Axel W.A. Baltzer

673 citations
21 papers · 473 indexed · h-index 10

Axel W.A. Baltzer

20 papers receiving 456 citations

Peers

Axel W.A. Baltzer
Comparison fields: 5 of 75
  • Orthopedics and Sports Medicine 139
  • Biological Psychiatry 37
  • Rheumatology 86
  • Rehabilitation 38
  • Behavioral Neuroscience 19
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Peter Angele Germany
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Citations per year

Countries citing papers authored by Axel W.A. Baltzer

Since Specialization
Citations

This map shows the geographic impact of Axel W.A. Baltzer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Axel W.A. Baltzer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Axel W.A. Baltzer more than expected).

Fields of papers citing papers by Axel W.A. Baltzer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Axel W.A. Baltzer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Axel W.A. Baltzer. The network helps show where Axel W.A. Baltzer may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Axel W.A. Baltzer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Axel W.A. Baltzer Line = papers co-authored together Axel W.A. Baltzer links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20250
2 20234
3 20213
4 201621
5 201336
6 200957
7 2005120
8 20048
9
Immunotherapy by gene transfer with plasmids encoding IL-12/IL-18 is superior to IL-23/IL-18 gene transfer in a rat osteosarcoma model.
200414
10 200310
11
rIL-18 triggered gene therapy based on a transduction with the IL-12 plasmid: a new option as immuno-therapy for osteosarcoma?
20034
12 200218
13
Interleukin-12 and interleukin-18 induce indoleamine 2,3-dioxygenase (IDO) activity in human osteosarcoma cell lines independently from interferon-gamma.
200262
14 200057
15 20004
16 20001
17 19999
18 19984
19 19982
20 199532

About Axel W.A. Baltzer

Axel W.A. Baltzer is a scholar working on Transplantation, Orthopedics and Sports Medicine and Complementary and Manual Therapy, having authored 21 papers that have together received 473 indexed citations. Recurring topics across this work include Virus-based gene therapy research (5 papers), Tendon Structure and Treatment (3 papers), Shoulder Injury and Treatment (3 papers), Bone fractures and treatments (3 papers), Orthopaedic implants and arthroplasty (2 papers), Biomedical Ethics and Regulation (2 papers), CAR-T cell therapy research (2 papers) and Musculoskeletal synovial abnormalities and treatments (2 papers). The work is most often cited by research in Orthopedics and Sports Medicine (139 citations), Biological Psychiatry (37 citations) and Rheumatology (86 citations). Axel W.A. Baltzer has collaborated with scholars based in Germany, United States and Poland. Frequent co-authors include Martin Ostapczuk, Paul D. Robbins, H. Merk, Christian Liebau, Christian Roesel, Sebastian Schmidt, Hans Bojar, Christiaan Karreman, W. Rüther and Janey D. Whalen. Their work appears in journals such as SHILAP Revista de lepidopterología, Clinical Orthopaedics and Related Research and Arthroscopy The Journal of Arthroscopic and Related Surgery.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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