Allison M. Eaton

586 total citations
7 papers, 485 citations indexed

About

Allison M. Eaton is a scholar working on Molecular Biology, Surgery and Rheumatology. According to data from OpenAlex, Allison M. Eaton has authored 7 papers receiving a total of 485 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Molecular Biology, 2 papers in Surgery and 2 papers in Rheumatology. Recurrent topics in Allison M. Eaton's work include Esophageal and GI Pathology (2 papers), Folate and B Vitamins Research (2 papers) and Glycosylation and Glycoproteins Research (1 paper). Allison M. Eaton is often cited by papers focused on Esophageal and GI Pathology (2 papers), Folate and B Vitamins Research (2 papers) and Glycosylation and Glycoproteins Research (1 paper). Allison M. Eaton collaborates with scholars based in United States. Allison M. Eaton's co-authors include Robert C. Millikan, Scott Winkel, Temitope O. Keku, Christopher Martin, Robert D Sandler, Gary S. Pittman, Chiu‐Kit Tse, Ruth M. Lunn, Douglas A. Bell and Eric J. Duell and has published in prestigious journals such as Journal of Medicinal Chemistry, Breast Cancer Research and Treatment and Mutation research. Fundamental and molecular mechanisms of mutagenesis.

In The Last Decade

Allison M. Eaton

7 papers receiving 469 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Allison M. Eaton United States 7 271 147 135 100 77 7 485
Elisabeth del Río Spain 14 267 1.0× 71 0.5× 221 1.6× 81 0.8× 86 1.1× 24 633
Judit Kralovánszky Hungary 15 138 0.5× 74 0.5× 266 2.0× 71 0.7× 88 1.1× 49 511
Arno van Rooij Netherlands 12 398 1.5× 120 0.8× 67 0.5× 89 0.9× 18 0.2× 16 630
P. Van Erps Belgium 9 52 0.2× 78 0.5× 112 0.8× 69 0.7× 39 0.5× 19 550
Isildinha Reis United States 9 513 1.9× 40 0.3× 74 0.5× 41 0.4× 45 0.6× 14 700
Esteban E. Sierra United States 12 233 0.9× 286 1.9× 120 0.9× 25 0.3× 13 0.2× 12 567
Seiji Arai Japan 17 285 1.1× 17 0.1× 201 1.5× 130 1.3× 38 0.5× 69 732
G. Monfette Canada 10 154 0.6× 33 0.2× 91 0.7× 112 1.1× 20 0.3× 17 886
Fábio Martins Laginha Brazil 9 105 0.4× 70 0.5× 166 1.2× 30 0.3× 45 0.6× 14 372
M Fontaine Belgium 8 184 0.7× 23 0.2× 118 0.9× 59 0.6× 36 0.5× 16 482

Countries citing papers authored by Allison M. Eaton

Since Specialization
Citations

This map shows the geographic impact of Allison M. Eaton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Allison M. Eaton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Allison M. Eaton more than expected).

Fields of papers citing papers by Allison M. Eaton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Allison M. Eaton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Allison M. Eaton. The network helps show where Allison M. Eaton may publish in the future.

Co-authorship network of co-authors of Allison M. Eaton

This figure shows the co-authorship network connecting the top 25 collaborators of Allison M. Eaton. A scholar is included among the top collaborators of Allison M. Eaton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Allison M. Eaton. Allison M. Eaton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Butler, Lesley M., Robert C. Millikan, Rashmi Sinha, et al.. (2007). Modification by N-acetyltransferase 1 genotype on the association between dietary heterocyclic amines and colon cancer in a multiethnic study. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 638(1-2). 162–174. 36 indexed citations
2.
Eaton, Allison M., Robert S. Sandler, John M. Carethers, et al.. (2005). 5,10-Methylenetetrahydrofolate Reductase 677 and 1298 Polymorphisms, Folate Intake, and Microsatellite Instability in Colon Cancer. Cancer Epidemiology Biomarkers & Prevention. 14(8). 2023–2029. 36 indexed citations
3.
Millikan, Robert C., Allison M. Eaton, M. Elizabeth Hodgson, et al.. (2003). HER2 Codon 655 Polymorphism and Risk of Breast Cancer in African Americans and Whites. Breast Cancer Research and Treatment. 79(3). 355–364. 65 indexed citations
4.
Keku, Temitope O., Robert C. Millikan, Scott Winkel, et al.. (2002). 5,10-Methylenetetrahydrofolate reductase codon 677 and 1298 polymorphisms and colon cancer in African Americans and whites.. PubMed. 11(12). 1611–21. 155 indexed citations
5.
Duell, Eric J., Robert C. Millikan, Gary S. Pittman, et al.. (2001). Polymorphisms in the DNA repair gene XRCC1 and breast cancer.. PubMed. 10(3). 217–22. 131 indexed citations
6.
Gilmore, John H., Cindy P. Lawler, Allison M. Eaton, & Richard B. Mailman. (1993). Postmortem stability of dopamine D1 receptor mRNA and D1 receptors. Molecular Brain Research. 18(4). 290–296. 24 indexed citations
7.
Bishop, John E., et al.. (1991). Synthesis and in vitro evaluation of 2,3-dimethoxy-5-(fluoroalkyl)-substituted benzamides: high-affinity ligands for CNS dopamine D2 receptors. Journal of Medicinal Chemistry. 34(5). 1612–1624. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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