Ashwin N. Ananthakrishnan
- Genetics top 0.02%
- Epidemiology top 0.05%
- Surgery top 0.2%
- Molecular Biology top 1%
- Infectious Diseases top 0.2%
- Co-authors
- David G. BinionHamed KhaliliEmily L. McGinleyAndrew T. ChanKia SaeianJames M. RichterVijay YajnikMillie D. Long
- Topics
- Inflammatory Bowel Disease (316 papers)Microscopic Colitis (264 papers)Eosinophilic Esophagitis (62 papers)
- Cited by
- GastroenterologyGeneticsEpidemiology
- Partner nations
- United StatesCanadaFrance
In The Last Decade
Ashwin N. Ananthakrishnan
469 papers receiving 22.1k citations
Hit Papers
Peers
Comparison fields: 5 of 180
- Genetics 11.6k
- Epidemiology 10.6k
- Surgery 5.8k
- Molecular Biology 4.4k
- Infectious Diseases 3.7k
Countries citing papers authored by Ashwin N. Ananthakrishnan
This map shows the geographic impact of Ashwin N. Ananthakrishnan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ashwin N. Ananthakrishnan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ashwin N. Ananthakrishnan more than expected).
Fields of papers citing papers by Ashwin N. Ananthakrishnan
This network shows the impact of papers produced by Ashwin N. Ananthakrishnan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ashwin N. Ananthakrishnan. The network helps show where Ashwin N. Ananthakrishnan may publish in the future.
Co-authorship network of co-authors of Ashwin N. Ananthakrishnan
This figure shows the co-authorship network connecting the top 25 collaborators of Ashwin N. Ananthakrishnan. A scholar is included among the top collaborators of Ashwin N. Ananthakrishnan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ashwin N. Ananthakrishnan. Ashwin N. Ananthakrishnan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 6 | |
| 3 | 0 | |
| 4 | 1 | |
| 5 | 11 | |
| 6 | 18 | |
| 7 | 2 | |
| 8 | 8 | |
| 9 | 9 | |
| 10 | 15 | |
| 11 | 5 | |
| 12 | 28 | |
| 13 | 8 | |
| 14 | 98 | |
| 15 | 64 | |
| 16 | 221 | |
| 17 | 111 | |
| 18 | 32 | |
| 19 | 27 | |
| 20 | 21 |
About Ashwin N. Ananthakrishnan
Ashwin N. Ananthakrishnan is a scholar working on Genetics, Gastroenterology and Epidemiology, having authored 498 papers that have together received 22.5k indexed citations. Recurring topics across this work include Inflammatory Bowel Disease (316 papers), Microscopic Colitis (264 papers) and Eosinophilic Esophagitis (62 papers). The work is most often cited by research in Gastroenterology (2.5k citations), Genetics (11.6k citations) and Epidemiology (10.6k citations). Ashwin N. Ananthakrishnan has collaborated with scholars based in United States, Canada and France. Frequent co-authors include David G. Binion, Hamed Khalili, Emily L. McGinley, Andrew T. Chan, Kia Saeian, James M. Richter, Vijay Yajnik, Millie D. Long, Leslie M. Higuchi and Charles S. Fuchs. Their work appears in journals such as New England Journal of Medicine, Journal of the American Chemical Society and Nature Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.