Ben Woodman

4.4k total citations
29 papers, 3.0k citations indexed

About

Ben Woodman is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Ben Woodman has authored 29 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 27 papers in Cellular and Molecular Neuroscience and 8 papers in Neurology. Recurrent topics in Ben Woodman's work include Genetic Neurodegenerative Diseases (27 papers), Mitochondrial Function and Pathology (18 papers) and Ubiquitin and proteasome pathways (12 papers). Ben Woodman is often cited by papers focused on Genetic Neurodegenerative Diseases (27 papers), Mitochondrial Function and Pathology (18 papers) and Ubiquitin and proteasome pathways (12 papers). Ben Woodman collaborates with scholars based in United Kingdom, United States and Switzerland. Ben Woodman's co-authors include Gillian P. Bates, Kirupa Sathasivam, Hilary Moffitt, Paolo Paganetti, Eric J. Bennett, Christopher H. Becker, Ron R. Kopito, Thomas A. Shaler, Tatiana S. Zaitseva and Howard Schulman and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Ben Woodman

29 papers receiving 3.0k citations

Peers

Ben Woodman
Judit Pallos United States
Alexander McCampbell United States
Kirupa Sathasivam United Kingdom
Xiao-Jiang Li United States
Kimberly B. Kegel United States
Montserrat Arrasate Spain
J. Jang‐Ho United States
Vikram G. Shakkottai United States
Kathryn Chase United States
J.-P. G. Vonsattel United States
Judit Pallos United States
Ben Woodman
Citations per year, relative to Ben Woodman Ben Woodman (= 1×) peers Judit Pallos

Countries citing papers authored by Ben Woodman

Since Specialization
Citations

This map shows the geographic impact of Ben Woodman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ben Woodman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ben Woodman more than expected).

Fields of papers citing papers by Ben Woodman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ben Woodman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ben Woodman. The network helps show where Ben Woodman may publish in the future.

Co-authorship network of co-authors of Ben Woodman

This figure shows the co-authorship network connecting the top 25 collaborators of Ben Woodman. A scholar is included among the top collaborators of Ben Woodman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ben Woodman. Ben Woodman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Diggle, Christine P., Isabel Martínez‐Garay, Zoltán Molnár, et al.. (2017). A tubulin alpha 8 mouse knockout model indicates a likely role in spermatogenesis but not in brain development. PLoS ONE. 12(4). e0174264–e0174264. 23 indexed citations
2.
Beconi, Maria, Omar Aziz, Kim L. Matthews, et al.. (2012). Oral Administration of the Pimelic Diphenylamide HDAC Inhibitor HDACi 4b Is Unsuitable for Chronic Inhibition of HDAC Activity in the CNS In Vivo. PLoS ONE. 7(9). e44498–e44498. 33 indexed citations
3.
Mielcarek, Michał, Caroline Benn, Sophie Franklin, et al.. (2011). SAHA Decreases HDAC 2 and 4 Levels In Vivo and Improves Molecular Phenotypes in the R6/2 Mouse Model of Huntington's Disease. PLoS ONE. 6(11). e27746–e27746. 128 indexed citations
4.
Landles, Christian, Kirupa Sathasivam, Andreas Weiss, et al.. (2010). Proteolysis of Mutant Huntingtin Produces an Exon 1 Fragment That Accumulates as an Aggregated Protein in Neuronal Nuclei in Huntington Disease. Journal of Biological Chemistry. 285(12). 8808–8823. 258 indexed citations
5.
Crittenden, Jill R., Denise Dunn, Ben Woodman, et al.. (2010). CalDAG-GEFI down-regulation in the striatum as a neuroprotective change in Huntington's disease. Human Molecular Genetics. 19(9). 1756–1765. 25 indexed citations
6.
Benn, Caroline, Rachel Butler, Hilary Moffitt, et al.. (2009). Genetic Knock-Down of HDAC7 Does Not Ameliorate Disease Pathogenesis in the R6/2 Mouse Model of Huntington's Disease. PLoS ONE. 4(6). e5747–e5747. 57 indexed citations
7.
Sathasivam, Kirupa, Justin Legleiter, Alice Warley, et al.. (2009). Identical oligomeric and fibrillar structures captured from the brains of R6/2 and knock-in mouse models of Huntington's disease. Human Molecular Genetics. 19(1). 65–78. 134 indexed citations
8.
Moffitt, Hilary, Graham McPhail, Ben Woodman, Carl Hobbs, & Gillian P. Bates. (2009). Formation of Polyglutamine Inclusions in a Wide Range of Non-CNS Tissues in the HdhQ150 Knock-In Mouse Model of Huntington's Disease. PLoS ONE. 4(11). e8025–e8025. 128 indexed citations
9.
Weiss, Andreas, Corinna Klein, Ben Woodman, et al.. (2007). Sensitive biochemical aggregate detection reveals aggregation onset before symptom development in cellular and murine models of Huntington’s disease. Journal of Neurochemistry. 104(3). 846–858. 94 indexed citations
10.
Bennett, Eric J., Thomas A. Shaler, Ben Woodman, et al.. (2007). Global changes to the ubiquitin system in Huntington's disease. Nature. 448(7154). 704–708. 417 indexed citations
11.
Gidalevitz, Tali, Mark Kristiansen, Christian Landles, et al.. (2007). Hsp27 overexpression in the R6/2 mouse model of Huntington's disease: chronic neurodegeneration does not induce Hsp27 activation. Human Molecular Genetics. 16(9). 1078–1090. 64 indexed citations
12.
Tarditi, Alessia, Alessandra Camurri, Katia Varani, et al.. (2006). Early and transient alteration of adenosine A2A receptor signaling in a mouse model of Huntington disease. Neurobiology of Disease. 23(1). 44–53. 63 indexed citations
13.
Woodman, Ben, Rachel Butler, Christian Landles, et al.. (2006). The HdhQ150/Q150 knock-in mouse model of HD and the R6/2 exon 1 model develop comparable and widespread molecular phenotypes. Brain Research Bulletin. 72(2-3). 83–97. 131 indexed citations
14.
Bett, John S., Geoffrey M. Goellner, Ben Woodman, et al.. (2006). Proteasome impairment does not contribute to pathogenesis in R6/2 Huntington's disease mice: exclusion of proteasome activator REGγ as a therapeutic target. Human Molecular Genetics. 15(4). 665–665. 1 indexed citations
15.
Valenza, Marta, Dorotea Rigamonti, Donato Goffredo, et al.. (2005). Dysfunction of the Cholesterol Biosynthetic Pathway in Huntington's Disease. Journal of Neuroscience. 25(43). 9932–9939. 218 indexed citations
16.
Bett, John S., Geoffrey M. Goellner, Ben Woodman, et al.. (2005). Proteasome impairment does not contribute to pathogenesis in R6/2 Huntington's disease mice: exclusion of proteasome activator REGγ as a therapeutic target. Human Molecular Genetics. 15(1). 33–44. 90 indexed citations
17.
Benn, Caroline, Christian Landles, Li He, et al.. (2005). Contribution of nuclear and extranuclear polyQ to neurological phenotypes in mouse models of Huntington's disease. Human Molecular Genetics. 14(20). 3065–3078. 99 indexed citations
18.
Peters, Peter J., Ke Ning, Rita L. Boshans, et al.. (2002). Arfaptin 2 regulates the aggregation of mutant huntingtin protein. Nature Cell Biology. 4(3). 240–245. 35 indexed citations
19.
Zabel, Claus, Daniel C. Chamrad, Josef Priller, et al.. (2002). Alterations in the Mouse and Human Proteome Caused by Huntington's Disease. Molecular & Cellular Proteomics. 1(5). 366–375. 73 indexed citations
20.
Smith, Donna L., R Portier, Ben Woodman, et al.. (2001). Inhibition of Polyglutamine Aggregation in R6/2 HD Brain Slices—Complex Dose–Response Profiles. Neurobiology of Disease. 8(6). 1017–1026. 57 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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