Alexes C. Daquinag

2.4k total citations
33 papers, 1.9k citations indexed

About

Alexes C. Daquinag is a scholar working on Physiology, Molecular Biology and Cell Biology. According to data from OpenAlex, Alexes C. Daquinag has authored 33 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 10 papers in Molecular Biology and 9 papers in Cell Biology. Recurrent topics in Alexes C. Daquinag's work include Adipose Tissue and Metabolism (14 papers), Adipokines, Inflammation, and Metabolic Diseases (7 papers) and Mesenchymal stem cell research (6 papers). Alexes C. Daquinag is often cited by papers focused on Adipose Tissue and Metabolism (14 papers), Adipokines, Inflammation, and Metabolic Diseases (7 papers) and Mesenchymal stem cell research (6 papers). Alexes C. Daquinag collaborates with scholars based in United States, Japan and Canada. Alexes C. Daquinag's co-authors include Mikhail G. Kolonin, Felipe Amaya‐Manzanares, Paul J. Simmons, Yan Zhang, Glauco R. Souza, Jeannette Kunz, Chieh Tseng, Zhan‐Guo Gao, Renata Pasqualini and Wadih Arap and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and The Journal of Cell Biology.

In The Last Decade

Alexes C. Daquinag

31 papers receiving 1.8k citations

Peers

Alexes C. Daquinag
Shigeki Sugii Singapore
Hao Ding Canada
Yoo‐Wook Kwon South Korea
Brian C. Capell United States
Chanhee Kang South Korea
Yun Zhong China
Hwan‐Woo Park South Korea
Shigeki Sugii Singapore
Alexes C. Daquinag
Citations per year, relative to Alexes C. Daquinag Alexes C. Daquinag (= 1×) peers Shigeki Sugii

Countries citing papers authored by Alexes C. Daquinag

Since Specialization
Citations

This map shows the geographic impact of Alexes C. Daquinag's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alexes C. Daquinag with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alexes C. Daquinag more than expected).

Fields of papers citing papers by Alexes C. Daquinag

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alexes C. Daquinag. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alexes C. Daquinag. The network helps show where Alexes C. Daquinag may publish in the future.

Co-authorship network of co-authors of Alexes C. Daquinag

This figure shows the co-authorship network connecting the top 25 collaborators of Alexes C. Daquinag. A scholar is included among the top collaborators of Alexes C. Daquinag based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alexes C. Daquinag. Alexes C. Daquinag is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Wareing, Nancy, Scott D. Collum, Minghua Wu, et al.. (2026). Adipocyte-Specific Deletion of Sine Oculis Homeobox Homolog 1 Inhibits Lipolysis and Reduces Skin Fibrosis. JCI Insight.
3.
Rupert, Joseph E., Alexes C. Daquinag, Yongmei Yu, et al.. (2024). Depletion of Adipose Stroma-Like Cancer-Associated Fibroblasts Potentiates Pancreatic Cancer Immunotherapy. Cancer Research Communications. 5(1). 5–12. 4 indexed citations
4.
Tovy, Ayala, Jaime M. Reyes, Linda Zhang, et al.. (2022). Constitutive loss of DNMT3A causes morbid obesity through misregulation of adipogenesis. eLife. 11. 15 indexed citations
5.
Gao, Zhan‐Guo, Aiping Lu, Alexes C. Daquinag, et al.. (2021). Partial Ablation of Non-Myogenic Progenitor Cells as a Therapeutic Approach to Duchenne Muscular Dystrophy. Biomolecules. 11(10). 1519–1519. 5 indexed citations
6.
Daquinag, Alexes C., Zhan‐Guo Gao, Renata Pasqualini, et al.. (2021). Fatty acid mobilization from adipose tissue is mediated by CD36 posttranslational modifications and intracellular trafficking. JCI Insight. 6(17). 61 indexed citations
7.
Zu, Yujiao, Ling Zhao, Hao Lei, et al.. (2021). Browning white adipose tissue using adipose stromal cell-targeted resveratrol-loaded nanoparticles for combating obesity. Journal of Controlled Release. 333. 339–351. 42 indexed citations
8.
Daquinag, Alexes C., et al.. (2020). Glycosaminoglycan Modification of Decorin Depends on MMP14 Activity and Regulates Collagen Assembly. Cells. 9(12). 2646–2646. 17 indexed citations
9.
Lin, Li‐Ling, Edward R. Kost, Philip T. Valente, et al.. (2020). PAI-1-Dependent Inactivation of SMAD4-Modulated Junction and Adhesion Complex in Obese Endometrial Cancer. Cell Reports. 33(2). 108253–108253. 15 indexed citations
10.
Tseng, Hubert, Alexes C. Daquinag, Glauco R. Souza, & Mikhail G. Kolonin. (2018). Three-Dimensional Magnetic Levitation Culture System Simulating White Adipose Tissue. Methods in molecular biology. 1773. 147–154. 23 indexed citations
11.
Daquinag, Alexes C., Ali Dadbin, Brad Snyder, et al.. (2017). Non-glycanated Decorin Is a Drug Target on Human Adipose Stromal Cells. Molecular Therapy — Oncolytics. 6. 1–9. 28 indexed citations
12.
Porter, Craig, David N. Herndon, Maria Chondronikola, et al.. (2016). Human and Mouse Brown Adipose Tissue Mitochondria Have Comparable UCP1 Function. Cell Metabolism. 24(2). 246–255. 100 indexed citations
13.
Daquinag, Alexes C., Chieh Tseng, Ahmad Salameh, et al.. (2014). Depletion of white adipocyte progenitors induces beige adipocyte differentiation and suppresses obesity development. Cell Death and Differentiation. 22(2). 351–363. 55 indexed citations
14.
Azhdarinia, Ali, Alexes C. Daquinag, Chieh Tseng, et al.. (2013). A peptide probe for targeted brown adipose tissue imaging. Nature Communications. 4(1). 2472–2472. 55 indexed citations
15.
Zhang, Yan, Alexes C. Daquinag, Felipe Amaya‐Manzanares, et al.. (2012). Stromal Progenitor Cells from Endogenous Adipose Tissue Contribute to Pericytes and Adipocytes That Populate the Tumor Microenvironment. Cancer Research. 72(20). 5198–5208. 181 indexed citations
16.
Daquinag, Alexes C., Glauco R. Souza, & Mikhail G. Kolonin. (2012). Adipose Tissue Engineering in Three-Dimensional Levitation Tissue Culture System Based on Magnetic Nanoparticles. Tissue Engineering Part C Methods. 19(5). 336–344. 144 indexed citations
17.
Daquinag, Alexes C., Yan Zhang, Felipe Amaya‐Manzanares, Paul J. Simmons, & Mikhail G. Kolonin. (2011). An Isoform of Decorin Is a Resistin Receptor on the Surface of Adipose Progenitor Cells. Cell stem cell. 9(1). 74–86. 166 indexed citations
18.
Daquinag, Alexes C., Yan Zhang, & Mikhail G. Kolonin. (2011). Vascular targeting of adipose tissue as an anti-obesity approach. Trends in Pharmacological Sciences. 32(5). 300–307. 61 indexed citations
19.
Zhang, Yan, Alexes C. Daquinag, Dmitry O. Traktuev, et al.. (2009). White Adipose Tissue Cells Are Recruited by Experimental Tumors and Promote Cancer Progression in Mouse Models. Cancer Research. 69(12). 5259–5266. 245 indexed citations
20.
Daquinag, Alexes C., et al.. (2005). The Pleckstrin Homology Domain Proteins Slm1 and Slm2 Are Required for Actin Cytoskeleton Organization in Yeast and Bind Phosphatidylinositol-4,5-Bisphosphate and TORC2. Molecular Biology of the Cell. 16(4). 1883–1900. 110 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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