Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics
197212.1k citationsJ. F. R. Kerr, A H Wyllie et al.British Journal of Cancerprofile →
Cell Death: The Significance of Apoptosis
19806.2k citationsA H Wyllie, J. F. R. Kerr et al.International review of cytologyprofile →
Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation
This map shows the geographic impact of A H Wyllie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A H Wyllie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A H Wyllie more than expected).
This network shows the impact of papers produced by A H Wyllie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A H Wyllie. The network helps show where A H Wyllie may publish in the future.
Co-authorship network of co-authors of A H Wyllie
This figure shows the co-authorship network connecting the top 25 collaborators of A H Wyllie.
A scholar is included among the top collaborators of A H Wyllie based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with A H Wyllie. A H Wyllie is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Clarke, Alan R., Colin A. Purdie, David J. Harrison, et al.. (1993). Thymocyte apoptosis induced by p53-dependent and independent pathways. Nature. 362(6423). 849–852.1997 indexed citations breakdown →
Wyllie, A H, Mark J. Arends, Russell E. Morris, S. W. Walker, & Gérard I. Evan. (1992). The apoptosis endonuclease and its regulation.. PubMed. 4(6). 389–97.98 indexed citations
Morris, Russell E., A D Hargreaves, E. Duvall, & A H Wyllie. (1984). Hormone-induced cell death. 2. Surface changes in thymocytes undergoing apoptosis.. PubMed. 115(3). 426–36.165 indexed citations
18.
Wyllie, A H, G. J. Beattie, & A D Hargreaves. (1981). Chromatin changes in apoptosis. The Histochemical Journal. 13(4). 681–692.146 indexed citations
19.
Wyllie, A H, J. F. R. Kerr, & A Currie. (1980). Cell Death: The Significance of Apoptosis. International review of cytology. 68. 251–306.6193 indexed citations breakdown →
20.
Wyllie, A H. (1980). Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation. Nature. 284(5756). 555–556.3930 indexed citations breakdown →
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.