Ashraf Ul Kabir

448 total citations
13 papers, 305 citations indexed

About

Ashraf Ul Kabir is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Immunology. According to data from OpenAlex, Ashraf Ul Kabir has authored 13 papers receiving a total of 305 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Endocrinology, Diabetes and Metabolism and 5 papers in Immunology. Recurrent topics in Ashraf Ul Kabir's work include Immune cells in cancer (4 papers), Angiogenesis and VEGF in Cancer (4 papers) and Natural Antidiabetic Agents Studies (3 papers). Ashraf Ul Kabir is often cited by papers focused on Immune cells in cancer (4 papers), Angiogenesis and VEGF in Cancer (4 papers) and Natural Antidiabetic Agents Studies (3 papers). Ashraf Ul Kabir collaborates with scholars based in United States, Bangladesh and South Korea. Ashraf Ul Kabir's co-authors include J. M. A. Hannan, Kyunghee Choi, Ahmed Arif, Karen Krchma, Samuel A. Wickline, Craig S. Smith, Hua Pan, Sunday S. Oladipupo, Jun Wu and David M. Ornitz and has published in prestigious journals such as Nature reviews. Immunology, Nature Immunology and PLoS ONE.

In The Last Decade

Ashraf Ul Kabir

13 papers receiving 301 citations

Peers

Ashraf Ul Kabir
Hyeon‐Hui Ki South Korea
Bo‐Ram Jin South Korea
Shu Dai China
Kyoung Jin Nho South Korea
Xichao Yu China
Hyun‐Kyoung Kim South Korea
Hyeon‐Hui Ki South Korea
Ashraf Ul Kabir
Citations per year, relative to Ashraf Ul Kabir Ashraf Ul Kabir (= 1×) peers Hyeon‐Hui Ki

Countries citing papers authored by Ashraf Ul Kabir

Since Specialization
Citations

This map shows the geographic impact of Ashraf Ul Kabir's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ashraf Ul Kabir with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ashraf Ul Kabir more than expected).

Fields of papers citing papers by Ashraf Ul Kabir

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ashraf Ul Kabir. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ashraf Ul Kabir. The network helps show where Ashraf Ul Kabir may publish in the future.

Co-authorship network of co-authors of Ashraf Ul Kabir

This figure shows the co-authorship network connecting the top 25 collaborators of Ashraf Ul Kabir. A scholar is included among the top collaborators of Ashraf Ul Kabir based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ashraf Ul Kabir. Ashraf Ul Kabir is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Kabir, Ashraf Ul, et al.. (2025). Linking tumour angiogenesis and tumour immunity. Nature reviews. Immunology. 26(1). 35–51. 5 indexed citations
2.
Kabir, Ashraf Ul, Jun Wu, Karen Krchma, et al.. (2024). ZBTB46 coordinates angiogenesis and immunity to control tumor outcome. Nature Immunology. 25(9). 1546–1554. 7 indexed citations
3.
Wu, Jun, Changxu Fan, Ashraf Ul Kabir, et al.. (2024). Baf155 controls hematopoietic differentiation and regeneration through chromatin priming. Cell Reports. 43(8). 114558–114558. 1 indexed citations
4.
Kabir, Ashraf Ul, et al.. (2023). Conserved angio-immune subtypes of the tumor microenvironment predict response to immune checkpoint blockade therapy. Cell Reports Medicine. 4(1). 100896–100896. 19 indexed citations
5.
Kabir, Ashraf Ul, Jun Wu, Karen Krchma, et al.. (2023). Tumor-derived interleukin-1α and leukemia inhibitory factor promote extramedullary hematopoiesis. PLoS Biology. 21(5). e3001746–e3001746. 6 indexed citations
6.
Kabir, Ashraf Ul, Dong Hun Lee, Xiaoli Wang, et al.. (2021). Dual role of endothelial Myct1 in tumor angiogenesis and tumor immunity. Science Translational Medicine. 13(583). 53 indexed citations
7.
Kabir, Ashraf Ul, Tae‐Jin Lee, Hua Pan, et al.. (2018). Requisite endothelial reactivation and effective siRNA nanoparticle targeting of Etv2/Er71 in tumor angiogenesis. JCI Insight. 3(8). 18 indexed citations
8.
Oladipupo, Sunday S., Ashraf Ul Kabir, Craig S. Smith, Kyunghee Choi, & David M. Ornitz. (2018). Impaired tumor growth and angiogenesis in mice heterozygous for Vegfr2 (Flk1). Scientific Reports. 8(1). 14724–14724. 21 indexed citations
10.
Hasan, Md Nazmul, et al.. (2017). PEG modification of Amorfrutin B from Amorpha fructicosa increases gastric absorption, circulation half-life and glucose uptake by T3T-L1 adipocytes. Biomedicine & Pharmacotherapy. 95. 513–519. 4 indexed citations
12.
Kabir, Ashraf Ul, et al.. (2014). Ethanolic Extract of Butea monosperma Leaves Elevate Blood Insulin Level in Type 2 Diabetic Rats, Stimulate Insulin Secretion in Isolated Rat Islets, and Enhance Hepatic Glycogen Formation. Evidence-based Complementary and Alternative Medicine. 2014(1). 356290–356290. 6 indexed citations
13.
Kabir, Ashraf Ul, et al.. (2014). Anti-hyperglycemic activity of Centella asiatica is partly mediated by carbohydrase inhibition and glucose-fiber binding. BMC Complementary and Alternative Medicine. 14(1). 31–31. 63 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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