Bente Lowin

2.2k total citations · 1 hit paper
10 papers, 1.9k citations indexed

About

Bente Lowin is a scholar working on Immunology, Molecular Biology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Bente Lowin has authored 10 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology, 4 papers in Molecular Biology and 3 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Bente Lowin's work include Cell death mechanisms and regulation (4 papers), Acute Lymphoblastic Leukemia research (3 papers) and Immune Cell Function and Interaction (3 papers). Bente Lowin is often cited by papers focused on Cell death mechanisms and regulation (4 papers), Acute Lymphoblastic Leukemia research (3 papers) and Immune Cell Function and Interaction (3 papers). Bente Lowin collaborates with scholars based in Switzerland. Bente Lowin's co-authors include Jürg Tschopp, Chantal Mattmann, Michael Hahne, Manuel C. Peitsch, Marianne Braun, Hans Acha‐Orbea, Lars E. French, A. Schmidt, Gerd Schmitz and Dieter E. Jenne and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Bente Lowin

10 papers receiving 1.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Cytolytic T-cell cytotoxicity is mediated through perforin and Fas lytic pathways

1994 920 citations Bente Lowin, Michael Hahne et al. Nature profile →

Peers

Countries citing papers authored by Bente Lowin

Since Specialization

Citations

This map shows the geographic impact of Bente Lowin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bente Lowin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bente Lowin more than expected).

Fields of papers citing papers by Bente Lowin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bente Lowin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bente Lowin. The network helps show where Bente Lowin may publish in the future.

Co-authorship network of co-authors of Bente Lowin

This figure shows the co-authorship network connecting the top 25 collaborators of Bente Lowin. A scholar is included among the top collaborators of Bente Lowin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bente Lowin. Bente Lowin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

#	Work	Indexed citations
1	Cytotoxic T cells deficient in both functional fas ligand and perforin show residual cytolytic activity yet lose their capacity to induce lethal acute graft-versus-host disease. 1996 ·The Journal of Experimental Medicine ·Marianne Braun, Bente Lowin, Lars E. French, Hans Acha‐Orbea, Jürg Tschopp	260
2	Comparison of Fas(Apo-1/CD95)- and perforin-mediated cytotoxicity in primary T lymphocytes 1996 ·International Immunology ·Bente Lowin, (unknown), Michael Hahne, Jürg Tschopp	58
3	Expression of the CTL-associated protein TIA-1 during murine embryogenesis 1996 ·The Journal of Immunology ·Bente Lowin, L. R. French, Jean‐Claude Martinou, J Tschopp	9
4	Regulation of Fas(Apo‐1/CD95)‐ and perforinmediated lytic pathways of primary cytotoxic T lymphocytes by the protooncogene bcl‐2 1995 ·European Journal of Immunology ·Michael Schröter, Bente Lowin, Christoph Borner, Jürg Tschopp	49
5	Perforin and Granzymes: Crucial Effector Molecules in Cytolytic T Lymphocyte and Natural Killer Cell-Mediated Cytotoxicity 1995 ·Current topics in microbiology and immunology ·Bente Lowin, Manuel C. Peitsch, Jürg Tschopp	121
6	Interaction of peptides derived from the Fas ligand with the Fyn‐SH3 domain 1995 ·FEBS Letters ·(unknown), Bente Lowin, Manuel C. Peitsch, Karin Becker, Jürg Tschopp	48
7	Cytolytic T-cell cytotoxicity is mediated through perforin and Fas lytic pathways breakdown → 1994 ·Nature ·Bente Lowin, Michael Hahne, Chantal Mattmann, Jürg Tschopp	920
8	A null mutation in the perforin gene impairs cytolytic T lymphocyte- and natural killer cell-mediated cytotoxicity. 1994 ·Proceedings of the National Academy of Sciences ·Bente Lowin, (unknown), A. Schmidt, Jürg Tschopp	175
9	Perforin and its role in T lymphocyte-mediated cytolysis 1992 ·Cellular and Molecular Life Sciences ·Bente Lowin, Olivier Krähenbühl, Christoph Müller, Marc Dupuis, Jürg Tschopp	11
10	Clusterin (complement lysis inhibitor) forms a high density lipoprotein complex with apolipoprotein A-I in human plasma 1991 ·Journal of Biological Chemistry ·Dieter E. Jenne, Bente Lowin, Manuel C. Peitsch, Andreas Böttcher, Gerd Schmitz, Jürg Tschopp	239

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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