Ben Sang

985 total citations
10 papers, 210 citations indexed

About

Ben Sang is a scholar working on Molecular Biology, Cell Biology and Cancer Research. According to data from OpenAlex, Ben Sang has authored 10 papers receiving a total of 210 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 5 papers in Cell Biology and 3 papers in Cancer Research. Recurrent topics in Ben Sang's work include Hippo pathway signaling and YAP/TAZ (2 papers), Ubiquitin and proteasome pathways (2 papers) and Cancer-related gene regulation (2 papers). Ben Sang is often cited by papers focused on Hippo pathway signaling and YAP/TAZ (2 papers), Ubiquitin and proteasome pathways (2 papers) and Cancer-related gene regulation (2 papers). Ben Sang collaborates with scholars based in China, Australia and United States. Ben Sang's co-authors include Rick F. Thorne, Xu Dong Zhang, Mian Wu, Guangzhi Liu, Rutong Yu, Xiuping Zhou, Qiong Cheng, Su Guo, Lei Jin and Yuan Yuan Zhang and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Ben Sang

9 papers receiving 208 citations

Peers

Ben Sang

MB

CR

CB

ONCOL

PHYSI

Yen Tran United States

Kiranmai Alapati United States

Laurensia Yuniati Netherlands

Teng Fei Qi China

Simone Jueliger United States

Johanna I. Englund Finland

Florian Christoph Sigloch Germany

Julie‐Ann Cavallo United States

Patrick Hofmann Germany

Angela De Laurentiis Italy

Yen Tran United States

Ben Sang

148 ×0.9

MB

48 ×0.5

CR

38 ×0.9

CB

46 ×1.5

ONCOL

33 ×2.1

PHYSI

Citations per year, relative to Ben Sang Ben Sang (= 1×) peers Yen Tran

Countries citing papers authored by Ben Sang

Since Specialization

Citations

This map shows the geographic impact of Ben Sang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ben Sang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ben Sang more than expected).

Fields of papers citing papers by Ben Sang

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ben Sang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ben Sang. The network helps show where Ben Sang may publish in the future.

Co-authorship network of co-authors of Ben Sang

This figure shows the co-authorship network connecting the top 25 collaborators of Ben Sang. A scholar is included among the top collaborators of Ben Sang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ben Sang. Ben Sang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Sang, Ben, et al.. (2025). The role of Atp2a2-mediated calcium imbalance and endoplasmic reticulum stress in hydrocortisone-induced neurotoxicity. Cell Stress and Chaperones. 30(6). 100112–100112.

2.

Cuevas-Navarro, Antonio, Feng Hu, Alberto Vides, et al.. (2024). Pharmacological restoration of GTP hydrolysis by mutant RAS. Nature. 637(8044). 224–229. 13 indexed citations

3.

Wang, Yan, et al.. (2021). Human giant larvae‑1 promotes migration and invasion of malignant glioma cells by regulating N‑cadherin. Oncology Letters. 21(2). 167–167. 3 indexed citations

4.

Sang, Ben, Guangzhi Liu, Jinming Li, et al.. (2020). SENEBLOC, a long non-coding RNA suppresses senescence via p53-dependent and independent mechanisms. Nucleic Acids Research. 48(6). 3089–3102. 39 indexed citations

5.

Sang, Ben, et al.. (2019). Ras-AKT signaling represses the phosphorylation of histone H1.5 at threonine 10 via GSK3 to promote the progression of glioma. Artificial Cells Nanomedicine and Biotechnology. 47(1). 2882–2890. 8 indexed citations

6.

Sang, Ben, Yuan Yuan Zhang, Su Guo, et al.. (2018). Dual functions for OVAAL in initiation of RAF/MEK/ERK prosurvival signals and evasion of p27-mediated cellular senescence. Proceedings of the National Academy of Sciences. 115(50). E11661–E11670. 55 indexed citations

7.

Liu, Xuejiao, Peng Ma, Huaqiang Liu, et al.. (2015). Hugl-1 inhibits glioma cell growth in intracranial model. Journal of Neuro-Oncology. 125(1). 113–121. 16 indexed citations

8.

Zhang, Xu, Ben Sang, Qiong Shi, et al.. (2014). GOLPH3 promotes glioblastoma cell migration and invasion via the mTOR‐YB1 pathway in vitro. Molecular Carcinogenesis. 54(11). 1252–1263. 55 indexed citations

9.

Zhou, Xiuping, Zhi Liu, Qiong Shi, et al.. (2013). Geranylgeranyltransferase I regulates HIF-1α promoting glioblastoma cell migration and invasion. Journal of Neuro-Oncology. 112(3). 365–374. 12 indexed citations

10.

Zhou, Xiuping, Lei Hua, Qiong Shi, et al.. (2012). Geranylgeranyltransferase I Promotes Human Glioma Cell Growth through Rac1 Membrane Association and Activation. Journal of Molecular Neuroscience. 49(1). 130–139. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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