Anna Skottner

4.4k total citations
83 papers, 3.6k citations indexed

About

Anna Skottner is a scholar working on Endocrinology, Diabetes and Metabolism, Molecular Biology and Physiology. According to data from OpenAlex, Anna Skottner has authored 83 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 55 papers in Endocrinology, Diabetes and Metabolism, 26 papers in Molecular Biology and 20 papers in Physiology. Recurrent topics in Anna Skottner's work include Growth Hormone and Insulin-like Growth Factors (55 papers), Metabolism, Diabetes, and Cancer (12 papers) and Muscle metabolism and nutrition (10 papers). Anna Skottner is often cited by papers focused on Growth Hormone and Insulin-like Growth Factors (55 papers), Metabolism, Diabetes, and Cancer (12 papers) and Muscle metabolism and nutrition (10 papers). Anna Skottner collaborates with scholars based in Sweden, United States and Norway. Anna Skottner's co-authors include Hans‐Arne Hansson, Eva Jennische, Martin Kanje, Linda Fryklund, Björn Rozell, Anders Nilsson, Jörgen Isgaard, Olle Isaksson, Ross Clark and Annica Dahlström and has published in prestigious journals such as Science, Journal of Biological Chemistry and The Journal of Clinical Endocrinology & Metabolism.

In The Last Decade

Anna Skottner

83 papers receiving 3.5k citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Anna Skottner 1.6k 1.3k 590 551 520 83 3.6k
Martin L. Adamo 2.4k 1.5× 2.7k 2.1× 329 0.6× 630 1.1× 308 0.6× 107 5.2k
Florent Elefteriou 560 0.3× 2.2k 1.8× 618 1.0× 483 0.9× 386 0.7× 74 5.8k
Guck T. Ooi 2.0k 1.2× 1.7k 1.4× 290 0.5× 337 0.6× 125 0.2× 51 3.7k
Ernst‐Martin Füchtbauer 479 0.3× 2.9k 2.3× 370 0.6× 646 1.2× 408 0.8× 99 4.5k
Hugo Vankelecom 1.3k 0.8× 1.9k 1.5× 267 0.5× 342 0.6× 205 0.4× 118 4.3k
Jong Deok Ahn 661 0.4× 2.3k 1.8× 211 0.4× 365 0.7× 162 0.3× 11 4.5k
Gonzalo Martı́nez de la Escalera 1.1k 0.7× 1.3k 1.0× 409 0.7× 282 0.5× 104 0.2× 152 4.2k
Wenhan Chang 354 0.2× 2.2k 1.8× 340 0.6× 411 0.7× 418 0.8× 127 4.6k
Li Sun 1.1k 0.7× 2.3k 1.9× 131 0.2× 283 0.5× 203 0.4× 85 4.7k
Pedro Cuevas 649 0.4× 1.7k 1.3× 656 1.1× 436 0.8× 375 0.7× 167 4.0k

Countries citing papers authored by Anna Skottner

Since Specialization
Citations

This map shows the geographic impact of Anna Skottner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anna Skottner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anna Skottner more than expected).

Fields of papers citing papers by Anna Skottner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anna Skottner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anna Skottner. The network helps show where Anna Skottner may publish in the future.

Co-authorship network of co-authors of Anna Skottner

This figure shows the co-authorship network connecting the top 25 collaborators of Anna Skottner. A scholar is included among the top collaborators of Anna Skottner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anna Skottner. Anna Skottner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Skottner, Anna, C. Post, Anette Ocklind, et al.. (2003). Anti‐Inflammatory Potential of Melanocortin Receptor‐Directed Drugs. Annals of the New York Academy of Sciences. 994(1). 84–89. 7 indexed citations
4.
Böhm, Markus, Meinhard Schiller, Sonja Ständer, et al.. (2002). Evidence for Expression of Melanocortin-1 Receptor in Human Sebocytes In Vitro and In Situ. Journal of Investigative Dermatology. 118(3). 533–539. 93 indexed citations
5.
Wikberg, Jarl E. S., Ruta Muceniece, Ilona Mandrika, et al.. (2000). New aspects on the melanocortins and their receptors. Pharmacological Research. 42(5). 393–420. 274 indexed citations
6.
Malmlöf, Kjell, et al.. (1999). Effects of Human Growth Hormone on the Porto-Arterial Concentration Differences of Glucose and Amino Acids in the Newborn Piglet. Hormone and Metabolic Research. 31(1). 22–26. 3 indexed citations
7.
Sonnewald, Ursula, et al.. (1996). New Aspects of Lactate Metabolism: IGF-I and Insulin Regulate Mitochondrial Function in Cultured Brain Cells during Normoxia and Hypoxia. Developmental Neuroscience. 18(5-6). 443–448. 18 indexed citations
8.
Halvorsen, S, et al.. (1994). Recombinant human insulin‐like growth factor 1 (rh‐IGF‐1) stimulates erythropoiesis in adult, but not in newborn mice. Acta Physiologica Scandinavica. 151(1). 117–123. 7 indexed citations
9.
Amselgruber, W., Fred Sinowatz, D. Schams, & Anna Skottner. (1994). Immunohistochemical aspects of insulin-like growth factors I and II in the bovine corpus luteum. Reproduction. 101(2). 445–451. 29 indexed citations
10.
Halvorsen, S, et al.. (1993). Growth Hormone Treatment and Development of Malignancy: Recombinant Human Growth Hormone Does not Induce Leukemia in AKR/O-Mice. Pediatric Hematology and Oncology. 10(1). 41–47. 7 indexed citations
11.
Merchav, Shoshana, et al.. (1993). In‐vitro response of erythroid progenitors from children with thalassaemia major to human growth hormone and insulin‐like growth factor I. Clinical Endocrinology. 39(2). 207–211. 4 indexed citations
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Roholl, Paul, et al.. (1990). Expression of insulin‐like growth factor 1 in sarcomas. Histopathology. 16(5). 455–460. 19 indexed citations
14.
Skottner, Anna, et al.. (1990). Anabolic and Tissue Repair Functions of Recombinant Insulin‐Like Growth Factor I. Acta Paediatrica. 79(s367). 63–66. 38 indexed citations
15.
Hansson, Hans‐Arne, et al.. (1989). Somatomedin C in the pancreas of young and adult, normal and obese, hyperinsulinemic mice. Cell and Tissue Research. 255(2). 467–74. 11 indexed citations
16.
Ullman, M. David, Hala S. Alameddine, Anna Skottner, & Anders Oldfors. (1989). Effects of growth hormone on skeletal muscle. II. Studies on regeneration and denervation in adult rats. Acta Physiologica Scandinavica. 135(4). 537–543. 22 indexed citations
17.
Hansson, Hans‐Arne, Dan Edwall, Björn Löwenadler, et al.. (1988). Insulin‐like growth factor I in the pancreas of normal and diabetic adult rats. Acta Physiologica Scandinavica. 132(4). 569–576. 47 indexed citations
18.
Philipps, Anthony F., Bengt Persson, Kerstin Hall, et al.. (1988). The Effects of Biosynthetic Insulin-Like Growth Factor-1 Supplementation on Somatic Growth, Maturation, and Erythropoiesis on the Neonatal Rat. Pediatric Research. 23(3). 298–305. 73 indexed citations
19.
Westermark, Kerstin, et al.. (1988). Growth factors and the thyroid: effects of treatment for hyper- and hypothyroidism on serum IGF-I and urinary epidermal growth factor concentrations. European Journal of Endocrinology. 118(3). 415–421. 19 indexed citations
20.
Hansson, Hans‐Arne, Eva Jennische, & Anna Skottner. (1987). Regenerating endothelial cells express insulin-like growth factor-I immunoreactivity after arterial injury. Cell and Tissue Research. 250(3). 499–505. 107 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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