Alba Ginés

653 total citations
8 papers, 528 citations indexed

About

Alba Ginés is a scholar working on Oncology, Molecular Biology and Hepatology. According to data from OpenAlex, Alba Ginés has authored 8 papers receiving a total of 528 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Oncology, 3 papers in Molecular Biology and 2 papers in Hepatology. Recurrent topics in Alba Ginés's work include Colorectal Cancer Treatments and Studies (6 papers), Cancer Treatment and Pharmacology (4 papers) and Hepatocellular Carcinoma Treatment and Prognosis (2 papers). Alba Ginés is often cited by papers focused on Colorectal Cancer Treatments and Studies (6 papers), Cancer Treatment and Pharmacology (4 papers) and Hepatocellular Carcinoma Treatment and Prognosis (2 papers). Alba Ginés collaborates with scholars based in Spain, United States and United Kingdom. Alba Ginés's co-authors include José Luís Zamora Manzano, Albert Abad, Anna Martínez‐Cardús, Eva Martinez‐Balibrea, Laura Layos, Sara Bystrup, Vicenç Ruiz de Porras, Manel Esteller, Cátia Moutinho and Cristina Bugés and has published in prestigious journals such as PLoS ONE, Cancer Research and International Journal of Cancer.

In The Last Decade

Alba Ginés

8 papers receiving 527 citations

Peers

Alba Ginés

ONCOL

MB

CR

PFM

PRM

Sara Bystrup Spain

Cristina Bugés Spain

Elisabetta Corna Italy

Vicenç Ruiz de Porras Spain

Yegang Ma China

Ziyan Y. Pessetto United States

Mengru Cao China

Jun Ji China

Oukseub Lee United States

Sara Bystrup Spain

Alba Ginés

194 ×0.7

ONCOL

246 ×0.9

MB

126 ×0.8

CR

48 ×0.7

PFM

34 ×0.6

PRM

Citations per year, relative to Alba Ginés Alba Ginés (= 1×) peers Sara Bystrup

Countries citing papers authored by Alba Ginés

Since Specialization

Citations

This map shows the geographic impact of Alba Ginés's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alba Ginés with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alba Ginés more than expected).

Fields of papers citing papers by Alba Ginés

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alba Ginés. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alba Ginés. The network helps show where Alba Ginés may publish in the future.

Co-authorship network of co-authors of Alba Ginés

This figure shows the co-authorship network connecting the top 25 collaborators of Alba Ginés. A scholar is included among the top collaborators of Alba Ginés based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alba Ginés. Alba Ginés is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Martinez‐Balibrea, Eva, Anna Martínez‐Cardús, Alba Ginés, et al.. (2015). Tumor-Related Molecular Mechanisms of Oxaliplatin Resistance. Molecular Cancer Therapeutics. 14(8). 1767–1776. 261 indexed citations

2.

Ginés, Alba, Sara Bystrup, Vicenç Ruiz de Porras, et al.. (2015). PKM2 Subcellular Localization Is Involved in Oxaliplatin Resistance Acquisition in HT29 Human Colorectal Cancer Cell Lines. PLoS ONE. 10(5). e0123830–e0123830. 32 indexed citations

3.

Porras, Vicenç Ruiz de, Sara Bystrup, Anna Martínez‐Cardús, et al.. (2015). Abstract 5478: Curcumin mediates reversion to oxaliplatin-acquired resistance in colorectal cancer cell lines through modulation of nuclear factor κB (NFκB) and cyclin-dependent kinase 5 (CDK5). Cancer Research. 75(15_Supplement). 5478–5478. 3 indexed citations

4.

Martinez‐Balibrea, Eva, Albert Abad, Anna Martínez‐Cardús, et al.. (2010). Abstract 2610: TYMS and XRCC1 gene polymorphisms as possible predictors for the addition of oxaliplatin to 5FU in the adjuvant treatment of stage II and III colorectal cancer. Cancer Research. 70(8_Supplement). 2610–2610. 1 indexed citations

5.

Martinez‐Balibrea, Eva, Anna Martínez‐Cardús, Eva Musulén, et al.. (2009). Increased levels of copper efflux transporter ATP7B are associated with poor outcome in colorectal cancer patients receiving oxaliplatin‐based chemotherapy. International Journal of Cancer. 124(12). 2905–2910. 60 indexed citations

6.

Martinez‐Balibrea, Eva, Carmen Plasencia, Alba Ginés, et al.. (2009). A proteomic approach links decreased pyruvate kinase M2 expression to oxaliplatin resistance in patients with colorectal cancer and in human cell lines. Molecular Cancer Therapeutics. 8(4). 771–778. 56 indexed citations

7.

Martínez‐Cardús, Anna, Eva Martinez‐Balibrea, Eva Bandrés, et al.. (2009). Pharmacogenomic approach for the identification of novel determinants of acquired resistance to oxaliplatin in colorectal cancer. Molecular Cancer Therapeutics. 8(1). 194–202. 57 indexed citations

8.

Martinez‐Balibrea, Eva, Albert Abad, Enrique Aranda, et al.. (2008). Pharmacogenetic approach for capecitabine or 5-fluorouracil selection to be combined with oxaliplatin as first-line chemotherapy in advanced colorectal cancer. European Journal of Cancer. 44(9). 1229–1237. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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