Bin Gotoh

3.3k total citations
56 papers, 2.8k citations indexed

About

Bin Gotoh is a scholar working on Epidemiology, Infectious Diseases and Immunology. According to data from OpenAlex, Bin Gotoh has authored 56 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Epidemiology, 23 papers in Infectious Diseases and 21 papers in Immunology. Recurrent topics in Bin Gotoh's work include Virology and Viral Diseases (24 papers), interferon and immune responses (18 papers) and Viral Infections and Vectors (16 papers). Bin Gotoh is often cited by papers focused on Virology and Viral Diseases (24 papers), interferon and immune responses (18 papers) and Viral Infections and Vectors (16 papers). Bin Gotoh collaborates with scholars based in Japan, United States and Czechia. Bin Gotoh's co-authors include Takayuki Komatsu, Kenji Takeuchi, Junko Yokoo, Michinari Hamaguchi, Tetsuya Toyoda, Yoshiyuki Nagai, Noel M. Inocencio, Takemasa Sakaguchi, Yasuo Ohnishi and Yoshinori Kitagawa and has published in prestigious journals such as The EMBO Journal, Journal of Virology and FEBS Letters.

In The Last Decade

Bin Gotoh

56 papers receiving 2.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Bin Gotoh Japan 28 1.7k 965 856 729 562 56 2.8k
Dominique Garcin Switzerland 38 2.2k 1.3× 1.8k 1.8× 1.6k 1.9× 1.0k 1.4× 703 1.3× 72 4.3k
D. F. Young United Kingdom 26 1.7k 1.0× 1.1k 1.1× 1.8k 2.0× 689 0.9× 309 0.5× 50 3.3k
Andrea Maisner Germany 31 1.7k 1.0× 964 1.0× 336 0.4× 558 0.8× 305 0.5× 66 2.4k
Ali Saı̈b France 33 968 0.6× 680 0.7× 1.0k 1.2× 1.4k 2.0× 166 0.3× 63 3.4k
Maxine L. Linial United States 34 1.1k 0.7× 559 0.6× 424 0.5× 1.5k 2.1× 539 1.0× 79 3.5k
Richard D. Barry Australia 28 1.0k 0.6× 681 0.7× 480 0.6× 802 1.1× 245 0.4× 67 2.5k
Machiko Nishio Japan 31 1.4k 0.8× 875 0.9× 432 0.5× 588 0.8× 186 0.3× 114 2.5k
Tim Skern Austria 33 712 0.4× 1.1k 1.1× 401 0.5× 2.2k 3.0× 422 0.8× 102 4.1k
Martin Löchelt Germany 33 1.4k 0.8× 581 0.6× 482 0.6× 817 1.1× 320 0.6× 99 3.0k
Katherine R. Spindler United States 26 740 0.4× 755 0.8× 402 0.5× 1.3k 1.8× 268 0.5× 65 3.1k

Countries citing papers authored by Bin Gotoh

Since Specialization
Citations

This map shows the geographic impact of Bin Gotoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bin Gotoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bin Gotoh more than expected).

Fields of papers citing papers by Bin Gotoh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bin Gotoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bin Gotoh. The network helps show where Bin Gotoh may publish in the future.

Co-authorship network of co-authors of Bin Gotoh

This figure shows the co-authorship network connecting the top 25 collaborators of Bin Gotoh. A scholar is included among the top collaborators of Bin Gotoh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bin Gotoh. Bin Gotoh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tanaka, Yukië, Naoko Morita, Yoshinori Kitagawa, Bin Gotoh, & Takayuki Komatsu. (2022). Human metapneumovirus M2-2 protein inhibits RIG-I signaling by preventing TRIM25-mediated RIG-I ubiquitination. Frontiers in Immunology. 13. 970750–970750. 12 indexed citations
2.
Gotoh, Bin. (2020). Transmission of Zika Virus. 1–2. 1 indexed citations
3.
4.
5.
Zhou, Min, et al.. (2012). Expeditious neutralization assay for human metapneumovirus based on a recombinant virus expressing Renilla luciferase. Journal of Clinical Virology. 56(1). 31–36. 9 indexed citations
6.
Kitagawa, Yoshinori, Min Zhou, Takayuki Komatsu, et al.. (2009). Human metapneumovirus M2-2 protein inhibits viral transcription and replication. Microbes and Infection. 12(2). 135–145. 24 indexed citations
7.
Komatsu, Takayuki, Kenji Takeuchi, & Bin Gotoh. (2007). Bovine parainfluenza virus type 3 accessory proteins that suppress beta interferon production. Microbes and Infection. 9(8). 954–962. 40 indexed citations
8.
Hiramatsu, Akira, Bin Gotoh, Yutaka Fujii, et al.. (2006). Suppression of interferon-related promoter activation by hepatitis C virus proteins expressed in cultured cells.. PubMed. 55(3). 71–7. 1 indexed citations
9.
Gotoh, Bin, Kenji Takeuchi, & Takayuki Komatsu. (2004). Inhibition of the gamma interferon response by a Sendai virus C protein mutant with no STAT1‐binding ability. FEBS Letters. 567(2-3). 291–296. 5 indexed citations
10.
Komatsu, Takayuki, Kenji Takeuchi, Junko Yokoo, & Bin Gotoh. (2004). C and V proteins of Sendai virus target signaling pathways leading to IRF-3 activation for the negative regulation of interferon-β production. Virology. 325(1). 137–148. 78 indexed citations
11.
Komatsu, Takayuki, Kenji Takeuchi, Junko Yokoo, & Bin Gotoh. (2002). Sendai virus C protein impairs both phosphorylation and dephosphorylation processes of Stat1. FEBS Letters. 511(1-3). 139–144. 57 indexed citations
12.
Gotoh, Bin, Takayuki Komatsu, Kenji Takeuchi, & Junko Yokoo. (2002). Paramyxovirus strategies for evading the interferon response. Reviews in Medical Virology. 12(6). 337–357. 112 indexed citations
13.
Zhong, Yu, Bin Gotoh, Michinari Hamaguchi, & Yoshiyuki Nagai. (1995). Antiviral action of interferon-β on Newcastle disease virus: selectivity to the hemagglutinin-neuraminidase gene expression. Medical Microbiology and Immunology. 184(1). 45–52. 4 indexed citations
14.
Gotoh, Bin, et al.. (1992). Isolation of factor Xa from chick embryo as the amniotic endoprotease responsible for paramyxovirus activation. FEBS Letters. 296(3). 274–278. 22 indexed citations
15.
Suzuki, Harukazu, Yumiko Hayashi, J Asaka, et al.. (1991). Primary structure of the virus activating protease: from chick embryo Its identity with the blood clotting factor Xa. FEBS Letters. 283(2). 281–285. 23 indexed citations
16.
Inocencio, Noel M., Bin Gotoh, Tetsuya Toyoda, Chieko Kitada, & Yoshinori Nagai. (1990). Evaluation of the antiviral effect of synthetic oligopeptides whose sequences are derived from paramyxovirus F1 N termini. Medical Microbiology and Immunology. 179(2). 87–94. 1 indexed citations
17.
Saso, Hitomi, Kaoru Shimokata, Tetsuya Toyoda, Bin Gotoh, & Yoshinori Nagai. (1990). Growth restriction of negative-strand-RNA viruses in a rat 3Y1 cell line. Medical Microbiology and Immunology. 179(1). 31–41. 1 indexed citations
18.
Sakaguchi, Takemasa, Tetsuya Toyoda, Bin Gotoh, et al.. (1989). Newcastle disease virus evolution. Virology. 169(2). 260–272. 139 indexed citations
19.
Toyoda, Tetsuya, et al.. (1989). Newcastle disease virus evolution. Virology. 169(2). 273–282. 146 indexed citations
20.
Gotoh, Bin, Takemasa Sakaguchi, Kazuo Nishikawa, et al.. (1988). Structural features unique to each of the three antigenic sites on the hemagglutinin-neuraminidase protein of newcastle disease virus. Virology. 163(1). 174–182. 45 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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