Bradley R. Pearse

442 citations
15 papers · 324 indexed · h-index 8
Topics
Toxin Mechanisms and Immunotoxins (5 papers)Glycosylation and Glycoproteins Research (4 papers)Endoplasmic Reticulum Stress and Disease (4 papers)
Partner nations
United States

In The Last Decade

Bradley R. Pearse

15 papers receiving 320 citations

Peers

Bradley R. Pearse
Comparison fields: 5 of 57
  • Molecular Biology 176
  • Cell Biology 115
  • Immunology 98
  • Immunology and Allergy 84
  • Epidemiology 58
Replace Ralf Sanzenbacher with:
Ralf Sanzenbacher Germany
W.E. Pullman United States
Robert S. Topping United States
Vanja Karamatic Crew United Kingdom
T Irimura Japan
Veronika Lutz Germany
Bruce Yong Japan
Emilia Alina Barbu United States
Weichen Qi China
Hideshi Kumai Japan
Bradley R. Pearse relative to Ralf Sanzenbacher Germany Ralf Sanzenbacher's profile →
Citations per field
00.5×6.7×
Ralf Sanzenbacher · 1×
Citations per year

Countries citing papers authored by Bradley R. Pearse

Since Specialization
Citations

This map shows the geographic impact of Bradley R. Pearse's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bradley R. Pearse with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bradley R. Pearse more than expected).

Fields of papers citing papers by Bradley R. Pearse

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bradley R. Pearse. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bradley R. Pearse. The network helps show where Bradley R. Pearse may publish in the future.

Co-authorship network of co-authors of Bradley R. Pearse

This figure shows the co-authorship network connecting the top 25 collaborators of Bradley R. Pearse. A scholar is included among the top collaborators of Bradley R. Pearse based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bradley R. Pearse. Bradley R. Pearse is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
#WorkIndexed citations
1 1
2 5
3 2
4 2
5 1
6 8
7 3
8 2
9 9
10 28
11 58
12 39
13 31
14 8
15 127

About Bradley R. Pearse

Bradley R. Pearse is a scholar working on Hematology, Immunology and Cell Biology, having authored 15 papers that have together received 324 indexed citations. Recurring topics across this work include Toxin Mechanisms and Immunotoxins (5 papers), Glycosylation and Glycoproteins Research (4 papers) and Endoplasmic Reticulum Stress and Disease (4 papers). The work is most often cited by research in Immunology and Allergy (84 citations), Cell Biology (115 citations) and Immunology (98 citations). Bradley R. Pearse has collaborated with scholars based in United States. Frequent co-authors include Daniel N. Hebert, Shelia M. Violette, Yukako Yokota, Amha Atakilit, Kenneth J. Simon, Paul H. Weinreb, Hisaaki Kawakatsu, Paul Rayhorn, Brian Dolinski and Diane R. Leone. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Cell Biology and Blood.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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