Ariel Kamsler
- Molecular Biology
- Cellular and Molecular Neuroscience top 5%
- Neurology top 5%
- Physiology
- Biological Psychiatry top 5%
- Co-authors
- Menahem SegalAvi AvitalGal Richter‐LevinInbal GoshenKerstin IverfeldtRaz YirmiyaAri BarzilaiA. Hochman
- Topics
- Neuroscience and Neuropharmacology Research (6 papers)Mitochondrial Function and Pathology (4 papers)Neuroinflammation and Neurodegeneration Mechanisms (4 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of NeuroscienceAntioxidants and Redox Signaling
- Partner nations
- IsraelUnited States
In The Last Decade
Ariel Kamsler
11 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 82
- Molecular Biology 503
- Cellular and Molecular Neuroscience 377
- Neurology 296
- Physiology 176
- Biological Psychiatry 131
Countries citing papers authored by Ariel Kamsler
This map shows the geographic impact of Ariel Kamsler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ariel Kamsler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ariel Kamsler more than expected).
Fields of papers citing papers by Ariel Kamsler
This network shows the impact of papers produced by Ariel Kamsler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ariel Kamsler. The network helps show where Ariel Kamsler may publish in the future.
Co-authorship network of co-authors of Ariel Kamsler
This figure shows the co-authorship network connecting the top 25 collaborators of Ariel Kamsler. A scholar is included among the top collaborators of Ariel Kamsler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ariel Kamsler. Ariel Kamsler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 46 | |
| 3 | 17 | |
| 4 | 26 | |
| 5 | 38 | |
| 6 | 113 | |
| 7 | 293 | |
| 8 | 83 | |
| 9 | 159 | |
| 10 | Increased oxidative stress in ataxia telangiectasia evidenced by alterations in redox state of brains from Atm-deficient mice. | 203 |
| 11 | 42 |
About Ariel Kamsler
Ariel Kamsler is a scholar working on Neurology, Cellular and Molecular Neuroscience and Behavioral Neuroscience, having authored 11 papers that have together received 1.0k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (6 papers), Mitochondrial Function and Pathology (4 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). The work is most often cited by research in Biological Psychiatry (131 citations), Neurology (296 citations) and Behavioral Neuroscience (92 citations). Ariel Kamsler has collaborated with scholars based in Israel and United States. Frequent co-authors include Menahem Segal, Avi Avital, Gal Richter‐Levin, Inbal Goshen, Kerstin Iverfeldt, Raz Yirmiya, Ari Barzilai, A. Hochman, N. Stern and Yosef Shiloh. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and Antioxidants and Redox Signaling.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.