Boris Pantic
Impact in
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- Mitochondrial Function and Pathology
- ATP Synthase and ATPases Research
- Muscle Physiology and Disorders
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- Genetic Neurodegenerative Diseases
Papers in
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- Mitochondrial Function and Pathology 8
- Muscle Physiology and Disorders 3
- Signaling Pathways in Disease 2
- Cell death mechanisms and regulation 1
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- Genetic Neurodegenerative Diseases 5
- Co-authors
- Andrea Rasola (3 shared papers)Paolo Bernardi (3 shared papers)Marco Sciacovelli (2 shared papers)W S Brusilow (1 shared paper)Federica Chiara (1 shared paper)Lodovica Vergani (5 shared papers)Adriana Malena (4 shared papers)Sergio Salvatori (2 shared papers)
- Journals
- Cell Death and Disease (2 papers)Autophagy (1 paper)Human Molecular Genetics (1 paper)Nature Communications (1 paper)FEBS Letters (1 paper)
- Partner nations
- ItalyUnited KingdomUnited States
In The Last Decade
Boris Pantic
10 papers receiving 564 citations
Peers
Comparison fields: 5 of 81
- Molecular Biology 467
- Cellular and Molecular Neuroscience 120
- Clinical Biochemistry 35
- Cancer Research 65
- Aging 5
Countries citing papers authored by Boris Pantic
This map shows the geographic impact of Boris Pantic's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Boris Pantic with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Boris Pantic more than expected).
Fields of papers citing papers by Boris Pantic
This network shows the impact of papers produced by Boris Pantic. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Boris Pantic. The network helps show where Boris Pantic may publish in the future.
Co-authors
The 25 scholars most cited alongside Boris Pantic, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 199 | |
| 2 | 2010 | 150 | |
| 3 | 2013 | 55 | |
| 4 | 2013 | 33 | |
| 5 | 2016 | 26 | |
| 6 | 2016 | 25 | |
| 7 | 2013 | 25 | |
| 8 | 2017 | 22 | |
| 9 | 2021 | 17 | |
| 10 | 2018 | 17 |
About Boris Pantic
Boris Pantic is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Cell Biology, Epidemiology and Cardiology and Cardiovascular Medicine, having authored 10 papers that have together received 569 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (8 papers), Genetic Neurodegenerative Diseases (5 papers), Muscle Physiology and Disorders (3 papers), Signaling Pathways in Disease (2 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Autophagy in Disease and Therapy (2 papers), Muscle metabolism and nutrition (1 paper) and Cell death mechanisms and regulation (1 paper). The work is most often cited by research in Molecular Biology (467 citations), Cellular and Molecular Neuroscience (120 citations), Clinical Biochemistry (35 citations), Cancer Research (65 citations) and Aging (5 citations). Boris Pantic has collaborated with scholars based in Italy, United Kingdom and United States. Frequent co-authors include Andrea Rasola, Paolo Bernardi, Marco Sciacovelli, W S Brusilow, Federica Chiara, Lodovica Vergani, Adriana Malena, Sergio Salvatori, Oriano Marin and Annalisa Botta. Their work appears in journals such as Cell Death and Disease, Autophagy, Human Molecular Genetics, Nature Communications and FEBS Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.