Béla Novák
- Molecular Biology top 0.5%
- Cell Biology top 0.1%
- Plant Science top 2%
- Oncology top 5%
- Genetics top 2%
- Co-authors
- John J. TysonKatherine ChenAttila Csikász‐NagyKathy ChenBéla GyörffyAndrea CilibertoJ. M. MitchisonÁkos Sveiczer
- Topics
- Microtubule and mitosis dynamics (96 papers)Gene Regulatory Network Analysis (63 papers)Fungal and yeast genetics research (48 papers)
- Cited by
- Cell BiologyMolecular BiologyAging
- Partner nations
- United KingdomUnited StatesHungary
In The Last Decade
Béla Novák
135 papers receiving 10.1k citations
Hit Papers
Peers
Comparison fields: 5 of 150
- Molecular Biology 8.7k
- Cell Biology 3.3k
- Plant Science 1.4k
- Oncology 898
- Genetics 876
Countries citing papers authored by Béla Novák
This map shows the geographic impact of Béla Novák's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Béla Novák with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Béla Novák more than expected).
Fields of papers citing papers by Béla Novák
This network shows the impact of papers produced by Béla Novák. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Béla Novák. The network helps show where Béla Novák may publish in the future.
Co-authorship network of co-authors of Béla Novák
This figure shows the co-authorship network connecting the top 25 collaborators of Béla Novák. A scholar is included among the top collaborators of Béla Novák based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Béla Novák. Béla Novák is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 0 | |
| 3 | 1 | |
| 4 | 17 | |
| 5 | 42 | |
| 6 | 73 | |
| 7 | 14 | |
| 8 | 276 | |
| 9 | 132 | |
| 10 | 5 | |
| 11 | 12 | |
| 12 | 5 | |
| 13 | 8 | |
| 14 | 12 | |
| 15 | 28 | |
| 16 | DEVELOPING A MATHEMATICAL MODEL FOR THE FISSION YEAST CELL CYCLE: SIMULATING MUTANTS OVEREXPRESSING EITHER CDC25 OR WEE1 | 1 |
| 17 | 444 | |
| 18 | A deterministic molecular model of the fission yeast cell cycle. | 1 |
| 19 | 229 | |
| 20 | Model scenarios for evolution of the eukaryotic cell cycle | 1 |
About Béla Novák
Béla Novák is a scholar working on Cell Biology, Molecular Biology and Plant Science, having authored 137 papers that have together received 10.3k indexed citations. Recurring topics across this work include Microtubule and mitosis dynamics (96 papers), Gene Regulatory Network Analysis (63 papers) and Fungal and yeast genetics research (48 papers). The work is most often cited by research in Cell Biology (3.3k citations), Molecular Biology (8.7k citations) and Aging (156 citations). Béla Novák has collaborated with scholars based in United Kingdom, United States and Hungary. Frequent co-authors include John J. Tyson, Katherine Chen, Attila Csikász‐Nagy, Kathy Chen, Béla Györffy, Andrea Ciliberto, J. M. Mitchison, Ákos Sveiczer, Orsolya Kapuy and Frank Uhlmann. Their work appears in journals such as Nature, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.