Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
High affinity VEGF binding and developmental expression suggest Flk-1 as a major regulator of vasculogenesis and angiogenesis
19931.7k citationsB Millauer, Harald Schnürch et al.Cellprofile →
Glioblastoma growth inhibited in vivo by a dominant-negative Flk-1 mutant
19941.0k citationsB Millauer, Laura K. Shawver et al.Natureprofile →
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late)
cites ·
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This map shows the geographic impact of B Millauer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by B Millauer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites B Millauer more than expected).
This network shows the impact of papers produced by B Millauer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by B Millauer. The network helps show where B Millauer may publish in the future.
Co-authorship network of co-authors of B Millauer
This figure shows the co-authorship network connecting the top 25 collaborators of B Millauer.
A scholar is included among the top collaborators of B Millauer based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with B Millauer. B Millauer is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
All Works
9 of 9 papers shown
1.
Millauer, B, et al.. (1996). Dominant-negative inhibition of Flk-1 suppresses the growth of many tumor types in vivo.. PubMed. 56(7). 1615–20.354 indexed citations
2.
Ciossek, Thomas, B Millauer, & A Ullrich. (1995). Identification of alternatively spliced mRNAs encoding variants of MDK1, a novel receptor tyrosine kinase expressed in the murine nervous system.. PubMed. 10(1). 97–108.60 indexed citations
3.
Alves, Frauke, Wolfgang F. Vogel, K G Mossie, et al.. (1995). Distinct structural characteristics of discoidin I subfamily receptor tyrosine kinases and complementary expression in human cancer.. PubMed. 10(3). 609–18.210 indexed citations
4.
Millauer, B, et al.. (1994). Glioblastoma growth inhibited in vivo by a dominant-negative Flk-1 mutant. Nature. 367(6463). 576–579.1036 indexed citations breakdown →
5.
Millauer, B, Harald Schnürch, Ricardo Martínez, et al.. (1993). High affinity VEGF binding and developmental expression suggest Flk-1 as a major regulator of vasculogenesis and angiogenesis. Cell. 72(6). 835–846.1651 indexed citations breakdown →
6.
Plate, Karl H., Georg Breier, B Millauer, A. Ullrich, & Werner Risau. (1993). Up-regulation of vascular endothelial growth factor and its cognate receptors in a rat glioma model of tumor angiogenesis.. PubMed. 53(23). 5822–7.440 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.