Abraham Benderly
- Molecular Biology
- Endocrinology, Diabetes and Metabolism top 10%
- Pediatrics, Perinatology and Child Health
- Organic Chemistry
- Genetics
- Co-authors
- Ze’ev HochbergEsther LeibermanShimon MosesTirza CohenJoseph SackNaim ShehadehA VonsoverMary Rudolf
- Topics
- Sexual Differentiation and Disorders (3 papers)Synthesis of heterocyclic compounds (2 papers)Metabolism and Genetic Disorders (2 papers)
- Partner nations
- IsraelCanadaUnited States
In The Last Decade
Abraham Benderly
19 papers receiving 316 citations
Peers
Comparison fields: 5 of 66
- Molecular Biology 107
- Endocrinology, Diabetes and Metabolism 104
- Pediatrics, Perinatology and Child Health 54
- Organic Chemistry 51
- Genetics 49
Countries citing papers authored by Abraham Benderly
This map shows the geographic impact of Abraham Benderly's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Abraham Benderly with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Abraham Benderly more than expected).
Fields of papers citing papers by Abraham Benderly
This network shows the impact of papers produced by Abraham Benderly. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Abraham Benderly. The network helps show where Abraham Benderly may publish in the future.
Co-authorship network of co-authors of Abraham Benderly
This figure shows the co-authorship network connecting the top 25 collaborators of Abraham Benderly. A scholar is included among the top collaborators of Abraham Benderly based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Abraham Benderly. Abraham Benderly is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 44 | |
| 3 | Effect of urine and urine components on the chemiluminescent response of bacteria-stimulated polymorphonuclear leukocytes. | 2 |
| 4 | 27 | |
| 5 | 50 | |
| 6 | 20 | |
| 7 | 3 | |
| 8 | 1 | |
| 9 | 29 | |
| 10 | 6 | |
| 11 | 6 | |
| 12 | 16 | |
| 13 | 67 | |
| 14 | 6 | |
| 15 | 9 | |
| 16 | 3 | |
| 17 | 4 | |
| 18 | 4 | |
| 19 | 39 |
About Abraham Benderly
Abraham Benderly is a scholar working on Clinical Biochemistry, Pharmacy and Endocrinology, Diabetes and Metabolism, having authored 19 papers that have together received 342 indexed citations. Recurring topics across this work include Sexual Differentiation and Disorders (3 papers), Synthesis of heterocyclic compounds (2 papers) and Metabolism and Genetic Disorders (2 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (104 citations), Genetics (49 citations) and Hematology (32 citations). Abraham Benderly has collaborated with scholars based in Israel, Canada and United States. Frequent co-authors include Ze’ev Hochberg, Esther Leiberman, Shimon Moses, Tirza Cohen, Joseph Sack, Naim Shehadeh, A Vonsover, Mary Rudolf, E Ben‐Porath and Ronald T. Coutts. Their work appears in journals such as PEDIATRICS, The Journal of Organic Chemistry and The Journal of Pediatrics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.