Ben T. Atkinson
Impact in
- Hematology top 2%
- Platelet Disorders and Treatments
- Blood Coagulation and Thrombosis Mechanisms
- Physiology top 2%
- Adenosine and Purinergic Signaling
Papers in
- Hematology 14
- Platelet Disorders and Treatments 12
- Blood Coagulation and Thrombosis Mechanisms 2
-
- Adenosine and Purinergic Signaling 2
- Co-authors
- Steve P. Watson (10 shared papers)Gavin E. Jarvis (4 shared papers)Wilfried Ellmeier (1 shared paper)Simon Christopher Robson (2 shared papers)Keiichi Enjyoji (1 shared paper)Karen M. Dwyer (1 shared paper)Jocelyn M. Auger (3 shared papers)Daniel C. Snell (2 shared papers)
- Journals
- Platelets (4 papers)Blood (3 papers)British Journal of Pharmacology (2 papers)Thrombosis and Haemostasis (1 paper)European Journal of Biochemistry (1 paper)
- Partner nations
- United KingdomUnited StatesAustria
In The Last Decade
Ben T. Atkinson
16 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 48
- Hematology 554
- Physiology 101
- Genetics 50
- Immunology and Allergy 15
- Equine 2
Countries citing papers authored by Ben T. Atkinson
This map shows the geographic impact of Ben T. Atkinson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ben T. Atkinson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ben T. Atkinson more than expected).
Fields of papers citing papers by Ben T. Atkinson
This network shows the impact of papers produced by Ben T. Atkinson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ben T. Atkinson. The network helps show where Ben T. Atkinson may publish in the future.
Co-authors
The 25 scholars most cited alongside Ben T. Atkinson, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2005 | 200 | |
| 2 | 2003 | 146 | |
| 3 | 2006 | 129 | |
| 4 | 2001 | 113 | |
| 5 | 2002 | 76 | |
| 6 | 2010 | 75 | |
| 7 | 2008 | 54 | |
| 8 | 2001 | 53 | |
| 9 | 2005 | 44 | |
| 10 | 2003 | 43 | |
| 11 | 2003 | 43 | |
| 12 | 2008 | 32 | |
| 13 | 2012 | 10 | |
| 14 | 2007 | 8 | |
| 15 | 2001 | 3 | |
| 16 | 2006 | 2 |
About Ben T. Atkinson
Ben T. Atkinson is a scholar working on Hematology, Physiology, Nuclear Energy and Engineering, Waste Management and Disposal and Infectious Diseases, having authored 16 papers that have together received 1.0k indexed citations. Recurring topics across this work include Platelet Disorders and Treatments (12 papers), Blood Coagulation and Thrombosis Mechanisms (2 papers) and Adenosine and Purinergic Signaling (2 papers). The work is most often cited by research in Hematology (554 citations), Physiology (101 citations), Genetics (50 citations), Immunology and Allergy (15 citations) and Equine (2 citations). Ben T. Atkinson has collaborated with scholars based in United Kingdom, United States and Austria. Frequent co-authors include Steve P. Watson, Gavin E. Jarvis, Wilfried Ellmeier, Simon Christopher Robson, Keiichi Enjyoji, Karen M. Dwyer, Jocelyn M. Auger, Daniel C. Snell, Neena Kalia and Andrew C. Pearce. Their work appears in journals such as Platelets, Blood, British Journal of Pharmacology, Thrombosis and Haemostasis and European Journal of Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.