Benjamin Van Camp
About
In The Last Decade
Benjamin Van Camp
62 papers receiving 1.9k citations
Peers
Comparison fields: 5 of 94
- Hematology 1.1k
- Molecular Biology 743
- Oncology 657
- Immunology 256
- Genetics 250
Countries citing papers authored by Benjamin Van Camp
This map shows the geographic impact of Benjamin Van Camp's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin Van Camp with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin Van Camp more than expected).
Fields of papers citing papers by Benjamin Van Camp
This network shows the impact of papers produced by Benjamin Van Camp. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin Van Camp. The network helps show where Benjamin Van Camp may publish in the future.
Co-authorship network of co-authors of Benjamin Van Camp
This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin Van Camp. A scholar is included among the top collaborators of Benjamin Van Camp based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin Van Camp. Benjamin Van Camp is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | CDM analysis on 65nm CMOS: Pitfalls when correlating results between IO test chips and product level | 4 |
| 3 | 33 | |
| 4 | Concept for body coupling in SOI MOS transistors to improve multi-finger triggering | 8 |
| 5 | 19 | |
| 6 | Current detection trigger scheme for SCR based ESD protection of output drivers in CMOS technologies avoiding competitive triggering | 4 |
| 7 | 26 | |
| 8 | Multifunctional role of matrix metalloproteinases in multiple myeloma: study in the 5T2mm mouse model. | 1 |
| 9 | 52 | |
| 10 | 99 | |
| 11 | Bone marrow endothelial cells express the osteoclastogenic factor RANKL and its decoy receptor OPG: Evidence for a role in the development of myeloma bone disease. | 2 |
| 12 | A systemic inflammatory response involving C-Reactive Protein, Interleukin-6, Interleukin-8 and Tumor Necrosis Factor-alpha contributes to the development of hepatic veno-occlusive disease after allogeneic bone marrow transplantation. | 1 |
| 13 | 15 | |
| 14 | Bone marrow microenvironmental-induced upregulation of MMP-9 activity in murine multiple myeloma cells | 2 |
| 15 | The chemokine receptor CCR2 is expressed by human multiple myeloma cells and mediates chemotaxis to monocyte chemotactic proteins MCP-1, 2 and 3. | 5 |
| 16 | 11 | |
| 17 | 15 | |
| 18 | 34 | |
| 19 | 120 | |
| 20 | 23 |
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.