B Ibarra
- Genetics top 5%
- Hemoglobinopathies and Related Disorders 30
- Forensic and Genetic Research 8
- Hematology top 5%
- Iron Metabolism and Disorders 17
- Blood groups and transfusion 11
- Genetics top 10%
- Hemoglobinopathies and Related Disorders 30
- Forensic and Genetic Research 8
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- Neonatal Health and Biochemistry 11
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- Metabolism and Genetic Disorders 8
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- Erythrocyte Function and Pathophysiology 13
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- Pancreatic function and diabetes 7
- Co-authors
- Francisco J. PereaG VacaHoracio RiveraJ. M. Cant�Lucila SandovalJ. M. CantúJosé María CantúAlma Rosa Villalobos-Arámbula
- Journals
- Human Genetics (8 papers)Blood Cells Molecules and Diseases (7 papers)American Journal of Human Biology (2 papers)
- Partner nations
- MexicoUnited StatesUnited Kingdom
In The Last Decade
B Ibarra
70 papers receiving 604 citations
Peers
Comparison fields: 5 of 66
- Genetics 209
- Hematology 166
- Genetics 246
- Pediatrics, Perinatology and Child Health 147
- Clinical Biochemistry 27
Countries citing papers authored by B Ibarra
This map shows the geographic impact of B Ibarra's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by B Ibarra with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites B Ibarra more than expected).
Fields of papers citing papers by B Ibarra
This network shows the impact of papers produced by B Ibarra. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by B Ibarra. The network helps show where B Ibarra may publish in the future.
Co-authorship network
The 25 scholars most cited alongside B Ibarra, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 9 | |
| 2 | 2013 | 28 | |
| 3 | Wayward effect of polymorphism (TA)8 in the promoter region of UGT1A1 gene in a Mexican family. | 2012 | 0 |
| 4 | 2009 | 3 | |
| 5 | 2009 | 4 | |
| 6 | 2007 | 9 | |
| 7 | 2007 | 5 | |
| 8 | 2004 | 10 | |
| 9 | 2004 | 2 | |
| 10 | Analysis of HLA DQA1 and DQB1 alleles and glycated hemoglobin in healthy Mexican mestizo individuals from families with type 1 diabetes mellitus | 2003 | 0 |
| 11 | 2003 | 21 | |
| 12 | 2002 | 26 | |
| 13 | 1999 | 0 | |
| 14 | 1999 | 22 | |
| 15 | 1997 | 15 | |
| 16 | 1996 | 1 | |
| 17 | 1991 | 12 | |
| 18 | Hematological and biochemical studies in children with Down syndrome. | 1990 | 18 |
| 19 | 1980 | 10 | |
| 20 | De novo partial trisomy of chromosome 18(pter yields q11:). Some observations on the phenotype mapping of chromosome 18 imbalances. | 1979 | 5 |
About B Ibarra
B Ibarra is a scholar working on Genetics, Hematology, Clinical Biochemistry, Genetics and Pediatrics, Perinatology and Child Health, having authored 74 papers that have together received 628 indexed citations. Recurring topics across this work include Hemoglobinopathies and Related Disorders (30 papers), Iron Metabolism and Disorders (17 papers), Erythrocyte Function and Pathophysiology (13 papers), Neonatal Health and Biochemistry (11 papers), Blood groups and transfusion (11 papers), Metabolism and Genetic Disorders (8 papers), Forensic and Genetic Research (8 papers) and Pancreatic function and diabetes (7 papers). The work is most often cited by research in Genetics (209 citations), Hematology (166 citations), Genetics (246 citations), Pediatrics, Perinatology and Child Health (147 citations) and Clinical Biochemistry (27 citations). B Ibarra has collaborated with scholars based in Mexico, United States and United Kingdom. Frequent co-authors include Francisco J. Perea, G Vaca, Horacio Rivera, J. M. Cant�, Lucila Sandoval, J. M. Cantú, José María Cantú, Alma Rosa Villalobos-Arámbula, Cantú Jm and Luis E. Figuera. Their work appears in journals such as Human Genetics, Blood Cells Molecules and Diseases, American Journal of Human Biology, American Journal of Hematology and Hemoglobin.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.