Alice Dreser

701 total citations
8 papers, 575 citations indexed

About

Alice Dreser is a scholar working on Neurology, Molecular Biology and Genetics. According to data from OpenAlex, Alice Dreser has authored 8 papers receiving a total of 575 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Neurology, 3 papers in Molecular Biology and 3 papers in Genetics. Recurrent topics in Alice Dreser's work include Amyotrophic Lateral Sclerosis Research (3 papers), Pharmacological Receptor Mechanisms and Effects (2 papers) and Neurogenetic and Muscular Disorders Research (2 papers). Alice Dreser is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (3 papers), Pharmacological Receptor Mechanisms and Effects (2 papers) and Neurogenetic and Muscular Disorders Research (2 papers). Alice Dreser collaborates with scholars based in Germany, Netherlands and Switzerland. Alice Dreser's co-authors include Antonio Sechi, Bernd Denecke, Giulio Abagnale, Arnold Gillner, Vu Hoa Nguyen, Bernd Hoffmann, Rudolf Merkel, Sylvia Joussen, Wolfgang Wagner and Michael F. Steger and has published in prestigious journals such as Biomaterials, Cell Death and Differentiation and Cell Death and Disease.

In The Last Decade

Alice Dreser

8 papers receiving 569 citations

Peers

Alice Dreser

BE

MB

CB

BIOMA

NEURO

Jackson G. DeStefano United States

Mario Bitsche Austria

Shirin Ilkhanizadeh United States

Héctor Martínez United States

Daisuke Iejima Japan

Hongyun Huang China

Albert Cheng United States

Chiara Argentati Italy

Jiayi Peng China

Max I. Bogorad United States

Jackson G. DeStefano United States

Alice Dreser

452 ×1.8

BE

274 ×1.5

MB

79 ×0.5

CB

85 ×0.9

BIOMA

49 ×0.6

NEURO

Citations per year, relative to Alice Dreser Alice Dreser (= 1×) peers Jackson G. DeStefano

Countries citing papers authored by Alice Dreser

Since Specialization

Citations

This map shows the geographic impact of Alice Dreser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alice Dreser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alice Dreser more than expected).

Fields of papers citing papers by Alice Dreser

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alice Dreser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alice Dreser. The network helps show where Alice Dreser may publish in the future.

Co-authorship network of co-authors of Alice Dreser

This figure shows the co-authorship network connecting the top 25 collaborators of Alice Dreser. A scholar is included among the top collaborators of Alice Dreser based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alice Dreser. Alice Dreser is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Tripathi, Priyanka, Haihong Guo, Alice Dreser, et al.. (2021). Pathomechanisms of ALS8: altered autophagy and defective RNA binding protein (RBP) homeostasis due to the VAPB P56S mutation. Cell Death and Disease. 12(5). 466–466. 16 indexed citations

2.

Nikoubashman, Omid, Marc‐Alexander Brockmann, Bernd Sellhaus, et al.. (2018). Development of a Polymer‐Based Biodegradable Neurovascular Stent Prototype: A Preliminary In Vitro and In Vivo Study. Macromolecular Bioscience. 18(7). e1700292–e1700292. 11 indexed citations

3.

Dreser, Alice, Jan Tilmann Vollrath, Antonio Sechi, et al.. (2017). The ALS-linked E102Q mutation in Sigma receptor-1 leads to ER stress-mediated defects in protein homeostasis and dysregulation of RNA-binding proteins. Cell Death and Differentiation. 24(10). 1655–1671. 80 indexed citations

4.

Paul, N, Bernd Denecke, Bong‐Sung Kim, et al.. (2017). The effect of mechanical stress on the proliferation, adipogenic differentiation and gene expression of human adipose-derived stem cells. Journal of Tissue Engineering and Regenerative Medicine. 12(1). 276–284. 32 indexed citations

5.

Jesse, Christopher Marvin, Akila Chandrasekar, Cynthia M. Simon, et al.. (2017). ALS‐Associated Endoplasmic Reticulum Proteins in Denervated Skeletal Muscle: Implications for Motor Neuron Disease Pathology. Brain Pathology. 27(6). 781–794. 16 indexed citations

6.

Abagnale, Giulio, Michael F. Steger, Vu Hoa Nguyen, et al.. (2015). Surface topography enhances differentiation of mesenchymal stem cells towards osteogenic and adipogenic lineages. Biomaterials. 61. 316–326. 325 indexed citations

7.

Vollrath, Jan Tilmann, Antonio Sechi, Alice Dreser, et al.. (2014). Loss of function of the ALS protein SigR1 leads to ER pathology associated with defective autophagy and lipid raft disturbances. Cell Death and Disease. 5(6). e1290–e1290. 82 indexed citations

8.

Goswami, Anand, Akila Chandrasekar, Jan Tilmann Vollrath, et al.. (2014). Accumulation of STIM1 is associated with the degenerative muscle fibre phenotype in ALS and other neurogenic atrophies. Neuropathology and Applied Neurobiology. 41(3). 304–318. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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