A. L. Schwarzman

1.4k total citations
38 papers, 1.1k citations indexed

About

A. L. Schwarzman is a scholar working on Molecular Biology, Physiology and Surgery. According to data from OpenAlex, A. L. Schwarzman has authored 38 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 20 papers in Physiology and 8 papers in Surgery. Recurrent topics in A. L. Schwarzman's work include Alzheimer's disease research and treatments (18 papers), Cholesterol and Lipid Metabolism (7 papers) and Cancer, Lipids, and Metabolism (6 papers). A. L. Schwarzman is often cited by papers focused on Alzheimer's disease research and treatments (18 papers), Cholesterol and Lipid Metabolism (7 papers) and Cancer, Lipids, and Metabolism (6 papers). A. L. Schwarzman collaborates with scholars based in Russia, United States and United Kingdom. A. L. Schwarzman's co-authors include Michael P. Vitek, Luisa Gregori, Dmitry Goldgaber, Warren J. Strittmatter, Patricia K. Coyle, Judith M. Silverman, Karl H. Weisgraber, R Bhasin, Paul H. Wen and Zhiping Shao and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and The EMBO Journal.

In The Last Decade

A. L. Schwarzman

37 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
A. L. Schwarzman Russia 14 656 637 211 196 151 38 1.1k
Silva Hečimović Croatia 20 818 1.2× 852 1.3× 199 0.9× 226 1.2× 145 1.0× 44 1.6k
Tadashi Nakaya Japan 19 697 1.1× 655 1.0× 295 1.4× 230 1.2× 171 1.1× 38 1.2k
Kanae Ando Japan 19 681 1.0× 856 1.3× 309 1.5× 273 1.4× 185 1.2× 49 1.3k
Georges Lévesque Canada 18 787 1.2× 778 1.2× 206 1.0× 233 1.2× 204 1.4× 43 1.3k
Jean‐Marie Sontag Australia 13 697 1.1× 399 0.6× 309 1.5× 198 1.0× 87 0.6× 21 1.2k
Hiroshi Hasegawa Japan 14 668 1.0× 674 1.1× 243 1.2× 253 1.3× 167 1.1× 22 1.2k
Philip J. Dolan United States 10 666 1.0× 574 0.9× 138 0.7× 215 1.1× 208 1.4× 14 1.1k
Matthew L. Hemming United States 18 691 1.1× 518 0.8× 112 0.5× 176 0.9× 207 1.4× 25 1.6k
Nyosha Alikhani Sweden 13 889 1.4× 681 1.1× 106 0.5× 122 0.6× 173 1.1× 16 1.3k
Bertrand P. Tseng United States 12 603 0.9× 826 1.3× 145 0.7× 225 1.1× 199 1.3× 15 1.3k

Countries citing papers authored by A. L. Schwarzman

Since Specialization
Citations

This map shows the geographic impact of A. L. Schwarzman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. L. Schwarzman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. L. Schwarzman more than expected).

Fields of papers citing papers by A. L. Schwarzman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. L. Schwarzman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. L. Schwarzman. The network helps show where A. L. Schwarzman may publish in the future.

Co-authorship network of co-authors of A. L. Schwarzman

This figure shows the co-authorship network connecting the top 25 collaborators of A. L. Schwarzman. A scholar is included among the top collaborators of A. L. Schwarzman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A. L. Schwarzman. A. L. Schwarzman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schwarzman, A. L., Konstantin Senkevich, Anton Emelyanov, & С. Н. Пчелина. (2019). Прионные свойства альфа-синуклеина. Молекулярная биология. 53(3). 380–387. 4 indexed citations
2.
Schwarzman, A. L. & С. В. Саранцева. (2017). Transmission of pathogenic protein aggregates in Alzheimer’s disease. Molecular Biology. 51(3). 368–371. 11 indexed citations
3.
Schwarzman, A. L., et al.. (2016). Role of the ABC transporters A1 and G1, key reverse cholesterol transport proteins, in atherosclerosis. Molecular Biology. 50(2). 193–199. 25 indexed citations
5.
Schwarzman, A. L., et al.. (2015). Expression of human amyloid precursor protein in Drosophila melanogaster nerve cells causes a decrease in presynaptic gene mRNA levels. Genetics and Molecular Research. 14(3). 9225–9232. 3 indexed citations
6.
Schwarzman, A. L., et al.. (2014). Reducing of LXRβ and PPARγ mRNA in M-CSF stimulated macrophages in patients with levels atherosclerosis. Ecological genetics. 12(1). 14–18. 1 indexed citations
8.
Пчелина, С. Н., et al.. (2011). Reduced Content of α-Synuclein in Peripheral Blood Leukocytes of Patients with LRRK2-Associated Parkinson’s Disease. Bulletin of Experimental Biology and Medicine. 150(6). 679–681. 8 indexed citations
9.
Саранцева, С. В., et al.. (2011). Dendrimer D5 is a Vector for Peptide Transport to Brain Cells. Bulletin of Experimental Biology and Medicine. 150(4). 429–431. 7 indexed citations
10.
Саранцева, С. В. & A. L. Schwarzman. (2009). Modern genetic approaches to searching for targets for medicinal preparations. Russian Journal of Genetics. 45(7). 761–770. 3 indexed citations
11.
Пчелина, С. Н., et al.. (2008). Screening for LRRK2 mutations in patients with Parkinson’s disease in Russia: identification of a novel LRRK2 variant. European Journal of Neurology. 15(7). 692–696. 21 indexed citations
12.
Schwarzman, A. L. & Dmitry Goldgaber. (2007). Interaction of Transthyretin with Amyloid β‐Protein: Binding and Inhibition of Amyloid Formation. Novartis Foundation symposium. 199. 146–169. 42 indexed citations
13.
Pchelina, Sofya, et al.. (2006). G2019S LRRK2 mutation in familial and sporadic Parkinson's disease in Russia. Movement Disorders. 21(12). 2234–2236. 18 indexed citations
14.
Schwarzman, A. L., Maria Tsiper, Luisa Gregori, et al.. (2005). Selection of peptides binding to the amyloid b-protein reveals potential inhibitors of amyloid formation. Amyloid. 12(4). 199–209. 18 indexed citations
15.
Baki, Lia, Junichi Shioi, Paul H. Wen, et al.. (2004). PS1 activates PI3K thus inhibiting GSK‐3 activity and tau overphosphorylation: effects of FAD mutations. The EMBO Journal. 23(13). 2586–2596. 247 indexed citations
16.
Schwarzman, A. L., et al.. (2004). Amyloidogenic and anti-amyloidogenic properties of recombinant transthyretin variants. Amyloid. 11(1). 1–10. 44 indexed citations
17.
Dranovsky, Alex, Inez Vincent, Luisa Gregori, et al.. (2001). Cdc2 phosphorylation of nucleolin demarcates mitotic stages and Alzheimer’s disease pathology. Neurobiology of Aging. 22(4). 517–528. 73 indexed citations
18.
Singh, Nandita, Maria Tsiper, Alex Dranovsky, et al.. (2001). The Role of Alzheimer's Disease-Related Presenilin 1 in Intercellular Adhesion. Experimental Cell Research. 263(1). 1–13. 24 indexed citations
19.
Schwarzman, A. L., Nandita Singh, Maria Tsiper, et al.. (1999). Endogenous presenilin 1 redistributes to the surface of lamellipodia upon adhesion of Jurkat cells to a collagen matrix. Proceedings of the National Academy of Sciences. 96(14). 7932–7937. 58 indexed citations
20.
Vostrov, Alexander A., et al.. (1995). USF binds to the APBα sequence in the promoter of the amyloid β-protein precursor gene. Nucleic Acids Research. 23(14). 2734–2741. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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