Bradley J. Turner

9.7k total citations
95 papers, 4.0k citations indexed

About

Bradley J. Turner is a scholar working on Neurology, Genetics and Molecular Biology. According to data from OpenAlex, Bradley J. Turner has authored 95 papers receiving a total of 4.0k indexed citations (citations by other indexed papers that have themselves been cited), including 59 papers in Neurology, 41 papers in Genetics and 39 papers in Molecular Biology. Recurrent topics in Bradley J. Turner's work include Amyotrophic Lateral Sclerosis Research (58 papers), Neurogenetic and Muscular Disorders Research (40 papers) and Parkinson's Disease Mechanisms and Treatments (18 papers). Bradley J. Turner is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (58 papers), Neurogenetic and Muscular Disorders Research (40 papers) and Parkinson's Disease Mechanisms and Treatments (18 papers). Bradley J. Turner collaborates with scholars based in Australia, United States and United Kingdom. Bradley J. Turner's co-authors include Kevin Talbot, Julie D. Atkin, Nirma D. Perera, Surindar S. Cheema, Elizabeth C. Lopes, Andrew F. Hill, Justin J. Yerbury, Malcolm Horne, Manal A. Farg and Neil R. Cashman and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Bradley J. Turner

92 papers receiving 3.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Bradley J. Turner Australia 36 1.9k 1.8k 1.2k 574 574 95 4.0k
Cécile Martinat France 21 896 0.5× 1.7k 1.0× 317 0.3× 852 1.5× 288 0.5× 55 2.7k
Shijie Jin Japan 39 547 0.3× 1.8k 1.0× 273 0.2× 699 1.2× 796 1.4× 73 4.6k
Takeshi Yasuda Japan 37 695 0.4× 2.0k 1.1× 337 0.3× 837 1.5× 394 0.7× 164 4.2k
Thomas E. Lloyd United States 37 1.1k 0.6× 2.4k 1.4× 600 0.5× 1.0k 1.8× 420 0.7× 92 4.9k
Michael Bonin Germany 34 677 0.4× 2.3k 1.3× 247 0.2× 828 1.4× 335 0.6× 110 4.2k
Jamal Nasir United Kingdom 27 1.1k 0.6× 2.8k 1.5× 336 0.3× 1.9k 3.2× 190 0.3× 68 3.9k
Tim Chataway Australia 28 1.2k 0.6× 968 0.5× 125 0.1× 656 1.1× 804 1.4× 90 3.1k
Hee Kyung Jin South Korea 35 192 0.1× 1.3k 0.7× 783 0.7× 547 1.0× 917 1.6× 123 3.6k
Katherine J. Ladner United States 22 322 0.2× 2.5k 1.4× 338 0.3× 230 0.4× 1.1k 1.9× 33 3.7k
Juan Arredondo United States 37 260 0.1× 2.0k 1.1× 413 0.4× 408 0.7× 161 0.3× 73 3.3k

Countries citing papers authored by Bradley J. Turner

Since Specialization
Citations

This map shows the geographic impact of Bradley J. Turner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bradley J. Turner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bradley J. Turner more than expected).

Fields of papers citing papers by Bradley J. Turner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bradley J. Turner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bradley J. Turner. The network helps show where Bradley J. Turner may publish in the future.

Co-authorship network of co-authors of Bradley J. Turner

This figure shows the co-authorship network connecting the top 25 collaborators of Bradley J. Turner. A scholar is included among the top collaborators of Bradley J. Turner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bradley J. Turner. Bradley J. Turner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Haidar, Mouna, Doris Tomas, Samuel A. Mills, et al.. (2025). Cortical hyperexcitability drives dying forward amyotrophic lateral sclerosis symptoms and pathology in mice. Progress in Neurobiology. 252. 102809–102809.
4.
Teyssou, Elisa, Delphine Roussel, Nirma D. Perera, et al.. (2022). The Amyotrophic Lateral Sclerosis M114T PFN1 Mutation Deregulates Alternative Autophagy Pathways and Mitochondrial Homeostasis. International Journal of Molecular Sciences. 23(10). 5694–5694. 12 indexed citations
5.
Barton, Samantha K., Chew L. Lau, Doris Tomas, et al.. (2020). Mutant TDP-43 Expression Triggers TDP-43 Pathology and Cell Autonomous Effects on Primary Astrocytes: Implications for Non-cell Autonomous Pathology in ALS. Neurochemical Research. 45(6). 1451–1459. 10 indexed citations
7.
Tan, Xin, Mujun Sun, Rhys D. Brady, et al.. (2018). Transactive Response DNA-Binding Protein 43 Abnormalities after Traumatic Brain Injury. Journal of Neurotrauma. 36(1). 87–99. 24 indexed citations
8.
Silverman, Judith M., Leslie I. Grad, Andrew F. Hill, et al.. (2016). Disease Mechanisms in ALS: Misfolded SOD1 Transferred Through Exosome-Dependent and Exosome-Independent Pathways. Cellular and Molecular Neurobiology. 36(3). 377–381. 79 indexed citations
9.
Kele, Julianna, Christopher R. Bye, Jonathan C. Niclis, et al.. (2014). Diverse Roles for Wnt7a in Ventral Midbrain Neurogenesis and Dopaminergic Axon Morphogenesis. Stem Cells and Development. 23(17). 1991–2003. 25 indexed citations
10.
Winbanks, Catherine E., Justin L. Chen, Hongwei Qian, et al.. (2013). The bone morphogenetic protein axis is a positive regulator of skeletal muscle mass. The Journal of Cell Biology. 203(2). 345–357. 168 indexed citations
11.
Turner, Bradley J., Neza Alfazema, Rebecca K. Sheean, et al.. (2013). Overexpression of survival motor neuron improves neuromuscular function and motor neuron survival in mutant SOD1 mice. Neurobiology of Aging. 35(4). 906–915. 35 indexed citations
12.
Turner, Bradley J. & Julie D. Atkin. (2012). Motor neuron diseases : causes, classification, and treatments. 2 indexed citations
13.
Turner, Bradley J., et al.. (2012). Integral sliding mode autopilot for rocket stabilization with unmatched disturbances. AIAA Guidance, Navigation, and Control Conference. 3 indexed citations
14.
Anderton, Ryan S., L. Price, Bradley J. Turner, et al.. (2012). Co-regulation of survival of motor neuron and Bcl-xL expression: Implications for neuroprotection in spinal muscular atrophy. Neuroscience. 220. 228–236. 14 indexed citations
15.
Phadwal, Kanchan, Bradley J. Turner, Peter L. Oliver, et al.. (2011). HspB8 mutation causing hereditary distal motor neuropathy impairs lysosomal delivery of autophagosomes. Journal of Neurochemistry. 119(6). 1155–1161. 47 indexed citations
16.
Turner, Bradley J., Nick Parkinson, Kay E. Davies, & Kevin Talbot. (2009). Survival motor neuron deficiency enhances progression in an amyotrophic lateral sclerosis mouse model. Neurobiology of Disease. 34(3). 511–517. 56 indexed citations
17.
Turner, Bradley J., Dirk Bäumer, Nick Parkinson, et al.. (2008). TDP-43 expression in mouse models of amyotrophic lateral sclerosis and spinal muscular atrophy. BMC Neuroscience. 9(1). 104–104. 53 indexed citations
18.
Turner, Bradley J., Julie D. Atkin, Manal A. Farg, et al.. (2005). Impaired Extracellular Secretion of Mutant Superoxide Dismutase 1 Associates with Neurotoxicity in Familial Amyotrophic Lateral Sclerosis. Journal of Neuroscience. 25(1). 108–117. 154 indexed citations
19.
Narayan, Kailash, Hui Kong, Elizabeth C. Lopes, et al.. (2004). Chemotherapy Delays Progression of Motor Neuron Disease in the SOD1 G93A Transgenic Mouse. Chemotherapy. 50(3). 138–142. 5 indexed citations
20.
Turner, Bradley J., Irwin K. Cheah, Elizabeth C. Lopes, et al.. (2003). Antisense peptide nucleic acid‐mediated knockdown of the p75 neurotrophin receptor delays motor neuron disease in mutant SOD1 transgenic mice. Journal of Neurochemistry. 87(3). 752–763. 79 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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