A. Di Pietro
-
- Drug Transport and Resistance Mechanisms 3
-
- Amino Acid Enzymes and Metabolism 1
- Pharmacology top 10%
-
- ATP Synthase and ATPases Research 3
- RNA and protein synthesis mechanisms 3
- Glycosylation and Glycoproteins Research 2
- Mitochondrial Function and Pathology 2
- Retinoids in leukemia and cellular processes 1
-
- Enzyme Structure and Function 2
- Co-authors
- D GautheronG. ComteDenís BarronCharles DumontetGwenaëlle ConseilDoriane TrompierGilles DivitaJean‐Michel Jault
- Cited by
- OncologyBiochemistryPharmacology
- Journals
- Journal of Biological Chemistry (3 papers)Cellular and Molecular Life Sciences (2 papers)Biochimica et Biophysica Acta (BBA) - Bioenergetics (1 paper)
- Partner nations
- FranceBelgiumUnited States
In The Last Decade
A. Di Pietro
9 papers receiving 467 citations
Peers
Comparison fields: 5 of 73
- Oncology 202
- Biochemistry 40
- Pharmacology 50
- Toxicology 15
- Molecular Biology 291
Countries citing papers authored by A. Di Pietro
This map shows the geographic impact of A. Di Pietro's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. Di Pietro with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. Di Pietro more than expected).
Fields of papers citing papers by A. Di Pietro
This network shows the impact of papers produced by A. Di Pietro. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. Di Pietro. The network helps show where A. Di Pietro may publish in the future.
Co-authorship network
The 25 scholars most cited alongside A. Di Pietro, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2003 | 50 | |
| 2 | 2002 | 201 | |
| 3 | 2001 | 92 | |
| 4 | 1993 | 11 | |
| 5 | 1993 | 10 | |
| 6 | 1993 | 49 | |
| 7 | 1989 | 11 | |
| 8 | 1988 | 32 | |
| 9 | 1975 | 23 |
About A. Di Pietro
A. Di Pietro is a scholar working on Clinical Biochemistry, Biochemistry, Cell Biology, Oncology and Molecular Biology, having authored 9 papers that have together received 479 indexed citations. Recurring topics across this work include Drug Transport and Resistance Mechanisms (3 papers), ATP Synthase and ATPases Research (3 papers), RNA and protein synthesis mechanisms (3 papers), Glycosylation and Glycoproteins Research (2 papers), Mitochondrial Function and Pathology (2 papers), Enzyme Structure and Function (2 papers), Amino Acid Enzymes and Metabolism (1 paper) and Retinoids in leukemia and cellular processes (1 paper). The work is most often cited by research in Oncology (202 citations), Biochemistry (40 citations), Pharmacology (50 citations), Toxicology (15 citations) and Molecular Biology (291 citations). A. Di Pietro has collaborated with scholars based in France, Belgium and United States. Frequent co-authors include D Gautheron, G. Comte, Denís Barron, Charles Dumontet, Gwenaëlle Conseil, Doriane Trompier, Gilles Divita, Jean‐Michel Jault, Catherine Godinot and Roger S. Goody. Their work appears in journals such as Journal of Biological Chemistry, Cellular and Molecular Life Sciences, Biochimica et Biophysica Acta (BBA) - Bioenergetics, Biochemical and Biophysical Research Communications and FEBS Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.