Behnaz Pezeshkpoor

556 total citations
39 papers, 357 citations indexed

About

Behnaz Pezeshkpoor is a scholar working on Hematology, Molecular Biology and Genetics. According to data from OpenAlex, Behnaz Pezeshkpoor has authored 39 papers receiving a total of 357 indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Hematology, 15 papers in Molecular Biology and 10 papers in Genetics. Recurrent topics in Behnaz Pezeshkpoor's work include Hemophilia Treatment and Research (24 papers), Platelet Disorders and Treatments (18 papers) and Cancer-related gene regulation (12 papers). Behnaz Pezeshkpoor is often cited by papers focused on Hemophilia Treatment and Research (24 papers), Platelet Disorders and Treatments (18 papers) and Cancer-related gene regulation (12 papers). Behnaz Pezeshkpoor collaborates with scholars based in Germany, United Kingdom and United States. Behnaz Pezeshkpoor's co-authors include Johannes Oldenburg, Anna Pavlova, Osman El‐Maarri, Jens Müller, Daniel Delev, I. Nanda, Ulrich Budde, Thomas Haaf, Joseph R. McGill and Zuben E. Sauna and has published in prestigious journals such as Blood, PLoS ONE and International Journal of Molecular Sciences.

In The Last Decade

Behnaz Pezeshkpoor

32 papers receiving 350 citations

Peers

Behnaz Pezeshkpoor
Behnaz Pezeshkpoor
Citations per year, relative to Behnaz Pezeshkpoor Behnaz Pezeshkpoor (= 1×) peers Teresa Sevivas

Countries citing papers authored by Behnaz Pezeshkpoor

Since Specialization
Citations

This map shows the geographic impact of Behnaz Pezeshkpoor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Behnaz Pezeshkpoor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Behnaz Pezeshkpoor more than expected).

Fields of papers citing papers by Behnaz Pezeshkpoor

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Behnaz Pezeshkpoor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Behnaz Pezeshkpoor. The network helps show where Behnaz Pezeshkpoor may publish in the future.

Co-authorship network of co-authors of Behnaz Pezeshkpoor

This figure shows the co-authorship network connecting the top 25 collaborators of Behnaz Pezeshkpoor. A scholar is included among the top collaborators of Behnaz Pezeshkpoor based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Behnaz Pezeshkpoor. Behnaz Pezeshkpoor is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Müller, Jens, et al.. (2025). Laboratory Monitoring in Patients Receiving Emicizumab. Hämostaseologie. 1 indexed citations
2.
Ugurlar, Deniz, Behnaz Pezeshkpoor, Arijit Biswas, et al.. (2024). In-depth structure-function profiling of the complex formation between clotting factor VIII and heme. Thrombosis Research. 237. 184–195.
3.
Pezeshkpoor, Behnaz, R. Fischer, Heiko Rühl, et al.. (2024). Modulation of Haemostatic Balance in Combined von Willebrand Disease and Antithrombin Deficiency: A Comprehensive Family Study. Haemophilia. 31(1). 140–147. 1 indexed citations
4.
Pezeshkpoor, Behnaz, et al.. (2024). Comprehensive domain-specific analysis and immunoglobulin G profiling of anti–factor VIII antibodies using a bead-based multiplex immunoassay. Journal of Thrombosis and Haemostasis. 22(6). 1591–1604. 1 indexed citations
5.
Pezeshkpoor, Behnaz, Heiko Rühl, Vytautas Ivaškevičius, et al.. (2024). Genetic Landscape of Factor VII Deficiency: Insights from a Comprehensive Analysis of Pathogenic Variants and Their Impact on Coagulation Activity. International Journal of Molecular Sciences. 25(4). 2384–2384.
6.
Pezeshkpoor, Behnaz, Jens Müller, Bernd Pötzsch, et al.. (2024). Unravelling the spectrum of von Willebrand factor variants in quantitative von Willebrand disease: results from a German cohort study. Journal of Thrombosis and Haemostasis. 22(11). 3010–3034. 4 indexed citations
7.
Pezeshkpoor, Behnaz, et al.. (2024). Comprehensive evaluation of anti-emicizumab antibodies in acquired hemophilia A: a detailed case study and methodological evaluation. Journal of Thrombosis and Haemostasis. 23(1). 85–96. 2 indexed citations
8.
Pezeshkpoor, Behnaz, Johannes Dodt, Amit Sharma, et al.. (2023). Heterozygosity in factor XIII genes and the manifestation of mild inherited factor XIII deficiency. Journal of Thrombosis and Haemostasis. 22(2). 379–393. 3 indexed citations
9.
Pezeshkpoor, Behnaz, Johannes Oldenburg, & Anna Pavlova. (2022). Experiences in Routine Genetic Analysis of Hereditary Hemorrhagic, Thrombotic, and Platelet Disorders. Hämostaseologie. 42(S 01). S5–S12. 7 indexed citations
10.
McGill, Joseph R., et al.. (2020). Further Evidence That MicroRNAs Can Play a Role in Hemophilia A Disease Manifestation: F8 Gene Downregulation by miR-19b-3p and miR-186-5p. Frontiers in Cell and Developmental Biology. 8. 669–669. 9 indexed citations
11.
Bachelet, Delphine, T. Albert, Signe Hässler, et al.. (2019). Risk stratification integrating genetic data for factor VIII inhibitor development in patients with severe hemophilia A. PLoS ONE. 14(6). e0218258–e0218258. 13 indexed citations
12.
Pezeshkpoor, Behnaz, et al.. (2019). In silico and in vitro evaluation of the impact of mutations in non-severe haemophilia A patients on assay discrepancies. Annals of Hematology. 98(8). 1855–1865. 9 indexed citations
14.
Pezeshkpoor, Behnaz, Arijit Biswas, Julia Driesen, et al.. (2017). An in silico and in vitro approach to elucidate the impact of residues flanking the cleavage scissile bonds of FVIII. PLoS ONE. 12(7). e0180456–e0180456. 4 indexed citations
15.
Pezeshkpoor, Behnaz, Elisabetta Castoldi, Jens Müller, et al.. (2016). Identification and functional characterization of a novel F5 mutation (Ala512Val, FVBonn) associated with activated protein C resistance. Journal of Thrombosis and Haemostasis. 14(7). 1353–1363. 22 indexed citations
16.
El‐Maarri, Osman, et al.. (2015). Novel characterization of a breakpoint in F8. Haemophilia. 3 indexed citations
17.
Pavlova, Anna, Daniel Delev, Behnaz Pezeshkpoor, Jens Müller, & Johannes Oldenburg. (2014). Haemophilia A mutations in patients with non-severe phenotype associated with a discrepancy between one-stage and chromogenic factor VIII activity assays. Thrombosis and Haemostasis. 112(5). 851–861. 55 indexed citations
18.
Pezeshkpoor, Behnaz, I. Nanda, Thomas Haaf, et al.. (2013). Deep intronic ‘mutations’ cause hemophilia A: application of next generation sequencing in patients without detectable mutation in F8 cDNA. Journal of Thrombosis and Haemostasis. 11(9). 1679–1687. 59 indexed citations
19.
Pezeshkpoor, Behnaz, Anna Pavlova, Johannes Oldenburg, & Osman El‐Maarri. (2013). F8 genetic analysis strategies when standard approaches fail. Hämostaseologie. 34(2). 167–173. 15 indexed citations
20.
Pezeshkpoor, Behnaz, Simone Rost, Johannes Oldenburg, & Osman El‐Maarri. (2012). Identification of a third rearrangement at Xq28 that causes severe hemophilia A as a result of homologous recombination between inverted repeats. Journal of Thrombosis and Haemostasis. 10(8). 1600–1608. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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