Alon Bajayo

1.0k total citations
10 papers, 820 citations indexed

About

Alon Bajayo is a scholar working on Molecular Biology, Geriatrics and Gerontology and Oncology. According to data from OpenAlex, Alon Bajayo has authored 10 papers receiving a total of 820 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 2 papers in Geriatrics and Gerontology and 2 papers in Oncology. Recurrent topics in Alon Bajayo's work include Bone Metabolism and Diseases (3 papers), Estrogen and related hormone effects (2 papers) and Sirtuins and Resveratrol in Medicine (2 papers). Alon Bajayo is often cited by papers focused on Bone Metabolism and Diseases (3 papers), Estrogen and related hormone effects (2 papers) and Sirtuins and Resveratrol in Medicine (2 papers). Alon Bajayo collaborates with scholars based in Israel, United States and Hungary. Alon Bajayo's co-authors include Itai Bab, Raz Yirmiya, Valér Csernus, Malka Attar-Namdar, Esther Shohami, Inbal Goshen, Joseph Tam, Hanna Artsi, Einav Cohen‐Kfir and Irina Gurt and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Endocrinology and Journal of Bone and Mineral Research.

In The Last Decade

Alon Bajayo

10 papers receiving 801 citations

Peers

Alon Bajayo

MB

PHYSI

PHARM

OSM

GG

Hyunbae Kim United States

Jing Xiong China

Xin Feng United States

Paula Mera Spain

Christine F. Conover United States

Mika Takarada‐Iemata Japan

Ella Zeldich United States

Scott C. Supowit United States

Liansheng Gao China

Hyunbae Kim United States

Alon Bajayo

304 ×0.9

MB

195 ×1.3

PHYSI

45 ×0.3

PHARM

76 ×0.6

OSM

36 ×0.4

GG

Citations per year, relative to Alon Bajayo Alon Bajayo (= 1×) peers Hyunbae Kim

Countries citing papers authored by Alon Bajayo

Since Specialization

Citations

This map shows the geographic impact of Alon Bajayo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alon Bajayo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alon Bajayo more than expected).

Fields of papers citing papers by Alon Bajayo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alon Bajayo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alon Bajayo. The network helps show where Alon Bajayo may publish in the future.

Co-authorship network of co-authors of Alon Bajayo

This figure shows the co-authorship network connecting the top 25 collaborators of Alon Bajayo. A scholar is included among the top collaborators of Alon Bajayo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alon Bajayo. Alon Bajayo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Kogan, Natalya M., Eitan Melamed, Aviva Breuer, et al.. (2015). Cannabidiol, a Major Non-Psychotropic Cannabis Constituent Enhances Fracture Healing and Stimulates Lysyl Hydroxylase Activity in Osteoblasts. Journal of Bone and Mineral Research. 30(10). 1905–1913. 78 indexed citations

2.

Artsi, Hanna, Einav Cohen‐Kfir, Irina Gurt, et al.. (2014). The Sirtuin1 Activator SRT3025 Down-Regulates Sclerostin and Rescues Ovariectomy-Induced Bone Loss and Biomechanical Deterioration in Female Mice. Endocrinology. 155(9). 3508–3515. 59 indexed citations

3.

Bajayo, Alon, Ádám Dénes, Vardit Kram, et al.. (2012). Skeletal parasympathetic innervation communicates central IL-1 signals regulating bone mass accrual. Proceedings of the National Academy of Sciences. 109(38). 15455–15460. 144 indexed citations

4.

Cohen‐Kfir, Einav, Hanna Artsi, Avi Levin, et al.. (2011). Sirt1 Is a Regulator of Bone Mass and a Repressor of Sost Encoding for Sclerostin, a Bone Formation Inhibitor. Endocrinology. 152(12). 4514–4524. 155 indexed citations

5.

Smoum, Reem, Bo Tan, Garry Milman, et al.. (2010). Oleoyl Serine, an Endogenous Regulator of Skeletal Mass. Bone. 46. S81–S81. 1 indexed citations

6.

Smoum, Reem, Bo Tan, Garry Milman, et al.. (2010). Oleoyl serine, an endogenous N -acyl amide, modulates bone remodeling and mass. Proceedings of the National Academy of Sciences. 107(41). 17710–17715. 72 indexed citations

7.

Kasher, Roni, Alon Bajayo, Yankel Gabet, et al.. (2009). Restrain of bone growth by Estrogen-Mimetic Peptide-1 (EMP-1): A micro-computed tomographic study. Peptides. 30(6). 1181–1186. 4 indexed citations

8.

Rozen, Nimrod, Tova Bick, Alon Bajayo, et al.. (2009). Transplanted blood-derived endothelial progenitor cells (EPC) enhance bridging of sheep tibia critical size defects. Bone. 45(5). 918–924. 77 indexed citations

9.

Yirmiya, Raz, Inbal Goshen, Alon Bajayo, et al.. (2006). Depression induces bone loss through stimulation of the sympathetic nervous system. Proceedings of the National Academy of Sciences. 103(45). 16876–16881. 174 indexed citations

10.

Bajayo, Alon, Inbal Goshen, Valér Csernus, et al.. (2005). Central IL-1 receptor signaling regulates bone growth and mass. Proceedings of the National Academy of Sciences. 102(36). 12956–12961. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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