Ali J. Masud

1.4k total citations · 1 hit paper
7 papers, 1.2k citations indexed

About

Ali J. Masud is a scholar working on Molecular Biology, Epidemiology and Clinical Biochemistry. According to data from OpenAlex, Ali J. Masud has authored 7 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 3 papers in Epidemiology and 2 papers in Clinical Biochemistry. Recurrent topics in Ali J. Masud's work include Mitochondrial Function and Pathology (6 papers), Metabolism and Genetic Disorders (2 papers) and Autophagy in Disease and Therapy (2 papers). Ali J. Masud is often cited by papers focused on Mitochondrial Function and Pathology (6 papers), Metabolism and Genetic Disorders (2 papers) and Autophagy in Disease and Therapy (2 papers). Ali J. Masud collaborates with scholars based in Finland, United States and Canada. Ali J. Masud's co-authors include Saquib A. Lakhani, Richard A. Flavell, Wajahat Z. Mehal, Irteza Inayat, George A. Porter, Carmen J. Booth, Keisuke Kuida, Alexander J. Kastaniotis, Razqallah Hakem and Alexander Mohapatra and has published in prestigious journals such as Science, Journal of Biological Chemistry and Biochemical Journal.

In The Last Decade

Ali J. Masud

7 papers receiving 1.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Caspases 3 and 7: Key Mediators of Mitochondrial Events of Apoptosis

2006 980 citations Saquib A. Lakhani, Ali J. Masud et al. Science profile →

Peers

Ali J. Masud

MB

EPIDE

IMMUN

CB

ONCOL

Irteza Inayat United States

Swati Dawar Australia

Sonia Shalini Australia

Chi Hin Cho Hong Kong

Émilie Hollville United States

Aida Peña‐Blanco Germany

Aarne Fleischer Spain

Shaohua Li China

Claudia Giampietri Italy

Irteza Inayat United States

Ali J. Masud

663 ×0.9

MB

244 ×1.2

EPIDE

185 ×1.1

IMMUN

93 ×0.6

CB

118 ×1.0

ONCOL

Citations per year, relative to Ali J. Masud Ali J. Masud (= 1×) peers Irteza Inayat

Countries citing papers authored by Ali J. Masud

Since Specialization

Citations

This map shows the geographic impact of Ali J. Masud's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ali J. Masud with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ali J. Masud more than expected).

Fields of papers citing papers by Ali J. Masud

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ali J. Masud. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ali J. Masud. The network helps show where Ali J. Masud may publish in the future.

Co-authorship network of co-authors of Ali J. Masud

This figure shows the co-authorship network connecting the top 25 collaborators of Ali J. Masud. A scholar is included among the top collaborators of Ali J. Masud based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ali J. Masud. Ali J. Masud is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

7 of 7 papers shown

1.

Shvetsova, Antonina, Ali J. Masud, Ulrich Bergmann, et al.. (2021). A hunt for OM45 synthetic petite interactions in Saccharomyces cerevisiae reveals a role for Miro GTPase Gem1p in cristae structure maintenance. MicrobiologyOpen. 10(5). e1238–e1238. 2 indexed citations

2.

Masud, Ali J., et al.. (2019). Mitochondrial acyl carrier protein (ACP) at the interface of metabolic state sensing and mitochondrial function. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1866(12). 118540–118540. 48 indexed citations

3.

Monteuuis, Geoffray, et al.. (2017). A conserved mammalian mitochondrial isoform of acetyl-CoA carboxylase ACC1 provides the malonyl-CoA essential for mitochondrial biogenesis in tandem with ACSF3. Biochemical Journal. 474(22). 3783–3797. 25 indexed citations

4.

Nair, R., Kaija J. Autio, Ali J. Masud, et al.. (2017). Genetic modifications of Mecr reveal a role for mitochondrial 2-enoyl-CoA/ACP reductase in placental development in mice. Human Molecular Genetics. 26(11). 2104–2117. 21 indexed citations

5.

Masud, Ali J., et al.. (2007). Endoplasmic Reticulum Stress-induced Death of Mouse Embryonic Fibroblasts Requires the Intrinsic Pathway of Apoptosis. Journal of Biological Chemistry. 282(19). 14132–14139. 87 indexed citations

6.

Qamar, Amir, Shehzad Z. Sheikh, Ali J. Masud, et al.. (2006). In Vitro and In Vivo Protection of Stellate Cells from Apoptosis by Leptin. Digestive Diseases and Sciences. 51(10). 1697–1705. 30 indexed citations

7.

Lakhani, Saquib A., Ali J. Masud, Keisuke Kuida, et al.. (2006). Caspases 3 and 7: Key Mediators of Mitochondrial Events of Apoptosis. Science. 311(5762). 847–851. 980 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact