Identification of a key pathway required for the sterile inflammatory response triggered by dying cells
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doi.org/10.1038/nm1603 →Countries where authors are citing Identification of a key pathway required for the sterile inflammatory response triggered by dying cells
This map shows the geographic impact of Identification of a key pathway required for the sterile inflammatory response triggered by dying cells. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Identification of a key pathway required for the sterile inflammatory response triggered by dying cells with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Identification of a key pathway required for the sterile inflammatory response triggered by dying cells more than expected).
Fields of papers citing Identification of a key pathway required for the sterile inflammatory response triggered by dying cells
This network shows the impact of Identification of a key pathway required for the sterile inflammatory response triggered by dying cells. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the Identification of a key pathway required for the sterile inflammatory response triggered by dying cells.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
This paper is also available at doi.org/10.1038/nm1603.