Andrew G. Clark
- Cell Biology top 1%
- Molecular Biology
- Biomedical Engineering top 5%
- Oncology top 5%
- Cancer Research top 10%
- Co-authors
- Danijela Matic VignjevicEwa K. PaluchKai DierkesGuillaume SalbreuxDavide A. D. CassaniPriyamvada ChughGuillaume CharrasMatthew B. Smith
- Topics
- Cellular Mechanics and Interactions (20 papers)Cancer Cells and Metastasis (6 papers)3D Printing in Biomedical Research (5 papers)
- Partner nations
- GermanyFranceUnited Kingdom
In The Last Decade
Andrew G. Clark
22 papers receiving 1.9k citations
Hit Papers
Peers
Comparison fields: 5 of 115
- Cell Biology 1.1k
- Molecular Biology 632
- Biomedical Engineering 591
- Oncology 522
- Cancer Research 184
Countries citing papers authored by Andrew G. Clark
This map shows the geographic impact of Andrew G. Clark's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andrew G. Clark with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andrew G. Clark more than expected).
Fields of papers citing papers by Andrew G. Clark
This network shows the impact of papers produced by Andrew G. Clark. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andrew G. Clark. The network helps show where Andrew G. Clark may publish in the future.
Co-authorship network of co-authors of Andrew G. Clark
This figure shows the co-authorship network connecting the top 25 collaborators of Andrew G. Clark. A scholar is included among the top collaborators of Andrew G. Clark based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andrew G. Clark. Andrew G. Clark is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 12 | |
| 2 | 1 | |
| 3 | 69 | |
| 4 | 8 | |
| 5 | 12 | |
| 6 | 19 | |
| 7 | 136 | |
| 8 | 33 | |
| 9 | 3 | |
| 10 | Actin cortex architecture regulates cell surface tensionbreakdown → | 290 |
| 11 | 231 | |
| 12 | 2 | |
| 13 | Modes of cancer cell invasion and the role of the microenvironmentbreakdown → | 570 |
| 14 | 97 | |
| 15 | 171 | |
| 16 | 5 | |
| 17 | 59 | |
| 18 | 105 | |
| 19 | 45 | |
| 20 | 3 |
About Andrew G. Clark
Andrew G. Clark is a scholar working on Cell Biology, Structural Biology and Biophysics, having authored 22 papers that have together received 1.9k indexed citations. Recurring topics across this work include Cellular Mechanics and Interactions (20 papers), Cancer Cells and Metastasis (6 papers) and 3D Printing in Biomedical Research (5 papers). The work is most often cited by research in Cell Biology (1.1k citations), Immunology and Allergy (145 citations) and Biophysics (119 citations). Andrew G. Clark has collaborated with scholars based in Germany, France and United Kingdom. Frequent co-authors include Danijela Matic Vignjevic, Ewa K. Paluch, Kai Dierkes, Guillaume Salbreux, Davide A. D. Cassani, Priyamvada Chugh, Guillaume Charras, Matthew B. Smith, Philippe P. Roux and Anan Ragab. Their work appears in journals such as Nature Communications, Nature Materials and The Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.