Boutaina Daher

544 total citations
8 papers, 380 citations indexed

About

Boutaina Daher is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Cancer Research. According to data from OpenAlex, Boutaina Daher has authored 8 papers receiving a total of 380 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Pulmonary and Respiratory Medicine and 3 papers in Cancer Research. Recurrent topics in Boutaina Daher's work include Epigenetics and DNA Methylation (7 papers), Ferroptosis and cancer prognosis (5 papers) and RNA modifications and cancer (2 papers). Boutaina Daher is often cited by papers focused on Epigenetics and DNA Methylation (7 papers), Ferroptosis and cancer prognosis (5 papers) and RNA modifications and cancer (2 papers). Boutaina Daher collaborates with scholars based in France, Monaco and Canada. Boutaina Daher's co-authors include Jacques Pouysségur, Milica Vučetić, Scott K. Parks, Jérôme Durivault, Yann Cormerais, Éric Tambutté, Ibtissam Marchiq, Maša Ždralević, Shamir Cassim and Maya Saleh and has published in prestigious journals such as Cancer Research, Cell Death and Disease and Cells.

In The Last Decade

Boutaina Daher

8 papers receiving 379 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Boutaina Daher France	6	252	201	190	63	45	8	380
Jonathan Lim Germany	6	315 1.3×	204 1.0×	223 1.2×	54 0.9×	43 1.0×	12	469
Brittany Aguila United States	7	326 1.3×	307 1.5×	150 0.8×	71 1.1×	48 1.1×	12	499
Aimee Falzone United States	9	361 1.4×	200 1.0×	198 1.0×	38 0.6×	41 0.9×	11	493
Adina Brett-Morris United States	4	287 1.1×	280 1.4×	144 0.8×	37 0.6×	54 1.2×	6	441
Jonathan Cimino Belgium	7	274 1.1×	276 1.4×	115 0.6×	67 1.1×	28 0.6×	8	434
Brendah K. Masisi United Kingdom	9	229 0.9×	181 0.9×	68 0.4×	69 1.1×	48 1.1×	12	324
Ming-zhe Ma China	9	261 1.0×	221 1.1×	118 0.6×	73 1.2×	29 0.6×	9	414
Mihir B. Doshi United States	7	222 0.9×	132 0.7×	160 0.8×	60 1.0×	14 0.3×	8	395
Ana M. Metelo Portugal	7	273 1.1×	259 1.3×	73 0.4×	58 0.9×	29 0.6×	10	419
Haixiang Pei China	9	325 1.3×	171 0.9×	169 0.9×	105 1.7×	32 0.7×	11	479

Countries citing papers authored by Boutaina Daher

Since Specialization

Citations

This map shows the geographic impact of Boutaina Daher's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Boutaina Daher with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Boutaina Daher more than expected).

Fields of papers citing papers by Boutaina Daher

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Boutaina Daher. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Boutaina Daher. The network helps show where Boutaina Daher may publish in the future.

Co-authorship network of co-authors of Boutaina Daher

This figure shows the co-authorship network connecting the top 25 collaborators of Boutaina Daher. A scholar is included among the top collaborators of Boutaina Daher based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Boutaina Daher. Boutaina Daher is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Daher, Boutaina, et al.. (2024). Revealing the Ferroptotic Phenotype of Medulloblastoma. Journal of Visualized Experiments. 1 indexed citations

2.

Daher, Boutaina, et al.. (2022). Genetic Disruption of the γ-Glutamylcysteine Ligase in PDAC Cells Induces Ferroptosis-Independent Cell Death In Vitro without Affecting In Vivo Tumor Growth. Cancers. 14(13). 3154–3154. 5 indexed citations

3.

Daher, Boutaina, et al.. (2021). The Metabolic Control of Myeloid Cells in the Tumor Microenvironment. Cells. 10(11). 2960–2960. 6 indexed citations

4.

Daher, Boutaina, Scott K. Parks, Yann Cormerais, et al.. (2021). A Cystine-Cysteine Intercellular Shuttle Prevents Ferroptosis in xCTKO Pancreatic Ductal Adenocarcinoma Cells. Cancers. 13(6). 1434–1434. 18 indexed citations

5.

Vučetić, Milica, et al.. (2020). Together we stand, apart we fall: how cell-to-cell contact/interplay provides resistance to ferroptosis. Cell Death and Disease. 11(9). 789–789. 27 indexed citations

6.

Daher, Boutaina, Milica Vučetić, & Jacques Pouysségur. (2020). Cysteine Depletion, a Key Action to Challenge Cancer Cells to Ferroptotic Cell Death. Frontiers in Oncology. 10. 723–723. 88 indexed citations

7.

Daher, Boutaina, Scott K. Parks, Jérôme Durivault, et al.. (2019). Genetic Ablation of the Cystine Transporter xCT in PDAC Cells Inhibits mTORC1, Growth, Survival, and Tumor Formation via Nutrient and Oxidative Stresses. Cancer Research. 79(15). 3877–3890. 160 indexed citations

8.

Ždralević, Maša, Milica Vučetić, Boutaina Daher, et al.. (2017). Disrupting the ‘Warburg effect’ re-routes cancer cells to OXPHOS offering a vulnerability point via ‘ferroptosis’-induced cell death. Advances in Biological Regulation. 68. 55–63. 75 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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