Bilal Unal

729 citations
9 papers · 496 indexed · h-index 6

Impact in

Papers in

    • Cancer-related molecular mechanisms research 2
    • MicroRNA in disease regulation 1
    • Endoplasmic Reticulum Stress and Disease 2

Bilal Unal

7 papers receiving 492 citations

Peers

Bilal Unal
Comparison fields: 5 of 72
  • Aging 18
  • Cancer Research 149
  • Physiology 153
  • Biotechnology 49
  • Molecular Biology 333
Replace Hongyu Deng with:
Hongyu Deng China
Bong-Kyeong Oh South Korea
Abdul M. Mondal United States
Yi Young Choi South Korea
Bárbara A. Santana-Lemos Brazil
M Yamada Japan
Frauke Goeman Italy
Anderson R. Frank United States
Xianli He China
Charles Dudognon France
Bilal Unal relative to Hongyu Deng China Hongyu Deng's profile →
Citations per field
00.5×
Hongyu Deng · 1×
Citations per year

Countries citing papers authored by Bilal Unal

Since Specialization
Citations

This map shows the geographic impact of Bilal Unal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bilal Unal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bilal Unal more than expected).

Fields of papers citing papers by Bilal Unal

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bilal Unal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bilal Unal. The network helps show where Bilal Unal may publish in the future.

Co-authors

The 25 scholars most cited alongside Bilal Unal, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Bilal Unal Line = papers co-authored together Bilal Unal links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 2016156
2 2017138
3 2016121
4 202048
5 202420
6 202410
7
STAMP2 suppresses autophagy in prostate cancer cells by modulating the integrated stress response pathway.
20223
8 20240
9 20250

About Bilal Unal

Bilal Unal is a scholar working on Cancer Research, Cell Biology, Pulmonary and Respiratory Medicine, Physiology and Molecular Biology, having authored 9 papers that have together received 496 indexed citations. Recurring topics across this work include Telomeres, Telomerase, and Senescence (2 papers), Prostate Cancer Treatment and Research (2 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Cancer-related molecular mechanisms research (2 papers), MicroRNA in disease regulation (1 paper), Genomics and Chromatin Dynamics (1 paper), RNA Interference and Gene Delivery (1 paper) and Bladder and Urothelial Cancer Treatments (1 paper). The work is most often cited by research in Aging (18 citations), Cancer Research (149 citations), Physiology (153 citations), Biotechnology (49 citations) and Molecular Biology (333 citations). Bilal Unal has collaborated with scholars based in Norway, Singapore and Australia. Frequent co-authors include Vinay Tergaonkar, Semih Can Akıncılar, Chen Li Chew, Stephanie A. Conos, Melissa J. Fullwood, Ekta Khattar, Taichi Noda, Masahito Ikawa, Lele Wu and Omer F. Kuzu. Their work appears in journals such as Biomarker Research, Trends in Molecular Medicine, Nature Communications, Gut and Theranostics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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