Arsalan Yousuf

456 total citations
14 papers, 321 citations indexed

About

Arsalan Yousuf is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Arsalan Yousuf has authored 14 papers receiving a total of 321 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 8 papers in Cellular and Molecular Neuroscience and 4 papers in Physiology. Recurrent topics in Arsalan Yousuf's work include Pharmacological Receptor Mechanisms and Effects (6 papers), Nicotinic Acetylcholine Receptors Study (6 papers) and Ion channel regulation and function (6 papers). Arsalan Yousuf is often cited by papers focused on Pharmacological Receptor Mechanisms and Effects (6 papers), Nicotinic Acetylcholine Receptors Study (6 papers) and Ion channel regulation and function (6 papers). Arsalan Yousuf collaborates with scholars based in Australia, United States and Austria. Arsalan Yousuf's co-authors include Stefan Boehm, MacDonald J. Christie, Stefan Schulz, Elke Miess, Klaus Schicker, Cornelius Krasel, Martin G. Goldner, Yunshi Yang, Moritz Bünemann and Meritxell Canals and has published in prestigious journals such as Pain, Journal of Medicinal Chemistry and Journal of Neurochemistry.

In The Last Decade

Arsalan Yousuf

12 papers receiving 318 citations

Peers

Arsalan Yousuf
Hye Sook Ahn South Korea
Wakako Fujita United States
Heather Guthrie United States
Shaifali Bhalla United States
Lin Pei China
Arsalan Yousuf
Citations per year, relative to Arsalan Yousuf Arsalan Yousuf (= 1×) peers M. Carmen Ruiz‐Cantero

Countries citing papers authored by Arsalan Yousuf

Since Specialization
Citations

This map shows the geographic impact of Arsalan Yousuf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Arsalan Yousuf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Arsalan Yousuf more than expected).

Fields of papers citing papers by Arsalan Yousuf

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Arsalan Yousuf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Arsalan Yousuf. The network helps show where Arsalan Yousuf may publish in the future.

Co-authorship network of co-authors of Arsalan Yousuf

This figure shows the co-authorship network connecting the top 25 collaborators of Arsalan Yousuf. A scholar is included among the top collaborators of Arsalan Yousuf based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Arsalan Yousuf. Arsalan Yousuf is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
2.
Tae, Han‐Shen, Marcelo O. Ortells, Arsalan Yousuf, et al.. (2024). Tabernanthalog and ibogainalog inhibit the α7 and α9α10 nicotinic acetylcholine receptors via different mechanisms and with higher potency than the GABAA receptor and CaV2.2 channel. Biochemical Pharmacology. 223. 116183–116183. 3 indexed citations
3.
Li, Xiao, Han‐Shen Tae, Chen Shen, et al.. (2024). Dual Antagonism of α9α10 nAChR and GABAB Receptor-Coupled CaV2.2 Channels by an Analgesic αO-Conotoxin Analogue. Journal of Medicinal Chemistry. 67(2). 971–987. 4 indexed citations
5.
Wang, Shuo, Peter H. Bartels, Cong Zhao, et al.. (2022). A 4/8 Subtype α-Conotoxin Vt1.27 Inhibits N-Type Calcium Channels With Potent Anti-Allodynic Effect. Frontiers in Pharmacology. 13. 881732–881732. 1 indexed citations
6.
7.
Arias, Hugo R., Han‐Shen Tae, Laura Micheli, et al.. (2020). Coronaridine congeners decrease neuropathic pain in mice and inhibit α9α10 nicotinic acetylcholine receptors and CaV2.2 channels. Neuropharmacology. 175. 108194–108194. 21 indexed citations
8.
Arias, Hugo R., Carla Ghelardini, Elena Lucarini, et al.. (2020). (E)-3-Furan-2-yl-N-p-tolyl-acrylamide and its Derivative DM489 Decrease Neuropathic Pain in Mice Predominantly by α7 Nicotinic Acetylcholine Receptor Potentiation. ACS Chemical Neuroscience. 11(21). 3603–3614. 16 indexed citations
9.
Rawling, Tristan, Sarasa A. Mohammadi, Subhodeep Sarker, et al.. (2019). Development of an N-Acyl Amino Acid That Selectively Inhibits the Glycine Transporter 2 To Produce Analgesia in a Rat Model of Chronic Pain. Journal of Medicinal Chemistry. 62(5). 2466–2484. 28 indexed citations
10.
Miess, Elke, Arisbel B. Gondin, Arsalan Yousuf, et al.. (2018). Multisite phosphorylation is required for sustained interaction with GRKs and arrestins during rapid μ-opioid receptor desensitization. Science Signaling. 11(539). 91 indexed citations
11.
Yousuf, Arsalan, et al.. (2016). Control of sensory neuron excitability by serotonin involves 5HT2C receptors and Ca2+-activated chloride channels. Neuropharmacology. 110(Pt A). 277–286. 32 indexed citations
12.
Yousuf, Arsalan, Elke Miess, Setareh Sianati, et al.. (2015). Role of Phosphorylation Sites in Desensitization of µ-Opioid Receptor. Molecular Pharmacology. 88(4). 825–835. 31 indexed citations
13.
Yousuf, Arsalan, et al.. (2011). Nucleotides control the excitability of sensory neurons via two P2Y receptors and a bifurcated signaling cascade. Pain. 152(8). 1899–1908. 36 indexed citations
14.
Dorostkar, Mario M., et al.. (2011). Concomitant facilitation of GABAA receptors and KV7 channels by the non‐opioid analgesic flupirtine. British Journal of Pharmacology. 166(5). 1631–1642. 48 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026