhibits HCO^-₃absorption via a nongenomic pathway in medullary thick ascending limb 2002 ·American Journal of Physiology-Renal Physiology ·David W. Good, Thampi George, Bruns A. Watts	33
13	Nerve Growth Factor Inhibits HCO3− Absorption in Renal Thick Ascending Limb through Inhibition of Basolateral Membrane Na+/H+ Exchange 1999 ·Journal of Biological Chemistry ·Bruns A. Watts, Thampi George, David W. Good	24
14	Hyposmolality stimulates apical membrane Na+/H+ exchange and HCO3– absorption in renal thick ascending limb 1999 ·Journal of Clinical Investigation ·Bruns A. Watts, David W. Good	47
15	p38 Kinase Activity Is Essential for Osmotic Induction of mRNAs for HSP70 and Transporter for Organic Solute Betaine in Madin-Darby Canine Kidney Cells 1998 ·Journal of Biological Chemistry ·David Sheikh‐Hamad, (unknown), Wadi N. Suki, Robert Safirstein, Bruns A. Watts, D. Rouse	166
16	Effect of long-term hyperosmolality on the Na+/H+ exchanger isoform NHE-3 in LLC-PK1 cells 1998 ·Kidney International ·Manoocher Soleimani, Bruns A. Watts, (unknown), David W. Good	14
17	Basolateral membrane Na+/H+ exchange enhances HCO3- absorption in rat medullary thick ascending limb: evidence for functional coupling between basolateral and apical membrane Na+/H+ exchangers. 1995 ·Proceedings of the National Academy of Sciences ·David W. Good, Thampi George, Bruns A. Watts	42
18	Effects of ammonium on intracellular pH in rat medullary thick ascending limb: mechanisms of apical membrane NH4+ transport. 1994 ·The Journal of General Physiology ·Bruns A. Watts, David W. Good	45
19	Effects of graded oxygen tension on adenosine release by renal medullary and thick ascending limb suspensions 1991 ·Kidney International ·Robert E. Beach, Bruns A. Watts, David W. Good, Claude R. Benedict, Thomas D. DuBose	31
20	Quantitation of adenosine, inosine and hypoxanthine in biological samples by microbore-column isocratic high-performance liquid chromatography 1991 ·Journal of Chromatography A ·(unknown), Bruns A. Watts, Robert E. Beach, Claude R. Benedict	18

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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