Amy Innes

597 total citations
7 papers, 465 citations indexed

About

Amy Innes is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Amy Innes has authored 7 papers receiving a total of 465 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Cellular and Molecular Neuroscience, 3 papers in Molecular Biology and 3 papers in Neurology. Recurrent topics in Amy Innes's work include Parkinson's Disease Mechanisms and Treatments (3 papers), Alzheimer's disease research and treatments (3 papers) and Amyotrophic Lateral Sclerosis Research (3 papers). Amy Innes is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (3 papers), Alzheimer's disease research and treatments (3 papers) and Amyotrophic Lateral Sclerosis Research (3 papers). Amy Innes collaborates with scholars based in United States, United Kingdom and Japan. Amy Innes's co-authors include Eileen McGowan, Suzanne J. Randle, Dennis W. Dickson, Wen-Lang Lin, Reza M. Salek, Piers C. Emson, Xin Yu, Brian C. Sweatman, Leonard Petrucelli and Michael Hutton and has published in prestigious journals such as Human Molecular Genetics, American Journal Of Pathology and Alzheimer s & Dementia.

In The Last Decade

Amy Innes

7 papers receiving 459 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Amy Innes United States	6	255	208	194	98	94	7	465
Julia van der Hoven Australia	11	275 1.1×	125 0.6×	198 1.0×	176 1.8×	130 1.4×	19	521
Younghwi Kwon South Korea	9	117 0.5×	214 1.0×	207 1.1×	54 0.6×	67 0.7×	13	432
Christopher J. Holler United States	11	356 1.4×	253 1.2×	307 1.6×	65 0.7×	95 1.0×	16	687
Sang‐Won Min United States	4	246 1.0×	122 0.6×	207 1.1×	63 0.6×	117 1.2×	4	514
Jean‐Jacques Hauw France	9	136 0.5×	145 0.7×	373 1.9×	127 1.3×	147 1.6×	10	541
Salomé McAllen United States	13	389 1.5×	124 0.6×	285 1.5×	126 1.3×	197 2.1×	17	665
Sheue-Houy Tyan United States	7	207 0.8×	76 0.4×	132 0.7×	99 1.0×	42 0.4×	9	347
Ariana Gatt United Kingdom	11	91 0.4×	136 0.7×	255 1.3×	113 1.2×	44 0.5×	15	423
Bilal Khalil United States	9	92 0.4×	184 0.9×	307 1.6×	103 1.1×	45 0.5×	13	476
Luis M. Guisasola Spain	13	192 0.8×	278 1.3×	264 1.4×	146 1.5×	153 1.6×	17	623

Countries citing papers authored by Amy Innes

Since Specialization

Citations

This map shows the geographic impact of Amy Innes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amy Innes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amy Innes more than expected).

Fields of papers citing papers by Amy Innes

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amy Innes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amy Innes. The network helps show where Amy Innes may publish in the future.

Co-authorship network of co-authors of Amy Innes

This figure shows the co-authorship network connecting the top 25 collaborators of Amy Innes. A scholar is included among the top collaborators of Amy Innes based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amy Innes. Amy Innes is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Kalmár, Bernadett, Amy Innes, Klaus Wanisch, et al.. (2017). Mitochondrial deficits and abnormal mitochondrial retrograde axonal transport play a role in the pathogenesis of mutant Hsp27-induced Charcot Marie Tooth Disease. Human Molecular Genetics. 26(17). 3313–3326. 44 indexed citations

2.

Rossor, Alexander M., Gabrielle L. Davidson, Julian Blake, et al.. (2012). A novel p.Glu175X premature stop mutation in the C‐terminal end of HSP27 is a cause of CMT2. Journal of the Peripheral Nervous System. 17(2). 201–205. 14 indexed citations

3.

Salek, Reza M., Jing Xia, Amy Innes, et al.. (2010). A metabolomic study of the CRND8 transgenic mouse model of Alzheimer's disease. Neurochemistry International. 56(8). 937–947. 124 indexed citations

4.

Ahmed, Zeshan, Sheng Hong, Ya-Fei Xu, et al.. (2010). Accelerated Lipofuscinosis and Ubiquitination in Granulin Knockout Mice Suggest a Role for Progranulin in Successful Aging. American Journal Of Pathology. 177(1). 311–324. 226 indexed citations

5.

Ahmed, Zeshan, Sheng Hong, Yafei Xu, et al.. (2010). O1‐07‐01: Accelerated lipofuscinosis and ubiquitination in granulin knockout mice suggests a role for progranulin in successful aging. Alzheimer s & Dementia. 6(4S_Part_3). 5 indexed citations

6.

Lewis, Jada, Zeshan Ahmed, Sheng Hong, et al.. (2009). O4‐06‐01: Neuropathological characterization of progranulin‐deficient mice: Accelerated lipofuscin accumulation suggests a role for progranulin in successful aging. Alzheimer s & Dementia. 5(4S_Part_6). 1 indexed citations

7.

Crook, Richard, Michael DeTure, Suzanne J. Randle, et al.. (2009). Overexpression of Wild-Type Murine Tau Results in Progressive Tauopathy and Neurodegeneration. American Journal Of Pathology. 175(4). 1598–1609. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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