Aurora Lopez‐Rosas

2.0k total citations · 1 hit paper
24 papers, 1.5k citations indexed

About

Aurora Lopez‐Rosas is a scholar working on Immunology, Physiology and Cell Biology. According to data from OpenAlex, Aurora Lopez‐Rosas has authored 24 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Immunology, 8 papers in Physiology and 7 papers in Cell Biology. Recurrent topics in Aurora Lopez‐Rosas's work include Immune cells in cancer (11 papers), Lysosomal Storage Disorders Research (8 papers) and Cellular transport and secretion (5 papers). Aurora Lopez‐Rosas is often cited by papers focused on Immune cells in cancer (11 papers), Lysosomal Storage Disorders Research (8 papers) and Cellular transport and secretion (5 papers). Aurora Lopez‐Rosas collaborates with scholars based in United States, Italy and Sweden. Aurora Lopez‐Rosas's co-authors include Maria I. Givogri, Ernesto R. Bongarzone, Yu Han, Aida Rashidi, Jason Miska, Maciej S. Lesniak, Peng Zhang, Catalina Lee-Chang, Richard B. van Breemen and Ludovico Cantuti‐Castelvetri and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Aurora Lopez‐Rosas

23 papers receiving 1.5k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

STING agonist-loaded, CD47/PD-L1-targeting nanoparticles potentiate antitumor immunity and radiotherapy for glioblastoma

2023 139 citations Peng Zhang, Aida Rashidi et al. Nature Communications profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Aurora Lopez‐Rosas United States	19	536	527	502	276	265	24	1.5k
Laura J. Lewis‐Tuffin United States	19	757 1.4×	181 0.3×	183 0.4×	326 1.2×	160 0.6×	23	1.5k
Stéphane C. Boutet United States	18	1.3k 2.3×	317 0.6×	153 0.3×	135 0.5×	171 0.6×	28	1.9k
Jillian Haight Canada	14	792 1.5×	535 1.0×	90 0.2×	225 0.8×	125 0.5×	20	1.6k
Tracy L. Hagemann United States	26	1.1k 2.1×	628 1.2×	134 0.3×	90 0.3×	458 1.7×	48	2.1k
Stefano Regis Italy	22	1.2k 2.2×	271 0.5×	521 1.0×	185 0.7×	317 1.2×	57	1.9k
Raymond K. Tong United States	12	1.1k 2.0×	200 0.4×	161 0.3×	582 2.1×	175 0.7×	15	1.7k
Karin Golan Israel	16	567 1.1×	435 0.8×	146 0.3×	184 0.7×	82 0.3×	30	1.4k
A.K.M. Ghulam Muhammad United States	18	1000 1.9×	517 1.0×	225 0.4×	303 1.1×	52 0.2×	35	2.1k
Mariko Kobayashi Japan	15	763 1.4×	417 0.8×	109 0.2×	273 1.0×	243 0.9×	33	1.9k
Siddhartha S. Mitra United States	23	960 1.8×	921 1.7×	106 0.2×	613 2.2×	103 0.4×	52	2.5k

Countries citing papers authored by Aurora Lopez‐Rosas

Since Specialization

Citations

This map shows the geographic impact of Aurora Lopez‐Rosas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aurora Lopez‐Rosas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aurora Lopez‐Rosas more than expected).

Fields of papers citing papers by Aurora Lopez‐Rosas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Aurora Lopez‐Rosas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aurora Lopez‐Rosas. The network helps show where Aurora Lopez‐Rosas may publish in the future.

Co-authorship network of co-authors of Aurora Lopez‐Rosas

This figure shows the co-authorship network connecting the top 25 collaborators of Aurora Lopez‐Rosas. A scholar is included among the top collaborators of Aurora Lopez‐Rosas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aurora Lopez‐Rosas. Aurora Lopez‐Rosas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zhang, Peng, Aida Rashidi, Junfei Zhao, et al.. (2023). STING agonist-loaded, CD47/PD-L1-targeting nanoparticles potentiate antitumor immunity and radiotherapy for glioblastoma. Nature Communications. 14(1). 1610–1610. 139 indexed citations breakdown →

Lopez‐Rosas, Aurora, et al.. (2023). IMMU-06. THE ROLE OF BRAIN TUMOR MICROENVIRONMENT ON T CELL TELOMERE SHORTENING. Neuro-Oncology. 25(Supplement_5). v142–v142.

Miska, Jason, Aida Rashidi, Catalina Lee-Chang, et al.. (2021). Polyamines drive myeloid cell survival by buffering intracellular pH to promote immunosuppression in glioblastoma. Science Advances. 7(8). 75 indexed citations

Hou, David, Brandyn Castro, Aida Rashidi, et al.. (2021). IMMU-36. B CELL-VACCINE ELICITS LONG TERM IMMUNITY AGAINST GLIOBLASTOMA VIA ACTIVATION AND DIFFERENTIATION OF TUMOR-SPECIFIC CD8+ MEMORY T CELLS. Neuro-Oncology. 23(Supplement_6). vi100–vi100. 1 indexed citations

Lee-Chang, Catalina, Jason Miska, David Hou, et al.. (2020). Activation of 4-1BBL+ B cells with CD40 agonism and IFNγ elicits potent immunity against glioblastoma. The Journal of Experimental Medicine. 218(1). 49 indexed citations

Lee-Chang, Catalina, Aida Rashidi, Jason Miska, et al.. (2019). Myeloid-Derived Suppressive Cells Promote B cell–Mediated Immunosuppression via Transfer of PD-L1 in Glioblastoma. Cancer Immunology Research. 7(12). 1928–1943. 120 indexed citations

Miska, Jason, Catalina Lee-Chang, Aida Rashidi, et al.. (2019). HIF-1α Is a Metabolic Switch between Glycolytic-Driven Migration and Oxidative Phosphorylation-Driven Immunosuppression of Tregs in Glioblastoma. Cell Reports. 27(1). 226–237.e4. 230 indexed citations

Rashidi, Aida, Jason Miska, Catalina Lee-Chang, et al.. (2019). GCN2 is essential for CD8+ T cell survival and function in murine models of malignant glioma. Cancer Immunology Immunotherapy. 69(1). 81–94. 40 indexed citations

Wu, Congyu, Megan E. Muroski, Jason Miska, et al.. (2018). Repolarization of myeloid derived suppressor cells via magnetic nanoparticles to promote radiotherapy for glioma treatment. Nanomedicine Nanotechnology Biology and Medicine. 16. 126–137. 45 indexed citations

10.

Rashidi, Aida, Jason Miska, Catalina Lee-Chang, et al.. (2018). IMMU-06. ABSENCE OF THE AMINO ACID STRESS-SENSOR GCN2 REDUCES SUPPRESSIVE EFFECTS OF MDSCs IN GLIOMA. Neuro-Oncology. 20(suppl_6). vi122–vi122. 1 indexed citations

11.

Panek, Wojciech K., Katarzyna C. Pituch, Jason Miska, et al.. (2018). Local Application of Autologous Platelet-Rich Fibrin Patch (PRF-P) Suppresses Regulatory T Cell Recruitment in a Murine Glioma Model. Molecular Neurobiology. 56(7). 5032–5040. 18 indexed citations

12.

Muroski, Megan E., Jason Miska, Alan L. Chang, et al.. (2017). Fatty Acid Uptake in T Cell Subsets Using a Quantum Dot Fatty Acid Conjugate. Scientific Reports. 7(1). 5790–5790. 26 indexed citations

13.

Cantuti‐Castelvetri, Ludovico, Michael S. Marshall, Ludovic D’Auria, et al.. (2015). Mechanism of Neuromuscular Dysfunction in Krabbe Disease. Journal of Neuroscience. 35(4). 1606–1616. 28 indexed citations

14.

Pituch, Katarzyna C., Ana Lis Moyano, Aurora Lopez‐Rosas, et al.. (2015). Dysfunction of Platelet-derived Growth Factor Receptor α (PDGFRα) Represses the Production of Oligodendrocytes from Arylsulfatase A-deficient Multipotential Neural Precursor Cells. Journal of Biological Chemistry. 290(11). 7040–7053. 37 indexed citations

15.

Moyano, Ana Lis, Guannan Li, Aurora Lopez‐Rosas, et al.. (2014). Distribution of C16:0, C18:0, C24:1, and C24:0 sulfatides in central nervous system lipid rafts by quantitative ultra-high-pressure liquid chromatography tandem mass spectrometry. Analytical Biochemistry. 467. 31–39. 26 indexed citations

16.

Cantuti‐Castelvetri, Ludovico, Maria I. Givogri, Audrey Hébert, et al.. (2013). The Sphingolipid Psychosine Inhibits Fast Axonal Transport in Krabbe Disease by Activation of GSK3 and Deregulation of Molecular Motors. Journal of Neuroscience. 33(24). 10048–10056. 76 indexed citations

17.

Cantuti‐Castelvetri, Ludovico, et al.. (2012). Psychosine induces the dephosphorylation of neurofilaments by deregulation of PP1 and PP2A phosphatases. Neurobiology of Disease. 46(2). 325–335. 43 indexed citations

18.

Cantuti‐Castelvetri, Ludovico, Maria I. Givogri, Hongling Zhu, et al.. (2011). Axonopathy is a compounding factor in the pathogenesis of Krabbe disease. Acta Neuropathologica. 122(1). 35–48. 81 indexed citations

19.

Givogri, Maria I., Aurora Lopez‐Rosas, Hongmei Cao, et al.. (2009). Psychosine Accumulates in Membrane Microdomains in the Brain of Krabbe Patients, Disrupting the Raft Architecture. Journal of Neuroscience. 29(19). 6068–6077. 138 indexed citations

20.

Galbiati, Francesca, Veronica Basso, Ludovico Cantuti‐Castelvetri, et al.. (2007). Autonomic Denervation of Lymphoid Organs Leads to Epigenetic Immune Atrophy in a Mouse Model of Krabbe Disease. Journal of Neuroscience. 27(50). 13730–13738. 50 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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