Allan D. Duby
- Immunology top 5%
- Molecular Biology
- Radiology, Nuclear Medicine and Imaging top 5%
- Oncology
- Cardiology and Cardiovascular Medicine top 10%
- Co-authors
- J.G. SeidmanJonathan G. SeidmanCornelis MurrePeter E. LipskyJ G SeidmanJohn A. SmithRobert M. GrahamC J Homcy
- Topics
- T-cell and B-cell Immunology (15 papers)Monoclonal and Polyclonal Antibodies Research (10 papers)CAR-T cell therapy research (7 papers)
- Partner nations
- United StatesAustraliaGermany
In The Last Decade
Allan D. Duby
25 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 82
- Immunology 605
- Molecular Biology 285
- Radiology, Nuclear Medicine and Imaging 234
- Oncology 173
- Cardiology and Cardiovascular Medicine 151
Countries citing papers authored by Allan D. Duby
This map shows the geographic impact of Allan D. Duby's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Allan D. Duby with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Allan D. Duby more than expected).
Fields of papers citing papers by Allan D. Duby
This network shows the impact of papers produced by Allan D. Duby. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Allan D. Duby. The network helps show where Allan D. Duby may publish in the future.
Co-authorship network of co-authors of Allan D. Duby
This figure shows the co-authorship network connecting the top 25 collaborators of Allan D. Duby. A scholar is included among the top collaborators of Allan D. Duby based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Allan D. Duby. Allan D. Duby is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 10 | |
| 2 | 28 | |
| 3 | 2 | |
| 4 | 68 | |
| 5 | 12 | |
| 6 | 119 | |
| 7 | 19 | |
| 8 | 23 | |
| 9 | 40 | |
| 10 | 30 | |
| 11 | 15 | |
| 12 | 119 | |
| 13 | 11 | |
| 14 | 25 | |
| 15 | 36 | |
| 16 | 63 | |
| 17 | 35 | |
| 18 | 35 | |
| 19 | Detection of a Frequent Restriction Fragment Length Polymorphism in the Human T Cell Antigen Receptor Beta Chain Locus | 7 |
| 20 | 201 |
About Allan D. Duby
Allan D. Duby is a scholar working on Immunology, Radiology, Nuclear Medicine and Imaging and Oncology, having authored 25 papers that have together received 1.1k indexed citations. Recurring topics across this work include T-cell and B-cell Immunology (15 papers), Monoclonal and Polyclonal Antibodies Research (10 papers) and CAR-T cell therapy research (7 papers). The work is most often cited by research in Immunology (605 citations), Radiology, Nuclear Medicine and Imaging (234 citations) and Immunology and Allergy (57 citations). Allan D. Duby has collaborated with scholars based in United States, Australia and Germany. Frequent co-authors include J.G. Seidman, J.G. Seidman, Jonathan G. Seidman, Cornelis Murre, Peter E. Lipsky, J G Seidman, John A. Smith, Robert M. Graham, C J Homcy and Christine E. Seidman. Their work appears in journals such as Nature, Science and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.