Akio Sekigawa

906 total citations
18 papers, 747 citations indexed

About

Akio Sekigawa is a scholar working on Neurology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Akio Sekigawa has authored 18 papers receiving a total of 747 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Neurology, 6 papers in Cellular and Molecular Neuroscience and 6 papers in Physiology. Recurrent topics in Akio Sekigawa's work include Parkinson's Disease Mechanisms and Treatments (11 papers), Nuclear Receptors and Signaling (5 papers) and Alzheimer's disease research and treatments (4 papers). Akio Sekigawa is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (11 papers), Nuclear Receptors and Signaling (5 papers) and Alzheimer's disease research and treatments (4 papers). Akio Sekigawa collaborates with scholars based in Japan, United States and Hungary. Akio Sekigawa's co-authors include Makoto Hashimoto, Masayo Fujita, Shuei Sugama, Takato Takenouchi, Kazunari Sekiyama, Masaaki Waragai, Yoshiki Takamatsu, Yoshifumi Iwamaru, Hiroshi Kitani and Jianshe Wei and has published in prestigious journals such as Nature Communications, The Journal of Immunology and International Journal of Molecular Sciences.

In The Last Decade

Akio Sekigawa

17 papers receiving 738 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Akio Sekigawa Japan 13 264 220 198 161 155 18 747
Raquel B. Dias Portugal 13 362 1.4× 280 1.3× 194 1.0× 173 1.1× 158 1.0× 16 832
Ljubisav Rakić Serbia 16 178 0.7× 206 0.9× 134 0.7× 109 0.7× 101 0.7× 46 657
Monica Armida Italy 16 311 1.2× 173 0.8× 265 1.3× 69 0.4× 135 0.9× 21 687
Carolina Cebrián Spain 9 325 1.2× 199 0.9× 277 1.4× 130 0.8× 210 1.4× 13 787
Patricia García‐Sanz Spain 12 245 0.9× 254 1.2× 302 1.5× 195 1.2× 97 0.6× 16 725
Rosa Luisa Potenza Italy 21 699 2.6× 455 2.1× 357 1.8× 123 0.8× 199 1.3× 38 1.2k
Marian Marvin United States 8 269 1.0× 228 1.0× 226 1.1× 110 0.7× 165 1.1× 8 695
Lilah Toker Israel 16 200 0.8× 307 1.4× 156 0.8× 112 0.7× 97 0.6× 27 757
Paula A. Pousinha France 12 272 1.0× 241 1.1× 178 0.9× 167 1.0× 233 1.5× 20 808
Yoori Choi South Korea 14 209 0.8× 320 1.5× 90 0.5× 227 1.4× 186 1.2× 31 872

Countries citing papers authored by Akio Sekigawa

Since Specialization
Citations

This map shows the geographic impact of Akio Sekigawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Akio Sekigawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Akio Sekigawa more than expected).

Fields of papers citing papers by Akio Sekigawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Akio Sekigawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Akio Sekigawa. The network helps show where Akio Sekigawa may publish in the future.

Co-authorship network of co-authors of Akio Sekigawa

This figure shows the co-authorship network connecting the top 25 collaborators of Akio Sekigawa. A scholar is included among the top collaborators of Akio Sekigawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Akio Sekigawa. Akio Sekigawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Sekigawa, Akio, Yoshiki Takamatsu, Kazunari Sekiyama, & Makoto Hashimoto. (2015). Role of α- and β-Synucleins in the Axonal Pathology of Parkinson’s Disease and Related Synucleinopathies. Biomolecules. 5(2). 1000–1011. 11 indexed citations
2.
Sekiyama, Kazunari, Masaaki Waragai, Hiroyasu Akatsu, et al.. (2014). Disease modifying effect of adiponectin in model of α ‐synucleinopathies. Annals of Clinical and Translational Neurology. 1(7). 479–489. 27 indexed citations
3.
Sekigawa, Akio, Yoshiki Takamatsu, Kazunari Sekiyama, et al.. (2013). Diversity of Mitochondrial Pathology in a Mouse Model of Axonal Degeneration in Synucleinopathies. Oxidative Medicine and Cellular Longevity. 2013. 1–6. 5 indexed citations
4.
Sekigawa, Akio, Masayo Fujita, Kazunari Sekiyama, et al.. (2012). Distinct mechanisms of axonal globule formation in mice expressing human wild type α-synuclein or dementia with Lewy bodies-linked P123H ß-synuclein. Molecular Brain. 5(1). 34–34. 13 indexed citations
5.
Sekiyama, Kazunari, Masayo Fujita, Akio Sekigawa, et al.. (2012). Ibuprofen ameliorates protein aggregation and astrocytic gliosis, but not cognitive dysfunction, in a transgenic mouse expressing dementia with Lewy bodies-linked P123H β-synuclein. Neuroscience Letters. 515(1). 97–101. 17 indexed citations
6.
Fujita, Masayo, Akio Sekigawa, Kazunari Sekiyama, Yoshiki Takamatsu, & Makoto Hashimoto. (2012). Possible Alterations in β-Synuclein, the Non-Amyloidogenic Homologue of α-Synuclein, during Progression of Sporadic α-Synucleinopathies. International Journal of Molecular Sciences. 13(9). 11584–11592. 8 indexed citations
7.
Sekiyama, Kazunari, Shuei Sugama, Masayo Fujita, et al.. (2012). Neuroinflammation in Parkinson's Disease and Related Disorders: A Lesson from Genetically Manipulated Mouse Models ofα-Synucleinopathies. Parkinson s Disease. 2012. 1–8. 44 indexed citations
8.
Kubota, Yoshiyuki, Naoki Shigematsu, Fuyuki Karube, et al.. (2011). Selective Coexpression of Multiple Chemical Markers Defines Discrete Populations of Neocortical GABAergic Neurons. Cerebral Cortex. 21(8). 1803–1817. 183 indexed citations
9.
Takenouchi, Takato, Yoshifumi Iwamaru, Shuei Sugama, et al.. (2011). The activation of P2X7 receptor induces cathepsin D-dependent production of a 20-kDa form of IL-1β under acidic extracellular pH in LPS-primed microglial cells. Journal of Neurochemistry. 117(4). no–no. 30 indexed citations
10.
Takenouchi, Takato, Kazunari Sekiyama, Akio Sekigawa, et al.. (2010). P2X7 Receptor Signaling Pathway as a Therapeutic Target for Neurodegenerative Diseases. Archivum Immunologiae et Therapiae Experimentalis. 58(2). 91–96. 102 indexed citations
11.
Fujita, Masayo, Shuei Sugama, Kazunari Sekiyama, et al.. (2010). A β-synuclein mutation linked to dementia produces neurodegeneration when expressed in mouse brain. Nature Communications. 1(1). 110–110. 66 indexed citations
12.
Waragai, Masaaki, Kazunari Sekiyama, Akio Sekigawa, et al.. (2010). α-Synuclein and DJ-1 as Potential Biological Fluid Biomarkers for Parkinson’s Disease. International Journal of Molecular Sciences. 11(11). 4257–4266. 26 indexed citations
13.
Takenouchi, Takato, Masaaki Nakai, Yoshifumi Iwamaru, et al.. (2009). The Activation of P2X7 Receptor Impairs Lysosomal Functions and Stimulates the Release of Autophagolysosomes in Microglial Cells. The Journal of Immunology. 182(4). 2051–2062. 115 indexed citations
14.
Fujita, Masayo, Akio Sekigawa, Kazunari Sekiyama, Shuei Sugama, & Makoto Hashimoto. (2009). Neurotoxic conversion of β-synuclein: a novel approach to generate a transgenic mouse model of synucleinopathies?. Journal of Neurology. 256(S3). 286–292. 8 indexed citations
15.
Wei, Jianshe, Masayo Fujita, Akio Sekigawa, et al.. (2009). Gangliosides protection against lysosomal pathology of synucleinopathies. Autophagy. 5(6). 860–861. 13 indexed citations
16.
Wei, Jianshe, Masayo Fujita, Masaaki Nakai, et al.. (2009). Protective Role of Endogenous Gangliosides for Lysosomal Pathology in a Cellular Model of Synucleinopathies. American Journal Of Pathology. 174(5). 1891–1909. 52 indexed citations
17.
Sekigawa, Akio, et al.. (2009). SYNDROMES OF INSULIN RESISTANCE. Nutrition Reviews. 36(4). 103–106.
18.
Yamada, Mari, Taro Saito, Yutaka Sato, et al.. (2007). Cdk5–p39 is a labile complex with the similar substrate specificity to Cdk5–p35. Journal of Neurochemistry. 102(5). 1477–1487. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Breakdown of academic impact, for papers by Raquel B. Dias Breakdown of academic impact, for papers by Ljubisav Rakić Breakdown of academic impact, for papers by Monica Armida Breakdown of academic impact, for papers by Carolina Cebrián Breakdown of academic impact, for papers by Patricia García‐Sanz Breakdown of academic impact, for papers by Rosa Luisa Potenza Breakdown of academic impact, for papers by Marian Marvin Breakdown of academic impact, for papers by Lilah Toker Breakdown of academic impact, for papers by Paula A. Pousinha Breakdown of academic impact, for papers by Yoori Choi
Rankless by CCL
2026