Abdullah A. Osman

3.7k total citations · 1 hit paper
36 papers, 2.1k citations indexed

About

Abdullah A. Osman is a scholar working on Oncology, Molecular Biology and Immunology. According to data from OpenAlex, Abdullah A. Osman has authored 36 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Oncology, 16 papers in Molecular Biology and 7 papers in Immunology. Recurrent topics in Abdullah A. Osman's work include Cancer-related Molecular Pathways (14 papers), interferon and immune responses (6 papers) and Cancer Immunotherapy and Biomarkers (4 papers). Abdullah A. Osman is often cited by papers focused on Cancer-related Molecular Pathways (14 papers), interferon and immune responses (6 papers) and Cancer Immunotherapy and Biomarkers (4 papers). Abdullah A. Osman collaborates with scholars based in United States, China and Japan. Abdullah A. Osman's co-authors include Lawrence J. Mandarino, Thongchai Pratipanawatr, Kenneth Cusi, Merri Pendergrass, Katsumi Maezono, Ralph A. DeFronzo, C. Ronald Kahn, Mary‐Elizabeth Patti, Jeffrey N. Myers and Ameeta A. Patel and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Journal of Clinical Oncology.

In The Last Decade

Abdullah A. Osman

35 papers receiving 2.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Insulin resistance differentially affects the PI 3-kinase– and MAP kinase–mediated signaling in human muscle

2000 856 citations Kenneth Cusi, Katsumi Maezono et al. Journal of Clinical Investigation profile →

Peers

Countries citing papers authored by Abdullah A. Osman

Since Specialization

Citations

This map shows the geographic impact of Abdullah A. Osman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Abdullah A. Osman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Abdullah A. Osman more than expected).

Fields of papers citing papers by Abdullah A. Osman

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Abdullah A. Osman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Abdullah A. Osman. The network helps show where Abdullah A. Osman may publish in the future.

Co-authorship network of co-authors of Abdullah A. Osman

This figure shows the co-authorship network connecting the top 25 collaborators of Abdullah A. Osman. A scholar is included among the top collaborators of Abdullah A. Osman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Abdullah A. Osman. Abdullah A. Osman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Title	Journal	Authors	Indexed citations
1	FAK Drives Resistance to Therapy in HPV-Negative Head and Neck Cancer in a p53-Dependent Manner	Clinical Cancer Research	Phillip M. Pifer, Liangpeng Yang et al.	13
2	Dysregulation and Epigenetic Reprogramming of NRF2 Signaling Axis Promote Acquisition of Cisplatin Resistance and Metastasis in Head and Neck Squamous Cell Carcinoma	Clinical Cancer Research	Abdullah A. Osman, Emre Arslan et al.	32
3	TP53 gain-of-function mutation modulates the immunosuppressive microenvironment in non-HPV-associated oral squamous cell carcinoma	Journal for ImmunoTherapy of Cancer	Yewen Shi, Xiaoyong Ren et al.	16
4	1494 Role of gain of function p53 mutations in macrophage polarization and myeloid-derived suppressor cells in head and neck squamous cell carcinoma	SHILAP Revista de lepidopterología	Roberto Rangel, Abdullah A. Osman et al.	1
5	Mutant p53 drives an immune cold tumor immune microenvironment in oral squamous cell carcinoma	Communications Biology	Yewen Shi, Tongxin Xie et al.	24
6	Evolutionary Action Score of TP53 Analysis in Pathologically High-Risk Human Papillomavirus-Negative Head and Neck Cancer From a Phase 2 Clinical Trial: NRG Oncology Radiation Therapy Oncology Group 0234	Advances in Radiation Oncology	Chieko Michikawa, Pedro A. Torres‐Saavedra et al.	3
7	Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress	Molecular Cancer Therapeutics	Antje Lindemann, Ameeta A. Patel et al.	18
8	Biology of the Radio- and Chemo-Responsiveness in HPV Malignancies	Seminars in Radiation Oncology	Michael T. Spiotto, Cullen M. Taniguchi et al.	21
9	Head and neck cancer organoids established by modification of the CTOS method can be used to predict in vivo drug sensitivity	Oral Oncology	Noriaki Tanaka, Abdullah A. Osman et al.	106
10	Gain-of-function mutant p53 promotes the oncogenic potential of head and neck squamous cell carcinoma cells by targeting the transcription factors FOXO3a and FOXM1	Oncogene	Noriaki Tanaka, Mei Zhao et al.	41
11	Wee-1 Kinase Inhibition Sensitizes High-Risk HPV+ HNSCC to Apoptosis Accompanied by Downregulation of MCl-1 and XIAP Antiapoptotic Proteins	Clinical Cancer Research	Noriaki Tanaka, Ameeta A. Patel et al.	46
12	Wee-1 Kinase Inhibition Overcomes Cisplatin Resistance Associated with High-Risk TP53 Mutations in Head and Neck Cancer through Mitotic Arrest Followed by Senescence	Molecular Cancer Therapeutics	Abdullah A. Osman, Marcus M. Monroe et al.	88
13	Indonesian tourism sector: a potential sector that has not been optimized.	AUSTRALIAN JOURNAL OF BASIC AND APPLIED SCIENCES	Zulkarnain Lubis, Abdullah A. Osman	4
14	The p53-Reactivating Small Molecule RITA Induces Senescence in Head and Neck Cancer Cells	PLoS ONE	Liang Yang, Alison L. Fitzgerald et al.	24
15	Chk1/2 Inhibition Overcomes the Cisplatin Resistance of Head and Neck Cancer Cells Secondary to the Loss of Functional p53	Molecular Cancer Therapeutics	Marcus V. Ortega Alves, Curtis R. Pickering et al.	95
16	Muscle-like nicotinic receptor accessory molecules in sensory hair cells of the inner ear	Molecular and Cellular Neuroscience	Abdullah A. Osman, Omar Akil et al.	13
17	The Final Stage of Cholinergic Differentiation Occurs Below Inner Hair Cells During Development of the Rodent Cochlea	Journal of the Association for Research in Otolaryngology	Abdullah A. Osman, Dwayne D. Simmons et al.	23
18	Activity-dependent transcription regulation of PSD-95 by neuregulin-1 and Eos	Nature Neuroscience	Jianxin Bao, Hana Lin et al.	135
19	Wolframin Expression Induces Novel Ion Channel Activity in Endoplasmic Reticulum Membranes and Increases Intracellular Calcium	Journal of Biological Chemistry	Abdullah A. Osman, Mitsuyoshi Saito et al.	158
20	Insulin resistance differentially affects the PI 3-kinase– and MAP kinase–mediated signaling in human muscle breakdown →	Journal of Clinical Investigation	Kenneth Cusi, Katsumi Maezono et al.	856

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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