Brian D. Crawford

675 total citations
17 papers, 344 citations indexed

About

Brian D. Crawford is a scholar working on Molecular Biology, Health, Toxicology and Mutagenesis and Nutrition and Dietetics. According to data from OpenAlex, Brian D. Crawford has authored 17 papers receiving a total of 344 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 5 papers in Health, Toxicology and Mutagenesis and 5 papers in Nutrition and Dietetics. Recurrent topics in Brian D. Crawford's work include Heavy Metal Exposure and Toxicity (5 papers), Trace Elements in Health (5 papers) and Viral Infectious Diseases and Gene Expression in Insects (3 papers). Brian D. Crawford is often cited by papers focused on Heavy Metal Exposure and Toxicity (5 papers), Trace Elements in Health (5 papers) and Viral Infectious Diseases and Gene Expression in Insects (3 papers). Brian D. Crawford collaborates with scholars based in United States and Spain. Brian D. Crawford's co-authors include Paul O. P. Ts’o, J. Carl Barrett, John Leavitt, Raymond L. Stallings, A. Christine Munk, Jonathan L. Longmire, C.E. Hildebrand, Lynda D. Hester, Deborah Grady and Barbara Griffith and has published in prestigious journals such as Nature, The Lancet and Molecular and Cellular Biology.

In The Last Decade

Brian D. Crawford

15 papers receiving 297 citations

Peers

Brian D. Crawford
Petra Skroch United States
Marco A. Cacciuttolo United States
Shou-Mei T. Chang United States
Shanyun Wu United States
M. Jorge Guimarães United States
R Guthrie United States
Adamson Rh United States
Petra Skroch United States
Brian D. Crawford
Citations per year, relative to Brian D. Crawford Brian D. Crawford (= 1×) peers Petra Skroch

Countries citing papers authored by Brian D. Crawford

Since Specialization
Citations

This map shows the geographic impact of Brian D. Crawford's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Brian D. Crawford with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Brian D. Crawford more than expected).

Fields of papers citing papers by Brian D. Crawford

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Brian D. Crawford. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Brian D. Crawford. The network helps show where Brian D. Crawford may publish in the future.

Co-authorship network of co-authors of Brian D. Crawford

This figure shows the co-authorship network connecting the top 25 collaborators of Brian D. Crawford. A scholar is included among the top collaborators of Brian D. Crawford based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Brian D. Crawford. Brian D. Crawford is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Barrett, J. Carl, et al.. (2024). Correlation of in vitro growth properties and tumorigenicity of Syrian hamster cell lines. PubMed. 39(5). 1504–10.
2.
Kelman, Ariella, et al.. (2018). Continued Access to Investigational Medicinal Products for Clinical Trial Participants—An Industry Approach. Cambridge Quarterly of Healthcare Ethics. 28(1). 124–133. 2 indexed citations
3.
Crawford, Brian D.. (2004). Raymond Federman : les polylogues de l'exil. Critique. n° 683(4). 321–326. 1 indexed citations
4.
Crawford, Brian D.. (2003). Open-access publishing: where is the value?. The Lancet. 362(9395). 1578–1580. 18 indexed citations
5.
Stallings, Raymond L., Brian D. Crawford, Roberta Black, & Esther H. Chang. (1986). Assignment of RAS proto-oncogenes in Chinese hamsters: implications for mammalian gene linkage conservation and neoplasia. Cytogenetic and Genome Research. 43(1-2). 2–5. 7 indexed citations
6.
Stallings, Raymond L., Brian D. Crawford, Robert A. Tobey, Judith G. Tesmer, & C.E. Hildebrand. (1986). 5-Azacytidine-induced conversion to cadmium resistance correlates with early S phase replication of inactive metallothionein genes in synchronized CHO cells. Somatic Cell and Molecular Genetics. 12(5). 423–432. 19 indexed citations
7.
Stallings, Raymond L., A. Christine Munk, Jonathan L. Longmire, et al.. (1985). Oncogenes and linkage groups: Conservation during mammalian chromosome evolution. Chromosoma. 92(2). 156–163. 13 indexed citations
8.
Crawford, Brian D., M. Duane Enger, Barbara Griffith, et al.. (1985). Coordinate amplification of metallothionein I and II genes in cadmium-resistant Chinese hamster cells: implications for mechanisms regulating metallothionein gene expression.. Molecular and Cellular Biology. 5(2). 320–329. 60 indexed citations
9.
Crawford, Brian D., M. Duane Enger, Barbara Griffith, et al.. (1985). Coordinate Amplification of Metallothionein I and II Genes in Cadmium-Resistant Chinese Hamster Cells: Implications for Mechanisms Regulating Metallothionein Gene Expression. Molecular and Cellular Biology. 5(2). 320–329. 13 indexed citations
10.
Stallings, Raymond L., A. Christine Munk, Jonathan L. Longmire, C.E. Hildebrand, & Brian D. Crawford. (1984). Assignment of genes encoding metallothioneins I and II to Chinese hamster chromosome 3: evidence for the role of chromosome rearrangement in gene amplification.. Molecular and Cellular Biology. 4(12). 2932–2936. 15 indexed citations
11.
Stallings, Raymond L., A. Christine Munk, Jonathan L. Longmire, C.E. Hildebrand, & Brian D. Crawford. (1984). Assignment of Genes Encoding Metallothioneins I and II to Chinese Hamster Chromosome 3: Evidence for the Role of Chromosome Rearrangement in Gene Amplification. Molecular and Cellular Biology. 4(12). 2932–2936. 4 indexed citations
12.
Crawford, Brian D., J. Carl Barrett, & Paul O. P. Ts’o. (1983). Neoplastic Conversion of Preneoplastic Syrian Hamster Cells: Rate Estimation by Fluctuation Analysis. Molecular and Cellular Biology. 3(5). 931–945. 16 indexed citations
13.
Tsutsui, Takeki, Brian D. Crawford, Paul O. P. Ts’o, & J. Carl Barrett. (1981). Comparison between mutagenesis in normal and transformed syrian hamster fibroblasts. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 80(2). 357–371. 12 indexed citations
14.
Leavitt, John, J. Carl Barrett, Brian D. Crawford, & Paul O. P. Ts’o. (1978). Butyric acid suppression of the in vitro neoplastic state of Syrian hamster cells. Nature. 271(5642). 262–265. 64 indexed citations
15.
Barrett, J. Carl, Brian D. Crawford, & Paul O. P. Ts’o. (1977). Quantitation of fibrinolytic activity of Syrian hamster fibroblasts using 3H-labeled fibrinogen prepared by reductive alkylation.. PubMed. 37(4). 1182–5. 29 indexed citations
16.
Leavitt, John, Brian D. Crawford, J. Carl Barrett, & Paul O. P. Ts’o. (1977). Regulation of requirements for anchorage-independent growth of Syrian hamster fibroblasts by somatic mutation. Nature. 269(5623). 63–65. 21 indexed citations
17.
Barrett, J. Carl, Brian D. Crawford, Deborah Grady, et al.. (1977). Temporal acquistion of enhanced fibrinolytic activity by syrian hamster embryo cells following treatment with benzo(a)pyrene.. PubMed. 37(10). 3815–23. 50 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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