Norihisa Matsuyoshi

4.9k total citations · 2 hit papers
43 papers, 4.0k citations indexed

About

Norihisa Matsuyoshi is a scholar working on Molecular Biology, Cell Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Norihisa Matsuyoshi has authored 43 papers receiving a total of 4.0k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Molecular Biology, 14 papers in Cell Biology and 9 papers in Pathology and Forensic Medicine. Recurrent topics in Norihisa Matsuyoshi's work include Wnt/β-catenin signaling in development and cancer (22 papers), Skin and Cellular Biology Research (10 papers) and Kruppel-like factors research (8 papers). Norihisa Matsuyoshi is often cited by papers focused on Wnt/β-catenin signaling in development and cancer (22 papers), Skin and Cellular Biology Research (10 papers) and Kruppel-like factors research (8 papers). Norihisa Matsuyoshi collaborates with scholars based in Japan, United States and Canada. Norihisa Matsuyoshi's co-authors include Masatoshi Takeichi, Shin‐Ichi Nishikawa, Satomi Nishikawa, Masanori Hirashima, Michinari Hamaguchi, Hiroaki Kodama, John R. Stanley, Akira Nagafuchi, Shöichiro Tsukita and S. Taniguchi and has published in prestigious journals such as Nature Medicine, The Journal of Cell Biology and The EMBO Journal.

In The Last Decade

Norihisa Matsuyoshi

43 papers receiving 3.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Progressive lineage analysis by cell sorting and culture identiﬁes FLK1+VEcadherin+ cells at a diverging point of endothelial and hemopoietic lineages

1998 663 citations Shin‐Ichi Nishikawa, Satomi Nishikawa et al. profile →
Expression of E-cadherin cell adhesion molecules in human breast cancer tissues and its relationship to metastasis.

1993 566 citations Hiroshi Oka, Hironori Shiozaki et al. PubMed profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Norihisa Matsuyoshi Japan	26	2.9k	1.2k	540	421	410	43	4.0k
Pamela J. Jensen United States	35	1.6k 0.6×	1.2k 1.0×	461 0.9×	469 1.1×	344 0.8×	73	4.0k
Tobias Gedde‐Dahl Norway	39	1.8k 0.6×	1.3k 1.1×	770 1.4×	1.2k 2.9×	223 0.5×	225	5.3k
Arthur Vogel United States	23	1.3k 0.5×	752 0.6×	685 1.3×	481 1.1×	249 0.6×	44	3.1k
Neil A. Hotchin United Kingdom	26	1.7k 0.6×	1.3k 1.1×	511 0.9×	485 1.2×	789 1.9×	42	3.2k
J J Cassiman Belgium	34	1.9k 0.7×	1.5k 1.3×	248 0.5×	204 0.5×	560 1.4×	66	3.4k
V.E. Koteliansky Russia	35	2.1k 0.7×	1.0k 0.9×	598 1.1×	375 0.9×	1.3k 3.2×	65	4.4k
S Munemitsu United States	24	5.1k 1.8×	908 0.8×	1.3k 2.3×	797 1.9×	316 0.8×	28	7.1k
Richard C. Bates United States	32	2.2k 0.7×	553 0.5×	1.6k 2.9×	568 1.3×	739 1.8×	51	4.3k
Patricia Simon‐Assmann France	41	2.3k 0.8×	912 0.8×	1.3k 2.4×	426 1.0×	1.1k 2.7×	95	4.9k
Sela Cheifetz United States	34	5.0k 1.7×	850 0.7×	1.3k 2.4×	352 0.8×	921 2.2×	48	6.7k

Countries citing papers authored by Norihisa Matsuyoshi

Since Specialization

Citations

This map shows the geographic impact of Norihisa Matsuyoshi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Norihisa Matsuyoshi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Norihisa Matsuyoshi more than expected).

Fields of papers citing papers by Norihisa Matsuyoshi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Norihisa Matsuyoshi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Norihisa Matsuyoshi. The network helps show where Norihisa Matsuyoshi may publish in the future.

Co-authorship network of co-authors of Norihisa Matsuyoshi

This figure shows the co-authorship network connecting the top 25 collaborators of Norihisa Matsuyoshi. A scholar is included among the top collaborators of Norihisa Matsuyoshi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Norihisa Matsuyoshi. Norihisa Matsuyoshi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Matsushima, Hironori, A. Utani, Hideharu Endo, et al.. (2003). The expression of nectin-1alpha in normal human skin and various skin tumours. British Journal of Dermatology. 148(4). 755–762. 35 indexed citations

Kore-eda, S., Norihisa Matsuyoshi, Masamichi Ueda, et al.. (2002). A Monoclonal Antibody Established from the Immunization of Basal Cell Carcinoma (BCC) Tissues Reacts to the Intercellular Space of BCC and Hair Follicles. The Journal of Dermatology. 29(11). 718–725. 1 indexed citations

Hanakawa, Yasushi, Norihisa Matsuyoshi, & John R. Stanley. (2002). Expression of Desmoglein 1 Compensates for Genetic Loss of Desmoglein 3 in Keratinocyte Adhesion. Journal of Investigative Dermatology. 119(1). 27–31. 59 indexed citations

Takeuchi, Tamotsu, Sheng‐Ben Liang, Norihisa Matsuyoshi, et al.. (2002). Loss of T-Cadherin (CDH13, H-Cadherin) Expression in Cutaneous Squamous Cell Carcinoma. Laboratory Investigation. 82(8). 1023–1029. 60 indexed citations

Amagai, Masayuki, et al.. (2000). Toxin in bullous impetigo and staphylococcal scalded-skin syndrome targets desmoglein 1. Nature Medicine. 6(11). 1275–1277. 332 indexed citations

Matsui, Miho, et al.. (2000). Cutaneous tuberculosis and pyoderma gangrenosum. International Journal of Dermatology. 39(1). 38–40. 7 indexed citations

Hirashima, Masanori, Hiroshi Kataoka, Satomi Nishikawa, Norihisa Matsuyoshi, & Shin‐Ichi Nishikawa. (1999). Maturation of Embryonic Stem Cells Into Endothelial Cells in an In Vitro Model of Vasculogenesis. Blood. 93(4). 1253–1263. 232 indexed citations

Matsuyoshi, Norihisa, Ken‐ichi Toda, & Sadao Imamura. (1998). N-cadherin expression in human adult T-cell leukemia cell line. Archives of Dermatological Research. 290(4). 223–225. 6 indexed citations

Furukawa, Fukumi, Kimio Fujii, Yuji Horiguchi, et al.. (1997). Roles of E- and P-cadherin in the human skin. Microscopy Research and Technique. 38(4). 343–352. 69 indexed citations

10.

Matsuyoshi, Norihisa, Ken‐ichi Toda, Y. Horiguchi, et al.. (1997). In vivo evidence of the critical role of cadherin-5 in murine vascular integrity.. PubMed. 109(4). 362–71. 75 indexed citations

11.

Matsuyoshi, Norihisa & Sadao Imamura. (1997). Multiple Cadherins Are Expressed in Human Fibroblasts. Biochemical and Biophysical Research Communications. 235(2). 355–358. 48 indexed citations

12.

Matsuyoshi, Norihisa, Toshihiro Tanaka, Ken‐ichi Toda, & Sadao Imamura. (1997). Identification of Novel Cadherins Expressed in Human Melanoma Cells. Journal of Investigative Dermatology. 108(6). 908–913. 29 indexed citations

13.

Morita, Ken, Chikako Nishigori, M.S. Sasaki, et al.. (1997). Werner's syndrome - chromosome analyses of cultured fibroblasts and mitogen-stimulated lymphocytes. British Journal of Dermatology. 136(4). 620–623. 3 indexed citations

14.

Tanaka, Toshihiro, et al.. (1996). Keratin 9 point mutation in the pedigree of epidermolytic hereditary palmoplantar keratoderma perturbs keratin intermediate filament network formation. FEBS Letters. 386(2-3). 149–155. 29 indexed citations

15.

Ikai, K., Masayuki Yamamoto, Norihisa Matsuyoshi, & Masanori Fukushima. (1995). Effect of a cytotoxic prostaglandin, Δ12-prostaglandin J2 on E-cadherin expression in transformed epidermal cells in culture. Prostaglandins Leukotrienes and Essential Fatty Acids. 52(5). 303–307. 4 indexed citations

16.

Tanaka, Toshihiro, Norihisa Matsuyoshi, Fukumi Furukawa, & S. Imamura. (1994). Production of Monoclonal Antibodies against the N-Terminal Noncollagenous Domain of Type VII Collagen Encoded by Epidermolysis bullosa acquisita Antigen cDNA. Dermatology. 189(1). 42–45. 5 indexed citations

17.

Hamaguchi, Michinari, Norihisa Matsuyoshi, Yasuo Ohnishi, et al.. (1993). p60v-src causes tyrosine phosphorylation and inactivation of the N-cadherin-catenin cell adhesion system.. The EMBO Journal. 12(1). 307–314. 370 indexed citations

18.

Oka, Hiroshi, Hironori Shiozaki, Kenji Kobayashi, et al.. (1993). Expression of E-cadherin cell adhesion molecules in human breast cancer tissues and its relationship to metastasis.. PubMed. 53(7). 1696–701. 566 indexed citations breakdown →

19.

Furukawa, Fukumi, Kouichi Ikai, Norihisa Matsuyoshi, Kyoko Shimizu, & Sadao Imamura. (1993). Relationship Between Heat Shock Protein Induction and the Binding of Antibodies to the Extractable Nuclear Antigens on Cultured Human Keratinocytes. Journal of Investigative Dermatology. 101(2). 191–195. 29 indexed citations

20.

Matsuyoshi, Norihisa, Y. Horiguchi, Toshiyuki Tanaka, et al.. (1989). Lymphomatoid papulosis: ultrastructural, immunohistochemical and gene analytical studies. British Journal of Dermatology. 121(3). 381–389. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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