Abi Li

2.4k total citations · 1 hit paper
12 papers, 1.4k citations indexed

About

Abi Li is a scholar working on Neurology, Molecular Biology and Genetics. According to data from OpenAlex, Abi Li has authored 12 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Neurology, 6 papers in Molecular Biology and 4 papers in Genetics. Recurrent topics in Abi Li's work include Parkinson's Disease Mechanisms and Treatments (5 papers), Neurological diseases and metabolism (3 papers) and Genetics and Neurodevelopmental Disorders (3 papers). Abi Li is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (5 papers), Neurological diseases and metabolism (3 papers) and Genetics and Neurodevelopmental Disorders (3 papers). Abi Li collaborates with scholars based in United Kingdom, United States and Germany. Abi Li's co-authors include Henry Houlden, John Hardy, Nicholas Wood, Janice L. Holton, Andrew Singleton, Andrew J. Lees, Tamás Révész, Kailash P. Bhatia, Coro Paisán‐Ruíz and Susanne A. Schneider and has published in prestigious journals such as Brain, Annals of Neurology and Neurobiology of Aging.

In The Last Decade

Abi Li

12 papers receiving 1.4k citations

Hit Papers

Glucocerebrosidase mutations in clinical and pathological... 2009 2026 2014 2020 2009 100 200 300 400 500

Peers

Abi Li
Reema Paudel United Kingdom
Manu Sharma Germany
Julie P. Taylor United States
Stephanie A. Cobb United States
Dawn H. W. Lau United Kingdom
P. Shashidharan United States
Reema Paudel United Kingdom
Abi Li
Citations per year, relative to Abi Li Abi Li (= 1×) peers Reema Paudel

Countries citing papers authored by Abi Li

Since Specialization
Citations

This map shows the geographic impact of Abi Li's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Abi Li with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Abi Li more than expected).

Fields of papers citing papers by Abi Li

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Abi Li. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Abi Li. The network helps show where Abi Li may publish in the future.

Co-authorship network of co-authors of Abi Li

This figure shows the co-authorship network connecting the top 25 collaborators of Abi Li. A scholar is included among the top collaborators of Abi Li based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Abi Li. Abi Li is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Wiethoff, Sarah, Charles Arber, Abi Li, et al.. (2015). Using human induced pluripotent stem cells to model cerebellar disease: Hope and hype. Journal of Neurogenetics. 29(2-3). 95–102. 8 indexed citations
2.
Paudel, Reema, Abi Li, John Hardy, et al.. (2015). DYT6 Dystonia: A Neuropathological Study. Neurodegenerative Diseases. 16(3-4). 273–278. 16 indexed citations
3.
Ayton, Scott, Kelly Bertram, Helen Ling, et al.. (2015). Serotonergic markers in Parkinson's disease and levodopa‐induced dyskinesias. Movement Disorders. 30(6). 796–804. 34 indexed citations
4.
Paudel, Reema, Aoife P. Kiely, Abi Li, et al.. (2014). Neuropathological features of genetically confirmed DYT1 dystonia: investigating disease-specific inclusions. Acta Neuropathologica Communications. 2(1). 159–159. 18 indexed citations
5.
Deas, Emma, Asif Machhada, Abi Li, et al.. (2014). PINK1 deficiency in β-cells increases basal insulin secretion and improves glucose tolerance in mice. Open Biology. 4(5). 140051–140051. 35 indexed citations
6.
Lashley, Tammaryn, et al.. (2014). Alterations in global DNA methylation and hydroxymethylation are not detected in Alzheimer's disease. Neuropathology and Applied Neurobiology. 41(4). 497–506. 68 indexed citations
7.
Dunn, Laura B., G Allen, Adamantios Mamais, et al.. (2013). Dysregulation of glucose metabolism is an early event in sporadic Parkinson's disease. Neurobiology of Aging. 35(5). 1111–1115. 185 indexed citations
8.
Paisán‐Ruíz, Coro, Abi Li, Susanne A. Schneider, et al.. (2010). Widespread Lewy body and tau accumulation in childhood and adult onset dystonia-parkinsonism cases with PLA2G6 mutations. Neurobiology of Aging. 33(4). 814–823. 161 indexed citations
9.
Novak, Marianne, Mary G. Sweeney, Abi Li, et al.. (2010). An ITPR1 gene deletion causes spinocerebellar ataxia 15/16: A genetic, clinical and radiological description. Movement Disorders. 25(13). 2176–2182. 43 indexed citations
10.
Brooks, Janet, Henry Houlden, Anna Melchers, et al.. (2010). Mutational analysis of parkin and PINK1 in multiple system atrophy. Neurobiology of Aging. 32(3). 548.e5–548.e7. 19 indexed citations
11.
Brás, José, Emma Deas, Sean S. O’Sullivan, et al.. (2009). Glucocerebrosidase mutations in clinical and pathologically proven Parkinson's disease. Brain. 132(7). 1783–1794. 509 indexed citations breakdown →
12.
Paisán‐Ruíz, Coro, Kailash P. Bhatia, Abi Li, et al.. (2008). Characterization of PLA2G6 as a locus for dystonia‐parkinsonism. Annals of Neurology. 65(1). 19–23. 328 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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