Biljana Čuljković

2.2k citations
29 papers · 1.5k indexed · h-index 13
Topics
Genetic Neurodegenerative Diseases (12 papers)PI3K/AKT/mTOR signaling in cancer (9 papers)Mitochondrial Function and Pathology (8 papers)

In The Last Decade

Biljana Čuljković

28 papers receiving 1.5k citations

Peers

Biljana Čuljković
Comparison fields: 5 of 79
  • Molecular Biology 1.3k
  • Immunology 170
  • Genetics 156
  • Oncology 135
  • Hematology 130
Replace Takeshi Ueda with:
Takeshi Ueda Japan
Éric Milot Canada
Konstantina Alexandropoulos United States
Alan J. Kinniburgh United States
Michele Ceribelli United States
Laurence Covassin United States
Venugopalan Cheriyath United States
A D Moulton United States
Lindsey Jones United States
Katia Fecchi Italy
Biljana Čuljković relative to Takeshi Ueda Japan Takeshi Ueda's profile →
Citations per field
00.5×1.5×
Takeshi Ueda · 1×
Citations per year

Countries citing papers authored by Biljana Čuljković

Since Specialization
Citations

This map shows the geographic impact of Biljana Čuljković's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Biljana Čuljković with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Biljana Čuljković more than expected).

Fields of papers citing papers by Biljana Čuljković

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Biljana Čuljković. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Biljana Čuljković. The network helps show where Biljana Čuljković may publish in the future.

Co-authorship network of co-authors of Biljana Čuljković

This figure shows the co-authorship network connecting the top 25 collaborators of Biljana Čuljković. A scholar is included among the top collaborators of Biljana Čuljković based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Biljana Čuljković. Biljana Čuljković is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 1
2 1
3 52
4 45
5 81
6 114
7 220
8 155
9 81
10 18
11 248
12 2
13 4
14 8
15 12
16 16
17 4
18 10
19 7
20 3

About Biljana Čuljković

Biljana Čuljković is a scholar working on Cellular and Molecular Neuroscience, Genetics and Molecular Biology, having authored 29 papers that have together received 1.5k indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (12 papers), PI3K/AKT/mTOR signaling in cancer (9 papers) and Mitochondrial Function and Pathology (8 papers). The work is most often cited by research in Molecular Biology (1.3k citations), Genetics (156 citations) and Hematology (130 citations). Biljana Čuljković has collaborated with scholars based in Serbia, Canada and United States. Frequent co-authors include Katherine L. B. Borden, Ivan Topisirović, Melisa Ruiz‐Gutierrez, Lucy Skrabanek, Ling Shao, Alex Kentsis, Abdellatif Amri, Keith Tan, Caroline Rousseau and Brian Leber. Their work appears in journals such as Proceedings of the National Academy of Sciences, The Journal of Cell Biology and Blood.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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