Brandon Shetuni

453 total citations
10 papers, 318 citations indexed

About

Brandon Shetuni is a scholar working on Surgery, Organic Chemistry and Molecular Biology. According to data from OpenAlex, Brandon Shetuni has authored 10 papers receiving a total of 318 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Surgery, 2 papers in Organic Chemistry and 2 papers in Molecular Biology. Recurrent topics in Brandon Shetuni's work include Eosinophilic Esophagitis (3 papers), Pancreatic and Hepatic Oncology Research (2 papers) and Gastroesophageal reflux and treatments (2 papers). Brandon Shetuni is often cited by papers focused on Eosinophilic Esophagitis (3 papers), Pancreatic and Hepatic Oncology Research (2 papers) and Gastroesophageal reflux and treatments (2 papers). Brandon Shetuni collaborates with scholars based in United States, Netherlands and Japan. Brandon Shetuni's co-authors include Peter J. Kahrilas, Sadis Matalon, Guo‐Yu Yang, John E. Pandolfino, Faraz Bishehsari, Ali Keshavarzian, Leo A. Paquette, Christopher B. Forsyth, Phillip A. Engen and Maliha Shaikh and has published in prestigious journals such as Journal of the American Chemical Society, International Journal of Molecular Sciences and International Journal of Cancer.

In The Last Decade

Brandon Shetuni

10 papers receiving 317 citations

Peers

Brandon Shetuni
A Lambling France
P St-Jean Canada
Ashhar Ali United States
Sherri A. Profitt United States
Brandon Shetuni
Citations per year, relative to Brandon Shetuni Brandon Shetuni (= 1×) peers Lovorka Batelja Vuletić

Countries citing papers authored by Brandon Shetuni

Since Specialization
Citations

This map shows the geographic impact of Brandon Shetuni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Brandon Shetuni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Brandon Shetuni more than expected).

Fields of papers citing papers by Brandon Shetuni

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Brandon Shetuni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Brandon Shetuni. The network helps show where Brandon Shetuni may publish in the future.

Co-authorship network of co-authors of Brandon Shetuni

This figure shows the co-authorship network connecting the top 25 collaborators of Brandon Shetuni. A scholar is included among the top collaborators of Brandon Shetuni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Brandon Shetuni. Brandon Shetuni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Bishehsari, Faraz, Phillip A. Engen, Robin M. Voigt, et al.. (2019). Abnormal Eating Patterns Cause Circadian Disruption and Promote Alcohol-Associated Colon Carcinogenesis. Cellular and Molecular Gastroenterology and Hepatology. 9(2). 219–237. 45 indexed citations
2.
Bishehsari, Faraz, Lijuan Zhang, Usman Barlass, et al.. (2018). KRAS mutation and epithelial–macrophage interplay in pancreatic neoplastic transformation. International Journal of Cancer. 143(8). 1994–2007. 31 indexed citations
3.
Bishehsari, Faraz, Abdulrahman Saadalla, Khashayarsha Khazaie, et al.. (2016). Light/Dark Shifting Promotes Alcohol-Induced Colon Carcinogenesis: Possible Role of Intestinal Inflammatory Milieu and Microbiota. International Journal of Molecular Sciences. 17(12). 2017–2017. 36 indexed citations
4.
Principe, Daniel R., Brian DeCant, Andrew M. Diaz, et al.. (2016). PEDF inhibits pancreatic tumorigenesis by attenuating the fibro-inflammatory reaction. Oncotarget. 7(19). 28218–28234. 25 indexed citations
5.
Matalon, Sadis, et al.. (2015). Histopathologic patterns among achalasia subtypes. Neurogastroenterology & Motility. 28(1). 139–145. 91 indexed citations
6.
Taroni, Jaclyn, Viktor Martyanov, Chiang‐Ching Huang, et al.. (2015). Molecular characterization of systemic sclerosis esophageal pathology identifies inflammatory and proliferative signatures. Arthritis Research & Therapy. 17(1). 194–194. 38 indexed citations
7.
Shetuni, Brandon, et al.. (2014). Histopathologic Patterns Among Achalasia Subtypes. The American Journal of Gastroenterology. 109. S16–S16. 1 indexed citations
8.
Shetuni, Brandon, et al.. (2011). Optimal specimen processing of fine needle aspirates of non‐hodgkin lymphoma. Diagnostic Cytopathology. 40(11). 984–986. 10 indexed citations
9.
Ohkata, Katsuo, Yukiko Tamura, Brandon Shetuni, et al.. (2004). Stereoselectivity Control by Oxaspiro Rings during Diels−Alder Cycloadditions to Cross-Conjugated Cyclohexadienones:  The Syn Oxygen Phenomenon. Journal of the American Chemical Society. 126(51). 16783–16792. 37 indexed citations
10.
Paquette, Leo A., Brandon Shetuni, & Judith C. Gallucci. (2003). π-Facial Stereoselectivity in Diels−Alder Cycloadditions to 1-Oxaspiro[4.5]deca-6,9-dien-8-one. The Strong Directive Effect of Ether Oxygen in a Cross-Conjugated Ketone Setting. Organic Letters. 5(15). 2639–2642. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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