A. Jonas Ekstrand

2.9k total citations · 1 hit paper
16 papers, 2.3k citations indexed

About

A. Jonas Ekstrand is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Oncology. According to data from OpenAlex, A. Jonas Ekstrand has authored 16 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 7 papers in Cellular and Molecular Neuroscience and 4 papers in Oncology. Recurrent topics in A. Jonas Ekstrand's work include Neuropeptides and Animal Physiology (6 papers), HER2/EGFR in Cancer Research (3 papers) and Neuroscience and Neuropharmacology Research (2 papers). A. Jonas Ekstrand is often cited by papers focused on Neuropeptides and Animal Physiology (6 papers), HER2/EGFR in Cancer Research (3 papers) and Neuroscience and Neuropharmacology Research (2 papers). A. Jonas Ekstrand collaborates with scholars based in Sweden, United States and Belgium. A. Jonas Ekstrand's co-authors include C. David James, V. Peter Collins, Noriaki Sugawa, V. Peter Collins, Webster K. Cavenee, Barbara Seliger, Ralf F. Pettersson, Hessameh Hassani, Lu Liu and Hamid Madanchi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Medicine and Journal of neurosurgery.

In The Last Decade

A. Jonas Ekstrand

16 papers receiving 2.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Genes for epidermal growth factor receptor, transforming growth factor alpha, and epidermal growth factor and their expression in human gliomas in vivo.

1991 519 citations A. Jonas Ekstrand, C. David James et al. PubMed profile →

Peers

A. Jonas Ekstrand

ONCOL

GENET

CMN

Kensuke Tateishi Japan

Lucia Vallar Italy

Daniela Virgintino Italy

Rainer Glaß Germany

Pauli Helén Finland

Maya H. Nisancioglu Sweden

Michael Vanlandewijck Sweden

Björn Scheffler Germany

Maria Lia Palomba United States

Chieko Koike Japan

Kensuke Tateishi Japan

A. Jonas Ekstrand

527 ×0.5

190 ×0.2

ONCOL

540 ×0.9

GENET

368 ×0.9

CMN

320 ×0.9

Citations per year, relative to A. Jonas Ekstrand A. Jonas Ekstrand (= 1×) peers Kensuke Tateishi

Countries citing papers authored by A. Jonas Ekstrand

Since Specialization

Citations

This map shows the geographic impact of A. Jonas Ekstrand's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. Jonas Ekstrand with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. Jonas Ekstrand more than expected).

Fields of papers citing papers by A. Jonas Ekstrand

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. Jonas Ekstrand. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. Jonas Ekstrand. The network helps show where A. Jonas Ekstrand may publish in the future.

Co-authorship network of co-authors of A. Jonas Ekstrand

This figure shows the co-authorship network connecting the top 25 collaborators of A. Jonas Ekstrand. A scholar is included among the top collaborators of A. Jonas Ekstrand based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A. Jonas Ekstrand. A. Jonas Ekstrand is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Ekstrand, A. Jonas, et al.. (2005). Treatment with angiotensin converting enzyme inhibitors, angiotensin-II-antagonists and beta-blockers in an unselected group of patients with chronic heart failure. European Journal of Clinical Pharmacology. 61(3). 209–214. 4 indexed citations

Quéva, Christophe, A. Edlund, A. Jonas Ekstrand, et al.. (2003). Effects of GABA agonists on body temperature regulation in GABA_B(1)^−/− mice. British Journal of Pharmacology. 140(2). 315–322. 100 indexed citations

Ekstrand, A. Jonas, Renhai Cao, Meit A. Björndahl, et al.. (2003). Deletion of neuropeptide Y (NPY) 2 receptor in mice results in blockage of NPY-induced angiogenesis and delayed wound healing. Proceedings of the National Academy of Sciences. 100(10). 6033–6038. 154 indexed citations

Lehmann, Anders, et al.. (2003). Effects of Repeated Administration of Baclofen to Rats on GABAB Receptor Binding Sites and Subunit Expression in the Brain. Neurochemical Research. 28(2). 387–393. 31 indexed citations

Naveilhan, Philippe, Lennart Svensson, Susanne Nyström, A. Jonas Ekstrand, & Patrik Ernfors. (2002). Attenuation of hypercholesterolemia and hyperglycemia in ob/ob mice by NPY Y2 receptor ablation. Peptides. 23(6). 1087–1091. 34 indexed citations

Marklund, Ulrica, Mona Byström, Karin Gedda, et al.. (2002). Intron-mediated expression of the human neuropeptide Y Y1 receptor. Molecular and Cellular Endocrinology. 188(1-2). 85–97. 30 indexed citations

Ekstrand, A. Jonas, et al.. (2002). INABILITY OF 50HZ MAGNETIC FIELDS TO REGULATE PKC‐ AND CA²⁺‐DEPENDENT GENE EXPRESSION IN JURKAT CELLS. Cell Biology International. 26(2). 203–209. 5 indexed citations

Naveilhan, Philippe, Hessameh Hassani, Josep M. Canals, et al.. (1999). Normal feeding behavior, body weight and leptin response require the neuropeptide Y Y2 receptor. Nature Medicine. 5(10). 1188–1193. 222 indexed citations

Malmström, Rickard E., Tomas Hökfelt, Jan‐Arne Björkman, et al.. (1998). Characterization and molecular cloning of vascular neuropeptide Y receptor subtypes in pig and dog. Regulatory Peptides. 75-76. 55–70. 30 indexed citations

10.

He, Ju, et al.. (1996). Transfection of IFNα in human glioblastoma cells and tumorigenicity in association with induction of PKR and OAS gene expression. Journal of neurosurgery. 85(6). 1085–1090. 3 indexed citations

11.

Ekstrand, A. Jonas, Lu Liu, Ju He, et al.. (1995). Altered subcellular location of an activated and tumour-associated epidermal growth factor receptor.. PubMed. 10(7). 1455–60. 45 indexed citations

12.

Ekstrand, A. Jonas, Nicola Longo, Hamid Madanchi, et al.. (1994). Functional characterization of an EGF receptor with a truncated extracellular domain expressed in glioblastomas with EGFR gene amplification.. PubMed. 9(8). 2313–20. 196 indexed citations

13.

Ekstrand, A. Jonas, Noriaki Sugawa, C. David James, & V. Peter Collins. (1992). Amplified and rearranged epidermal growth factor receptor genes in human glioblastomas reveal deletions of sequences encoding portions of the N- and/or C-terminal tails.. Proceedings of the National Academy of Sciences. 89(10). 4309–4313. 469 indexed citations

14.

Ekstrand, A. Jonas, C. David James, Webster K. Cavenee, et al.. (1991). Genes for epidermal growth factor receptor, transforming growth factor alpha, and epidermal growth factor and their expression in human gliomas in vivo.. PubMed. 51(8). 2164–72. 519 indexed citations breakdown →

15.

Sugawa, Noriaki, A. Jonas Ekstrand, C. David James, & V. Peter Collins. (1990). Identical splicing of aberrant epidermal growth factor receptor transcripts from amplified rearranged genes in human glioblastomas.. Proceedings of the National Academy of Sciences. 87(21). 8602–8606. 475 indexed citations

16.

Ekstrand, A. Jonas. (1987). Human c-fos proto-oncogene mapped to chromosome 14, band q24.3?q31 possibilities for oncogene activation by chromosomal rearrangements in human neoplasms*1. Experimental Cell Research. 169(1). 262–266. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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