A. Weindl

5.1k citations
77 papers · 3.2k indexed · h-index 30

A. Weindl

77 papers receiving 3.0k citations

Peers

A. Weindl
Comparison fields: 5 of 119
  • Endocrine and Autonomic Systems 721
  • Cellular and Molecular Neuroscience 1.5k
  • Behavioral Neuroscience 241
  • Neurology 418
  • Neurology 647
Replace WH Oertel with:
WH Oertel Germany
Kunio Kitahama France
Gunnar Skagerberg Sweden
Marcel Tappaz France
Leif Wiklund Sweden
Hitoo Nishino Japan
Olivier Bosler France
David V. Pow Australia
Tetsuo Sugimoto Japan
Richard W. Clough United States
A. Weindl relative to WH Oertel Germany WH Oertel's profile →
Citations per field
00.5×2.9×
WH Oertel · 1×
Citations per year

Countries citing papers authored by A. Weindl

Since Specialization
Citations

This map shows the geographic impact of A. Weindl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. Weindl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. Weindl more than expected).

Fields of papers citing papers by A. Weindl

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. Weindl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. Weindl. The network helps show where A. Weindl may publish in the future.

Co-authorship network

The 25 scholars most cited alongside A. Weindl, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with A. Weindl Line = papers co-authored together A. Weindl links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 201035
2 20097
3 200851
4 200637
5 2005143
6 200456
7 200228
8 19998
9 199613
10 199514
11 199567
12 19941
13 199329
14 199219
15 199023
16 198918
17 1987109
18
Brain-endocrine interaction
1972263
19
[Spherical lipid bodies inthe epithelium of the choid plexus in the rabbit].
196910
20 196558

About A. Weindl

A. Weindl is a scholar working on Cellular and Molecular Neuroscience, Endocrine and Autonomic Systems and Neurology, having authored 77 papers that have together received 3.2k indexed citations. Recurring topics across this work include Neurological disorders and treatments (16 papers), Genetic Neurodegenerative Diseases (12 papers), Neuropeptides and Animal Physiology (10 papers), Neuroendocrine regulation and behavior (10 papers), Neuroscience and Neuropharmacology Research (9 papers), Receptor Mechanisms and Signaling (9 papers), Mitochondrial Function and Pathology (6 papers) and Neuroscience of respiration and sleep (6 papers). The work is most often cited by research in Endocrine and Autonomic Systems (721 citations), Cellular and Molecular Neuroscience (1.5k citations) and Behavioral Neuroscience (241 citations). A. Weindl has collaborated with scholars based in Germany, United States and Switzerland. Frequent co-authors include Michael V. Sofroniew, Karl M. Knigge, R. Wetzstein, David E. Scott, Thomas Arzberger, I. Schinko, K Krampfl, Alexander Peinemann, A. Strüppler and Jan Kassubek. Their work appears in journals such as Cell and Tissue Research, Movement Disorders, Brain Research, Neurology and Journal of Cardiovascular Pharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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