B. Fakler

2.3k total citations · 1 hit paper
17 papers, 1.9k citations indexed

About

B. Fakler is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, B. Fakler has authored 17 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 12 papers in Cellular and Molecular Neuroscience and 8 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in B. Fakler's work include Ion channel regulation and function (15 papers), Cardiac electrophysiology and arrhythmias (8 papers) and Neuroscience and Neuropharmacology Research (8 papers). B. Fakler is often cited by papers focused on Ion channel regulation and function (15 papers), Cardiac electrophysiology and arrhythmias (8 papers) and Neuroscience and Neuropharmacology Research (8 papers). B. Fakler collaborates with scholars based in Germany, United States and Switzerland. B. Fakler's co-authors include J. P. Ruppersberg, Hans‐Peter Zenner, U. Brändle, Elisabeth Glowatzki, Chris T. Bond, Susanne Weidemann, John P. Adelman, J. E. Keen, Gary A. Wayman and Teresa L. Johnson‐Pais and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

B. Fakler

17 papers receiving 1.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mechanism of calcium gating in small-conductance calcium-activated potassium channels

1998 731 citations Xuefeng Xia, B. Fakler et al. Nature profile →

Peers

Countries citing papers authored by B. Fakler

Since Specialization

Citations

This map shows the geographic impact of B. Fakler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by B. Fakler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites B. Fakler more than expected).

Fields of papers citing papers by B. Fakler

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by B. Fakler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by B. Fakler. The network helps show where B. Fakler may publish in the future.

Co-authorship network of co-authors of B. Fakler

This figure shows the co-authorship network connecting the top 25 collaborators of B. Fakler. A scholar is included among the top collaborators of B. Fakler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with B. Fakler. B. Fakler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

#	Work	Indexed citations
1	pH gating of ROMK (K _ir 1.1) channels: Control by an Arg-Lys-Arg triad disrupted in antenatal Bartter syndrome 1999 ·Proceedings of the National Academy of Sciences ·Uwe Schulte, Hartmut Hahn, Moira Konrad, Nikola Jeck, Christian Derst, K. Wild, Susanne Weidemann, J. P. Ruppersberg, B. Fakler, Jost Ludwig	123
2	Mechanism of calcium gating in small-conductance calcium-activated potassium channels breakdown → 1998 ·Nature ·Xuefeng Xia, B. Fakler, (unknown), Gary A. Wayman, Teresa L. Johnson‐Pais, J. E. Keen, Takahiro Ishii, Birgit Hirschberg, Chris T. Bond, Svetlana Lutsenko, James Maylie, John P. Adelman	731
3	Interaction of Permeant and Blocking Ions in Cloned Inward-Rectifier K+ Channels 1998 ·Biophysical Journal ·Dominik Oliver, Hartmut Hahn, (unknown), J. P. Ruppersberg, B. Fakler	45
4	Coexpression and stimulation of parathyroid hormone receptor positively regulates slowly activating IsK channels expressed in Xenopus oocytes 1996 ·Kidney International ·(unknown), Gertraud Raber, (unknown), J. P. Ruppersberg, B. Fakler, Heini Murer, (unknown), Andreas Büsch	3
5	Complexity of the Regulation of K ir 2.1 K + Channels 1996 ·Neuropharmacology ·J. P. Ruppersberg, B. Fakler	26
6	Heterooligomeric assembly of inward-rectifier K + channels from subunits of different subfamilies: K ir 2.1 (IRK1) and K ir 4.1 (BIR10) 1996 ·Pflügers Archiv - European Journal of Physiology ·B. Fakler, Chris T. Bond, J.P. Adelman, J. P. Ruppersberg	38
7	Identification of a titratable lysine residue that determines sensitivity of kidney potassium channels (ROMK) to intracellular pH. 1996 ·The EMBO Journal ·B. Fakler, Jobst‐Hendrik Schultz, (unknown), Uwe Schulte, U. Brändle, Hans‐Peter Zenner, Lily Yeh Jan, J. P. Ruppersberg	157
8	Functional and Molecular Diversity Classifies the Family of Inward-Rectifier K<sup>+</sup> Channels 1996 ·Cellular Physiology and Biochemistry ·B. Fakler, J. P. Ruppersberg	29
9	Strong voltage-dependent inward rectification of inward rectifier K+ channels is caused by intracellular spermine 1995 ·Cell ·B. Fakler, U. Brändle, Elisabeth Glowatzki, Susanne Weidemann, Hans‐Peter Zenner, J. P. Ruppersberg	313
10	Cell-specific expression of theα9 n-ACh receptor subunit in auditory hair cells revealed by single-cell RT-PCR 1995 ·Proceedings of the Royal Society B Biological Sciences ·Elisabeth Glowatzki, K. Wild, U. Brändle, (unknown), B. Fakler, H. P. Zenner, J. P. Ruppersberg	54
11	Subunit‐specific inhibition of inward‐rectifier K⁺ channels by quinidine 1995 ·FEBS Letters ·Tomomitsu Doi, B. Fakler, Jobst‐Hendrik Schultz, Heimo Ehmke, U. Brändle, Hans‐Peter Zenner, (unknown), Florian Läng, J. P. Ruppersberg, Andreas Büsch	12
12	Subunit-dependent assembly of inward-rectifier K ⁺ channels 1995 ·Proceedings of the Royal Society B Biological Sciences ·Elisabeth Glowatzki, (unknown), U. Brändle, U Rexhausen, Hans‐Peter Zenner, J. P. Ruppersberg, B. Fakler	100
13	Kir2.1 inward rectifier K+ channels are regulated independently by protein kinases and ATP hydrolysis 1994 ·Neuron ·B. Fakler, U. Brändle, Elisabeth Glowatzki, Hans‐Peter Zenner, J. P. Ruppersberg	127
14	A structural determinant of differential sensitivity of cloned inward rectifier K⁺ channels to intracellular spermine 1994 ·FEBS Letters ·B. Fakler, U. Brändle, (unknown), Elisabeth Glowatzki, Christiane König, J.P. Adelman, Hans‐Peter Zenner, J. P. Ruppersberg	119
15	Short antisense oligonucleotide-mediated inhibition is strongly dependent on oligo length and concentration but almost independent of location of the target sequence. 1994 ·Journal of Biological Chemistry ·B. Fakler, Stefan Herlitze, Beate Amthor, Hans‐Peter Zenner, J. P. Ruppersberg	39
16	Stereoselective interaction of tocainide and its chiral analogs with the sodium channels in human myoballs 1991 ·Pflügers Archiv - European Journal of Physiology ·Domenico Tricarico, B. Fakler, (unknown), J. P. Ruppersberg, (unknown), Carlo Franchini, Vincenzo Tortorella, Diana Conte Camerino, R. R�del	8
17	Inactivation of human sodium channels and the effect of tocainide 1990 ·Pflügers Archiv - European Journal of Physiology ·B. Fakler, J. P. Ruppersberg, (unknown), Reinhardt Rüdel	22

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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